creatine/stroke

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[Creatine phosphokinase in the cerebrospinal fluid of patients with stroke in the acute period].

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Creatine phosphokinase (CP) activity was increased in the cerebrospinal fluid (CSF) in stroke patients as compared to a group of control patients. CP activity tended to be higher in hemorrhagic stroke as compared to ischemic one. The brain isoenzyme CP has a major impact on its total activity (above

Correlation of the apparent diffusion coefficient and the creatine level in early ischemic stroke: a comparison of different patterns by magnetic resonance.

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It has been reported that reduction of the apparent diffusion coefficient (ADC) after stroke can persist for several days, after which the ADC increases gradually to an abnormally high level. We evaluated ADC values of stroke lesions and compared the results to the cellular density of the lesion by

Choline and creatine are not reliable denominators for calculating metabolite ratios in acute ischemic stroke.

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OBJECTIVE Choline and creatine are commonly used as denominators for other metabolites in ischemic stroke spectroscopy, assuming that they do not change. We investigated their concentration variation over time after stroke. METHODS Choline and creatine concentrations were measured by proton MR

Cardiac Troponin T and creatine kinase MB fraction levels among patients with acute ischemic stroke in Nigeria.

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BACKGROUND Stroke has been a global burden, with increasing morbidity and mortality. Serum cardiac troponin t (cTnT) and creatine kinase (CK-MB) fraction are reported to be elevated in patients admitted with acute ischaemic stroke and high level of these biomarkers indicated more severe stroke and

Serum creatine kinase activity in the elderly following a stroke.

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A serial measurement of serum creatine kinase (CK) activity and its isoenzymes were made in elderly patients following an acute stroke. Seven out of ten patients had elevated CK levels. The maximum concentration was observed between 12-24 h and the level returned to normal within 84 h after a

A complicated case of exertional heat stroke in a military setting with persistent elevation of creatine phosphokinase.

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A case is presented of exertional heat stroke in a military setting with numerous complications that have been previously described. In addition, the complication of late rhabdomyolysis with profound elevation of creatine phosphokinase (CPK) is here described for the first time. This rare condition

Creatine kinase BB activity in serum of patients with acute stroke: correlation with the severity of brain damage.

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BB-CK activity was measured in 11 patients with stroke and in 10 controls. Blood samples were taken 36 hours after the clinical stroke onset in every patient. Sera were stored at -80 degrees and analyzed within two months. The creatine kinase isoenzymatic pattern was determined by ion-exchange

Role of creatine phosphokinase in predicting acute renal failure in hypocalcemic exertional heat stroke.

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Recruits frequently develop hypocalcemia in exertional heat stroke (ExHS) with rhabdomyolysis and acute renal failure (ARF) from intensive training. It usually indicated severe skeletal muscle damage. However, the relative risk of ARF in ExHS patients complicated with hypocalcemia was unknown. The

Early time course of N-acetylaspartate, creatine and phosphocreatine, and compounds containing choline in the brain after acute stroke. A proton magnetic resonance spectroscopy study.

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OBJECTIVE The early time course after acute stroke of cerebral N-acetylaspartate, creatine and phosphocreatine, and compounds containing choline was studied in vivo by means of localized water-suppressed proton magnetic resonance spectroscopy. METHODS Eight patients with acute stroke were studied

Creatine loading does not impact on stroke performance in tennis.

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The effect of acute creatine supplementation on stroke quality was investigated during simulated match play. Well-trained tennis players reported to the test center on two occasions. On each occasion they performed the Leuven Tennis Performance Test (LTPT) and a 70 m shuttle run (SHR). During 5 days

Improved reperfusion and neuroprotection by creatine in a mouse model of stroke.

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Stroke leads to energy failure and subsequent neuronal cell loss. Creatine and phosphocreatine constitute a cellular energy buffering and transport system, and dietary creatine supplementation was shown to protect neurons in several models of neurodegeneration. Although creatine has recently been

Creatine kinase-MB elevation after stroke is not cardiac in origin: comparison with troponin T levels.

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OBJECTIVE Creatine kinase-MB (CK-MB) increases in some patients with stroke, with no clear evidence of an acute coronary syndrome. Its elevations have been suggested to represent a biological marker for stroke-related myocardial injury. Troponin T has superior sensitivity and specificity to CK-MB in

Myelin basic protein and creatine kinase BB isoenzyme as CSF markers of intracranial tumors and stroke.

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In patients with intracranial tumors (ICT) and acute cerebral infarctions (CI), both necrosis and reversible changes occur in central nervous system (CNS) tissue. The damaged CNS cells release specific substances into the cerebrospinal fluid (CFS). Radioimmunoassay (RIA)-determined myelin basic

Evaluation of creatine phosphokinase in cases of cerebrovascular accidents.

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Relationship between circulating platelets and serum concentrations of creatine kinase in stroke.

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