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glyceride/infarction
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[Changes in the erythrocyte concentrations of various phospholipids-glycerides, erythrocyte peroxide resistance and plasma and erythrocyte alpha-tocopherol levels in acute myocardial infarct].
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Free fatty acids, neutral glycerides, and phosphoglycerides in transient focal cerebral ischemia.
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Cerebral ischemia is known to cause an increase in levels of free fatty acids (FFAs) and diacylglycerols (DGs), although the mechanism(s) leading to these changes is not well understood. In this study, we examined FFA and DG levels along with those of other lipids in rats during and after transient
Unrecognized diabetes and myocardial necrosis: predictors of hyperglycemia in myocardial infarction.
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BACKGROUND
Hyperglycemia in the acute phase of myocardial infarction is an important prognostic factor. However, its pathophysiology is not fully understood.
OBJECTIVE
To analyze simultaneously the correlation between hyperglycemia and biochemical markers related to stress, glucose and lipid
Abnormalities of composition and of in vitro lipolysis products of human small very low density lipoproteins in hypertriglyceridemia.
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Small (Sf 20-100) very low density lipoprotein (VLDL) particles were prepared by density gradient ultracentrifugation of plasma from normolipidemic and type IV hypertriglyceridemic post-infarction patients and healthy controls. The small VLDL separated from the plasma of severely
Pathophysiology of long-chain polyene fatty acids in heart muscle.
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The polyene fatty acid composition of cardiac phospholipids is modified by age, diet and stress in the rat. In the aging heart there is a progressive replacement of 18:2n6 by 20:4n6 in phosphatidylcholine (PC) and a replacement of 18:2n6 by 22:6n3 in phosphatidylethanolamine (PE). Norepinephrine
Vitamin D and ischaemic heart disease.
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Vitamin D has been proposed as a risk factor of ischaemic heart disease. In 12 patients with acute myocardial infarction the major circulating vitamin D metabolite, 25-hydroxy-cholecalciferol (25-HCC), did not show any fluctuations during the first 4 days after onset of symptoms. The serum 25-HCC
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