glutamic/necrosis

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Tumor necrosis factor microsatellite polymorphism influences the development of insulin dependency in adult-onset diabetes patients with the DRB11502-DQB10601 allele and anti-glutamic acid decarboxylase antibodies.

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Recently, several studies have demonstrated that tumor necrosis factor microsatellite polymorphism (TNFalpha) contributes to the susceptibility of type 1 diabetes. This study investigates the influence of TNFalpha on the predisposition to insulin dependency in adult-onset diabetic patients with type

[L-Glutamic acid modulates the cytotoxic effect of tumor necrosis factor in the HL-60 cell line].

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L-Glutamic acid was shown to increase the stability of cells of the HL-60 line of human promyelocyte leukemia to the cytotoxic action of tumor necrosis factor alpha (TNF-alpha) due to the inhibition of apoptotic and NF-kappaB-activating cascades induced by this cytokine. At the same time, L-glutamic

A comparison of serum creatine phosphokinase and serum glutamic oxalacetic transaminase in skeletal muscle necrosis.

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Rabbits were poisoned by the intraperitoneal injection of plasmocid dihydroiodide at a dosage of 80 mg. per kg. body weight. Animals were euthanatized 1, 2, 5, 7, 9, 12, 24 and 48 hours after plasmocid injection and serum and diaphragmatic muscle collected. The serum was examined for its serum

Influence of L-glutamic acid on binding of interleukin-1beta, tumour necrosis factor-alpha and interleukin-6 to HL-60 cells.

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To elucidate the mechanism of differentiating activity on L-Glu to HL-60 cells, its influence on binding of human recombinant interleukine-1beta (rHuIL-1beta), tumour necrosis factor-alpha (rHuTNF-alpha) and interleukine-6 (IL-6) by HL-60 cells was studied. It was established, that L-Glu converted

[Behavior of serum transaminase activity (glutamic acid-ox-alacetic acid transaminase) in allyl alcohol necrosis of rat liver].

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Serum glutamic-oxaloacetic transaminase activity as an index of centrilobular liver cell necrosis in cardiac and circulatory failure.

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Serum glutamic oxalacetic-transaminase activity in the diagnosis of myocardial infarction and hepatic necrosis.

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Acute hepatic necrosis following iproniazid therapy; value of glutamic-oxaloacetic transaminase estimation in early detection.

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Studies on necrosis of mouse liver in vitro. Alterations in activities of succinoxidase, succinic dehydrogenase, glutamic dehydrogenase, acid phosphatase, uricase, glucose-6-phosphatase and NAD-pyrophosphorylase.

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[Glutamic dehydrogenase and alcohol dehydrogenase of blood in various conditions of experimental hepatic necrosis: hepatitis due to MHV-3 virus, acute poisoning due to carbon tetrachloride].

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Administration of poly-D-glutamic acid induces proliferation of erythropoietin-producing peritubular cells in rat kidney.

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Erythropoietin (EPO), a 34-kDa glycoprotein, is produced predominantly by peritubular interstitial cells (PIC) in the renal cortex and is physiologically released when ambient oxygen pressure falls. However, the exact nature of EPO-producing cells in the kidney is not well understood. We discovered

CD4(+) T cells from glutamic acid decarboxylase (GAD)65-specific T cell receptor transgenic mice are not diabetogenic and can delay diabetes transfer.

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Glutamic acid decarboxylase (GAD)65 is an early and important antigen in both human diabetes mellitus and the nonobese diabetic (NOD) mouse. However, the exact role of GAD65-specific T cells in diabetes pathogenesis is unclear. T cell responses to GAD65 occur early in diabetes pathogenesis, yet only

[Effect of L-glutamic acid on the reception of cytokines by HL-60 cells].

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L-Glutamic acid at a concentration of 0.1 microM was found to induce differentiation of the cell line of HL-60 promyelocytic leukemia into granulocytes or neutrophils. The HL-60 cells have no specific glutamate-binding sites, but L-glutamic acid influences the reception of several cytokines by these

Glutamic-oxaloacetic transaminase activity of serum in mice with viral hepatitis.

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Experimental infectious mouse hepatitis is associated with an increase in glutamic-oxaloacetic transaminase activity of the serum (SGO-T). A relationship appears to exist between the rise in SGO-T activity and (a) the size of the virus inoculum, (b) the blood virus titer, and (c) the degree of liver

[Studying the neuroprotective effect of the novel glutamic acid derivative neiroglutam on focal cerebral ischemia in rats].

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We have studied the neuroprotective effect of the novel glutamic acid derivative neiroglutam on reversible focal cerebral ischemia in rats. The neuroprotective drug action was assessed by the ability to reduce the severity of neurological deficit (1, 2, 3, 5 and 7 days), forelimb fine-motor

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glutamic/necrosis

Tumor necrosis factor microsatellite polymorphism influences the development of insulin dependency in adult-onset diabetes patients with the DRB1*1502-DQB1*0601 allele and anti-glutamic acid decarboxylase antibodies.

[L-Glutamic acid modulates the cytotoxic effect of tumor necrosis factor in the HL-60 cell line].

A comparison of serum creatine phosphokinase and serum glutamic oxalacetic transaminase in skeletal muscle necrosis.

Influence of L-glutamic acid on binding of interleukin-1beta, tumour necrosis factor-alpha and interleukin-6 to HL-60 cells.

[Behavior of serum transaminase activity (glutamic acid-ox-alacetic acid transaminase) in allyl alcohol necrosis of rat liver].

Serum glutamic-oxaloacetic transaminase activity as an index of centrilobular liver cell necrosis in cardiac and circulatory failure.

Serum glutamic oxalacetic-transaminase activity in the diagnosis of myocardial infarction and hepatic necrosis.

Acute hepatic necrosis following iproniazid therapy; value of glutamic-oxaloacetic transaminase estimation in early detection.

Studies on necrosis of mouse liver in vitro. Alterations in activities of succinoxidase, succinic dehydrogenase, glutamic dehydrogenase, acid phosphatase, uricase, glucose-6-phosphatase and NAD-pyrophosphorylase.

[Glutamic dehydrogenase and alcohol dehydrogenase of blood in various conditions of experimental hepatic necrosis: hepatitis due to MHV-3 virus, acute poisoning due to carbon tetrachloride].

Administration of poly-D-glutamic acid induces proliferation of erythropoietin-producing peritubular cells in rat kidney.

CD4(+) T cells from glutamic acid decarboxylase (GAD)65-specific T cell receptor transgenic mice are not diabetogenic and can delay diabetes transfer.

[Effect of L-glutamic acid on the reception of cytokines by HL-60 cells].

Glutamic-oxaloacetic transaminase activity of serum in mice with viral hepatitis.

[Studying the neuroprotective effect of the novel glutamic acid derivative neiroglutam on focal cerebral ischemia in rats].

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Tumor necrosis factor microsatellite polymorphism influences the development of insulin dependency in adult-onset diabetes patients with the DRB11502-DQB10601 allele and anti-glutamic acid decarboxylase antibodies.