ascites/triglyceride

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Changes in the rates of lipogenesis and hepatic triglyceride secretion in mice bearing Ehrlich ascites carcinoma.

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The growth of Ehrlich ascites tumors (EAT) in mice induces hypertriglyceridemia and depletion of lipid stores. H-Riop: Swiss mice in early and late stages of tumor growth were examined to investigate whether an increase in liver synthesis of fatty acids (FA) and/or an increase in the liver

Inhibition of fatty acid incorporation into adipose tissue triglycerides in Ehrlich ascites tumor-bearing mice.

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Using a recently developed technique of direct tracer injection into selective adipose tissue sites (Baker et al., Mech. Ageing Dev., 27: 295-313, 1984), we have studied the esterification of free fatty acids (FFA) to triglyceride fatty acids in the epididymal fat pads of normal and Ehrlich ascites

Ascites chylomicron: a poor substrate for hepatic triglyceride lipase.

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A patient with nephrotic syndrome and morbus Kimura (eosinophilic granuloma) showed chylous ascites. Ascites chylomicrons were analyzed and used to study the substrate specificity of lipoprotein lipase and hepatic triglyceride lipase. Ascites triglyceride and cholesterol concentrations were 191 and

Tumor extracellular triglycerides in mice during growth of Ehrlich ascites carcinoma.

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Our earlier work with Swiss-Webster mice has shown that most of the lipid in Ehrlich ascites tumor extracellular fluid is in the form of free fatty acids. This finding is in direct contradiction to earlier and subsequent reports from another laboratory that has found free fatty acids to be a very

Utilization of ascites plasma very low density lipoprotein triglycerides by Ehrlich cells.

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Much of the lipid present in the ascites plasma in which Ehrlich cells grow is contained in very low density lipoproteins (VLDL). Chemical measurements indicated that triglycerides were taken up by the cells during in vitro incubation with ascites VLDL. When tracer amounts of radioactive triolein

The opacity of portal hypertension-related ascites correlates with the fluid's triglyceride concentration.

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To determine if an elevated triglyceride concentration can explain the opacity of some cirrhotic ascites specimens, the authors measured triglyceride concentration by Coulter DACOS (Hialeah, FL) on 133 paired serum and ascitic fluid specimens. The specimens were categorized as clear or cloudy by

Lymphangiomyomatosis with chylous ascites: treatment with dietary fat restriction and medium chain triglycerides.

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Lymphangiomyomyoma is a tumor of lymphatic channels and lymph nodes, clinically manifested by recurrent chylous pleural effusions and ascites. The disease is usually progressive and unresponsive to surgery, chemotherapy, or irradiation. A case of lymphangiomyomatosis in a 24-year-old woman who

Perturbations of triglycerides but not of cholesterol metabolism are prevented by anti-tumour necrosis factor treatment in rats bearing an ascites hepatoma (Yoshida AH-130).

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Rats transplanted with the ascites hepatoma Yoshida AH-130 developed a severely progressive cachexia, characterised by marked alterations in protein and lipid metabolism. In particular, high levels of serum triglycerides and free fatty acids were associated with altered levels and distribution of

Demonstration of hepatic triglyceride lipase-like activity in ascites fluid of mice with sarcoma 180.

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The contents of apolipoproteins of plasma and ascites fluid of mice with Sarcoma 180 were measured. The apolipoprotein A-I contents of plasma decreased with development of the tumor. The apolipoprotein C-II and C-III contents of plasma reached a maximum on day 7 after tumor inoculation and then

Establishment of triglyceride cut-off values to detect chylous ascites and pleural effusions.

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OBJECTIVE Lipoprotein electrophoresis is the gold standard for the detection of chylous ascites and pleural effusions. It is, however, not suitable as a front-line test and not widely available. Most clinicians must rely solely on the quantitative determination of lipids. The aim of this work was to

Letter to the editor regarding the usefulness of the refrigeration test in addition to triglyceride levels for the detection of chylous ascites and pleural effusions.

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Chylous ascites. Management with medium-chain triglycerides and exacerbation by lymphangiography.

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ascites/triglyceride

Changes in the rates of lipogenesis and hepatic triglyceride secretion in mice bearing Ehrlich ascites carcinoma.

Inhibition of fatty acid incorporation into adipose tissue triglycerides in Ehrlich ascites tumor-bearing mice.

Ascites chylomicron: a poor substrate for hepatic triglyceride lipase.

Tumor extracellular triglycerides in mice during growth of Ehrlich ascites carcinoma.

Utilization of ascites plasma very low density lipoprotein triglycerides by Ehrlich cells.

The opacity of portal hypertension-related ascites correlates with the fluid's triglyceride concentration.

Lymphangiomyomatosis with chylous ascites: treatment with dietary fat restriction and medium chain triglycerides.

Perturbations of triglycerides but not of cholesterol metabolism are prevented by anti-tumour necrosis factor treatment in rats bearing an ascites hepatoma (Yoshida AH-130).

Demonstration of hepatic triglyceride lipase-like activity in ascites fluid of mice with sarcoma 180.

Establishment of triglyceride cut-off values to detect chylous ascites and pleural effusions.

[Chylous ascites associated with liver cirrhosis treated with medium-chain triglycerides].

Failure of medium chain triglycerides in the treatment of persistent chylous ascites secondary to lymphosarcoma.

Turnover and transport of plasma very-low-density lipoprotein triglycerides in mice bearing Ehrlich ascites carcinoma.

Letter to the editor regarding the usefulness of the refrigeration test in addition to triglyceride levels for the detection of chylous ascites and pleural effusions.

Chylous ascites. Management with medium-chain triglycerides and exacerbation by lymphangiography.

Baz done ki pi konplè remèd fèy medsin te apiye nan syans