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glutamic/infarction

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Acute myocardial infarction. Urine glutamic oxalacetic transminase activity.

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The urinary content of glutamic oxalacetic transaminase (UGO-T) was determined in 16 consecutive patients with acute myocardial infarction. In all of them it was above normal. In some patients the UGO-T remained elevated for a longer period than did the blood content of that enzyme.It is possible
The serum iron was subnormal on the first three days, following acute myocardial infarction in thirty patients. This is significantly less than the incidence of abnormalities demonstrated by assays of creatine phosphokinase (CPK) and glutamic oxalacetic transaminase (SGOT) in serum. Occasional
BACKGROUND Myocardial infarction (MI) is a leading cause of death worldwide. Given that a family history is an independent risk factor for coronary artery disease, genetic variants are thought to contribute directly to the development of this condition. The identification of susceptibility genes for

[Behavior of glutamic-pyruvic transaminase during the clinical course of acute myocardial infarct].

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The increase in serum Alanine-aminotransferase (SGPT) levels in 104 patients with acute myocardial infarction is studied. The time course of serum enzyme changes shows two peaks of activity: in the first days after infarction and at the end of the second week of the disease. The clinical

[Serum glutamic-oxalacetic and glutamic-pyruvic transaminases in myocardial infarct].

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[Glutamic oxalacetic transaminase & myocardial infarct].

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[Glutamic oxalacetic transaminase & myocardial infarct].

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[The glutamic-oxalacetic and glutamic-pyruvic transaminase activity of the serum in experimental cerebral infarction].

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[Glutamic-oxalacetic transaminase and myocardial infarct].

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