heart arrest/tyrosine

Lyen an sove nan clipboard la

Chwazi kalite sòt

Paj 1 soti nan 39 rezilta yo

Regulation of tyrosine protein kinase receptor Trk-B and motor function in rats following cardiac arrest.

Se sèlman itilizatè ki anrejistre yo ki ka tradwi atik yo

Following 10 min cardiac arrest and resuscitation, male Sprague-Dawley rats developed posthypoxic myoclonus. Sixty days later, the motor function of the animals was restored. In the present study, we investigated brain levels of tyrosine protein kinase receptor Trk-B with quantitative immunoblot

Therapeutic hypothermia attenuates brain edema in a pig model of cardiac arrest: Possible role of the angiopoietin-Tie-2 system.

Se sèlman itilizatè ki anrejistre yo ki ka tradwi atik yo

OBJECTIVE This study aimed to clarify whether therapeutic hypothermia protects against cerebral edema following cardiac arrest (CA) and cardiopulmonary resuscitation (CPR) in a porcine model via regulating the angiopoietin-Tie-2 ligand-receptor system. METHODS Male pigs were randomized into the

Detoxification with hemabsorption after cardiac arrest does not improve neurologic recovery. Review and outcome study in dogs.

Se sèlman itilizatè ki anrejistre yo ki ka tradwi atik yo

We and others hypothesized that noxious substances released after prolonged cardiac arrest from malfunctioning liver, kidneys, or intestine (e.g. bacterial toxins, aromatic amino acids), might hamper recovery of the brain. The highly detoxifying effect of hemabsorption (i.e. hemoperfusion) with

Cardiovascular toxicity associated with small molecule tyrosine kinase inhibitors currently in clinical use.

Se sèlman itilizatè ki anrejistre yo ki ka tradwi atik yo

BACKGROUND Tyrosine kinase inhibitors (TKIs) have changed the concepts of systemic therapy for a variety of advanced solid and hematologic malignancies. However, their toxicity can be significant, and includes both cardiac and non-cardiac effects. METHODS The authors evaluate comprehensively the

Comparison of low-flow cardiopulmonary bypass and circulatory arrest on brain oxygen and metabolism.

Se sèlman itilizatè ki anrejistre yo ki ka tradwi atik yo

BACKGROUND In the neonatal brain we measured oxygen (Bo(2)), extracellular striatal dopamine (DA), and striatal tissue levels of ortho-tyrosine (o-tyr) during low-flow cardiopulmonary bypass (LFCPB) or deep hypothermic circulatory arrest (DHCA) and the post-bypass recovery period. METHODS Newborn

Effect of granulocyte-colony stimulating factor on expression of selected proteins involved in regulation of apoptosis in the brain of newborn piglets after cardiopulmonary bypass and deep hypothermic circulatory arrest.

Se sèlman itilizatè ki anrejistre yo ki ka tradwi atik yo

OBJECTIVE The study objective was to investigate the effect of granulocyte-colony stimulating factor on the expression of proteins that regulate apoptosis in newborn piglet brain after cardiopulmonary bypass and deep hypothermic circulatory arrest. METHODS The newborn piglets were assigned to 3

Protein tyrosine phosphorylation in the ischemic brain.

Se sèlman itilizatè ki anrejistre yo ki ka tradwi atik yo

Cerebral ischemia, a pathological condition in which brain tissue experiences a shortage of cerebral blood flow, is associated with cerebrovascular disease, brain trauma, epilepsy, and cardiac arrest. A reduction in blood flow leaves the brain tissue unsupplied with oxygen and glucose, thus leading

Insulin induces tyrosine phosphorylation of a 90-kDa protein during postischemic brain reperfusion.

Se sèlman itilizatè ki anrejistre yo ki ka tradwi atik yo

Rat brain nuclear proteins were examined for tyrosine phosphorylation after resuscitation from a 10-min cardiac arrest. Insulin (1 unit/kg intravenously), given immediately after resuscitation, caused a marked increase in tyrosine phosphorylation of a 90-kDa brain protein. This effect occurred

Effects of NaHS and hydroxylamine on the expressions of brain-derived neurotrophic factor and its receptors in rats after cardiac arrest and cardiopulmonary resuscitation.

Se sèlman itilizatè ki anrejistre yo ki ka tradwi atik yo

BACKGROUND

H₂S can also protect nerve cells. The objective of the study is to investigate the effects of hydrogen sulfide (H₂S) on the expressions of brain-derived neurotrophic factor (BDNF) and its receptors, tyrosine protein kinase B (TrkB) and p75

Hypothermic reperfusion after cardiac arrest augments brain-derived neurotrophic factor activation.

Se sèlman itilizatè ki anrejistre yo ki ka tradwi atik yo

Induction of mild hypothermia improves neurologic outcome after global cerebral ischemia. This study measured levels of brain-derived neurotrophic factor (BDNF) and nerve growth factor (NGF) in hippocampal tissue of rats after resuscitation from 8 minutes of normothermic, asphyxial cardiac arrest.

Increases in striatal and hippocampal impedance and extracellular levels of amino acids by cardiac arrest in freely moving rats.

Se sèlman itilizatè ki anrejistre yo ki ka tradwi atik yo

The time course of changes in the tissue impedance and the levels of extracellular transmitter and non-transmitter amino acids was studied in the striatum and hippocampus of the unanesthetized rat after cardiac arrest. Electrodes were implanted for the continuous measurement of tissue impedance so

Selection of reference genes for quantitative real-time PCR in a rat asphyxial cardiac arrest model.

Se sèlman itilizatè ki anrejistre yo ki ka tradwi atik yo

BACKGROUND Cardiac arrest, and the associated arrest of blood circulation, immediately leads to permanent brain damage because of the exhaustion of oxygen, glucose and energy resources in the brain. Most hippocampal CA1 neurons die during the first week post the insult. Molecular data concerning the

Early mitochondrial dysfunction in electron transfer activity and reactive oxygen species generation after cardiac arrest.

Se sèlman itilizatè ki anrejistre yo ki ka tradwi atik yo

OBJECTIVE Mitochondrial biology appears central to many conditions that progress to death but remains poorly characterized after cardiac arrest. Mitochondrial dysfunction in electron transfer and reactive oxygen species leakage during ischemia may lead to downstream events including mitochondrial

Src tyrosine kinase inhibition prevents pulmonary ischemia-reperfusion-induced acute lung injury.

Se sèlman itilizatè ki anrejistre yo ki ka tradwi atik yo

OBJECTIVE Pulmonary ischemia-reperfusion is a pathological process seen in several clinical conditions, including lung transplantation, cardiopulmonary bypass, resuscitation for circulatory arrest, atherosclerosis, and pulmonary embolism. A better understanding of its molecular mechanisms is very

Activation of mitochondrial STAT-3 and reduced mitochondria damage during hypothermia treatment for post-cardiac arrest myocardial dysfunction.

Se sèlman itilizatè ki anrejistre yo ki ka tradwi atik yo

While therapeutic hypothermia improves the outcomes of individuals in cardiac arrest, the hemodynamic responses and mechanisms which underlie hypothermia-induced cardioprotection are not fully understood. Therefore, we investigated the mechanism by which induced hypothermia preserves cardiac

Antre nan paj
facebook nou an

Herbal & Natural Medicine

Baz done ki pi konplè remèd fèy medsin te apiye nan syans

Travay nan 55 lang
Geri èrbal te apiye nan syans
Remèd fèy rekonesans pa imaj
Kat entèaktif GPS - tag zèb sou kote (vini byento)
Li piblikasyon syantifik ki gen rapò ak rechèch ou an
Search remèd fèy medsin pa efè yo
Izeganize enterè ou yo ak rete kanpe fè dat ak rechèch la nouvèl, esè klinik ak rive

Tape yon sentòm oswa yon maladi epi li sou remèd fèy ki ta ka ede, tape yon zèb ak wè maladi ak sentòm li itilize kont.
* Tout enfòmasyon baze sou rechèch syantifik pibliye