angiotensin/arachis hypogaea

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Expression of components of the renin-angiotensin system in autosomal recessive polycystic kidney disease.

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Hypertension is a common complication in children with autosomal recessive polycystic kidney disease (ARPKD) who have survived the neonatal period. No information is available regarding the mechanism of hypertension in this condition. The renin-angiotensin system (RAS) is thought to play a role in

Arachin derived peptides as selective angiotensin I-converting enzyme (ACE) inhibitors: structure-activity relationship.

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Current attention focuses on mechanisms of controlling blood pressure through the inhibition of angiotensin I-converting enzyme (ACE). Bioactive antihypertensive peptides of food origin are increasingly gaining importance as alternates to synthetic drugs in hypertension therapy. The ACE inhibitory

In vitro formation and expansion of cysts derived from human renal cortex epithelial cells.

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Acquired renal cysts derive from terminally differentiated tubular epithelium in adults as a consequence of increased epithelial cell proliferation, fluid accumulation and extracellular matrix remodelling. To understand better how human epithelial cysts may be initiated and progressively expand,

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