coronary artery disease/nicotine

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Serum cotinine levels in pipe smokers: evidence against nicotine as cause of coronary heart disease.

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Serum levels of cotinine (a principal metabolite of nicotine) were studied in men who did not smoke (28), and in men who smoked cigarettes only (150), cigars only (70), and pipes only (56). The mean cotinine level for pipe smokers was 389 ng/ml, significantly higher than the mean level for cigarette

Short-Term Safety of Nicotine Replacement in Smokers Hospitalized With Coronary Heart Disease.

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Background Little is known about the safety of nicotine replacement therapy ( NRT ) in smokers hospitalized with coronary heart disease. Methods and Results We examined the short-term safety of NRT use among smokers hospitalized for coronary heart disease in a geographically and structurally diverse

Smoking and smokeless tobacco consumption: possible risk factors for coronary heart disease among young patients attending a tertiary care cardiac hospital in Bangladesh.

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OBJECTIVE To determine the risk of coronary heart disease (CHD) associated with various types of tobacco consumption among young patients aged 20-49 years attending a tertiary care cardiac hospital in Bangladesh. METHODS Case-control study. METHODS The study was undertaken at the National Institute

Cardiovascular safety of transdermal nicotine patches in patients with coronary artery disease who try to quit smoking.

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Nicotine patches are commonly used by people who try to quit smoking. Because high doses of nicotine may increase heart rate and potentiate cardiac arrhythmia or ischemia, its use in patients with coronary artery disease was investigated. The objective was to assess the cardiovascular safety of

Publication bias in the environmental tobacco smoke/coronary heart disease epidemiologic literature.

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Two approaches are used to assess publication bias in the environmental tobacco smoke/coronary heart disease (ETS/CHD) literature: (1) Statistical tests applied to all sex-specific relative risk (rr) estimates from 14 previously published studies indicate that publication bias is likely. A funnel

Tobacco smoke affects expression of peroxisome proliferator-activated receptor-gamma in monocyte/macrophages of patients with coronary heart disease.

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OBJECTIVE Tobacco smoke represents a relevant risk factor for coronary heart disease (CHD). Although peroxisome proliferator-activated receptor (PPAR)gamma activation reduces inflammation and atherosclerosis, expression of PPARgamma in cells and its modulation by smoking are poorly investigated. We

Tobacco smoking, paraoxonase-1 (PON1) activity and pleiotropic effects of simvastatin treatment in patients with stable coronary artery disease (CAD).

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BACKGROUND Tobacco smoking is the leading atherosclerosis risk factor and substantially influences its pathophysiology. OBJECTIVE The aim of this study was to assess the impact of tobacco smoking on paraoxonase-1 (PON1) activity and the relationship with pleiotropic effects of simvastatin therapy

Environmental tobacco smoke and deaths from coronary heart disease in Canada.

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A series of recent meta-analyses have concluded that non-smokers who live with smokers face an elevated risk of coronary heart disease (CHD). In this study, we estimated the number of CHD deaths among non-smokers attributable to environmental tobacco smoke (ETS) exposure in their homes.

The effects of maximising the UK's tobacco control score on inequalities in smoking prevalence and premature coronary heart disease mortality: a modelling study.

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BACKGROUND Smoking is more than twice as common among the most disadvantaged socioeconomic groups in England compared to the most affluent and is a major contributor to health-related inequalities. The United Kingdom (UK) has comprehensive smoking policies in place: regular tax increases; public

Why is environmental tobacco smoke more strongly associated with coronary heart disease than expected? A review of potential biases and experimental data.

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Despite exposure levels estimated to be equivalent to smoking only 0. 1-1.0 cigarettes per day, exposure to environmental tobacco smoke (ETS) is estimated to increase the risk of death from coronary heart disease (CHD) between 25 and 35% above the risk of nonexposed persons. This surprisingly large

Is there any association between use of smokeless tobacco products and coronary heart disease in Bangladesh?

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BACKGROUND Most epidemiological studies exploring the association between smokeless tobacco (SLT) use and coronary heart disease (CHD) have been in Western populations, and have focused on SLT products used in those countries. Few studies come from South Asian countries. Our objective was to

Toenail nicotine levels as predictors of coronary heart disease among women.

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The authors assess the ability of toenail nicotine levels as a biomarker to predict incident coronary heart disease (CHD). A nested case-control study was carried out among 62,641 women aged 36-61 years in the Nurses' Health Study cohort who provided toenail clippings in 1982. Between 1984 and 1998,

Measurement of markers of tobacco smoking in patients with coronary heart disease.

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591 patients with a history of coronary heart disease had one or more biochemical markers of tobacco smoking measured. 26% were self reported smokers and a further 4% were apparent 'smoking deceivers'. The urinary nicotine metabolite concentration is an excellent marker for tobacco smoking; breath

Association of GSTM1 and GSTT1 gene polymorphisms with coronary artery disease in relation to tobacco smoking.

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BACKGROUND Recent studies suggest that the common variant in the glutathione S-transferase (GST) M1 (GSTM1) and T1 (GSTT1) gene is associated with the risk of smoking-related coronary artery disease (CAD). Intra-ethnic as well as inter-ethnic differences are known to impact the frequencies of GST

Environmental tobacco smoke and coronary heart disease in the American Cancer Society CPS-II cohort.

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BACKGROUND Thirteen of 14 epidemiological studies have shown an increased risk of approximately 20% for coronary heart disease (CHD) for never-smokers exposed to environmental tobacco smoke (ETS), but this association remains controversial. If true, ETS might account for an estimated 35,000 to

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