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Paj 1 soti nan 804 rezilta yo
[Changes in serum and bile tumor necrosis factor alpha before and after percutaneous transhepatic cholangio-drainage for obstructive jaundice].
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The aim of this study was to evaluate the correlation between tumor necrosis factor alpha (TNF alpha) and jaundice reduction by percutaneous transhepatic cholangio-drainage (PTCD) in patients with obstructive jaundice. Thirty patients who had undergone PTCD were divided into two groups: those with
Obstructive jaundice in rats results in exaggerated hepatic production of tumor necrosis factor-alpha and systemic and tissue tumor necrosis factor-alpha levels after endotoxin.
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BACKGROUND
Obstructive jaundice (OJ) predisposes patients to postoperative sepsis. We determined whether OJ led to an increased endotoxin stimulated tumor necrosis factor-alpha (TNF-alpha) production by macrophage-rich organs and whether a lack of intraluminal gut bile contributed to this increased
Alterations of tumor necrosis factor-alpha and interleukin 6 production and activity of the reticuloendothelial system in experimental obstructive jaundice in rats.
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BACKGROUND
Immunological changes are well recognised in obstructive jaundice. The aim of this study was to monitor plasma tumor necrosis factor alpha (TNF-alpha) and interleukin-6 (IL-6) levels in rats with obstructive jaundice.
METHODS
The ability of splenocytes and peritoneal exudate cells (PEC)
Effects of different drainage procedures on levels of serum endotoxin and tumor necrosis factor in patients with malignant obstructive jaundice.
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OBJECTIVE
To assess the influence of different drainage procedures on the levels of serum endotoxin and tumor necrosis factor (TNF) in patients with malignant obstructive jaundice (MOJ).
METHODS
The levels of endotoxin and TNF in 46 patients with MOJ were measured before and after
Effect of bile acid replacement on endotoxin-induced tumor necrosis factor-alpha production in obstructive jaundice.
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There is a high incidence of perioperative morbidity and mortality in patients with obstructive jaundice due to sepsis. Tumor necrosis factor-a (TNF-alpha) is considered a crucial mediator in inducing and processing the inflammatory cascade. We hypothesize that obstructive jaundice leads to an
Hepatic tumor necrosis factor-alpha production and distant organ dysfunction in a murine model of obstructive jaundice.
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BACKGROUND
Multisystem organ dysfunction frequently occurs following obstructive jaundice, but its etiology remains unclear. This study was undertaken to evaluate the role for endogenous tumor necrosis factor-alpha (TNF-alpha) production in the renal and pulmonary injury that accompanies obstructive
[Effect of jiawei dachaihu decoction on blood T lymphocyte subsets and tumor necrosis factor in patients with obstructive jaundice].
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OBJECTIVE
To observe the effect of the preoperative Jiawei Dachaihu decoction in improving cellular immune functions in patients with obstructive jaundice.
METHODS
Forty patients were randomly divided into two groups with (group B) and without (group A) the herbal therapy. And their preoperative T
A FURTHER CONTRIBUTION TO THE KNOWLEDGE OF MULTIPLE NON-INFLAMMATORY NECROSIS OF THE LIVER WITH JAUNDICE (HEPAR NECROTICUM CUM ICTERO), AND TO THE KNOWLEDGE OF CELL DEGENERATION AND CYTOLYSIS IN GENERAL.
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[Jaundice due to choledochal necrosis during acute pancreatitis].
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[Cardioarticular rheumatism and jaundice with centro-lobular necrosis].
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[An autopsy case of an elderly man with jaundice due to the necrosis and loss of intrahepatic interlobular bile ducts].
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Liver cell necrosis in chlorpromazine jaundice (allergic cholangiolitis). A serial study of twenty-six needle biopsy specimens in nine patients.
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[Unusual forms of severe jaundice due to hepatitis: massive hepatic steatosis and renal tubular necrosis. (Apropos of 2 cases)].
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[Postoperative fever, jaundice and cognition disorder: acute hepatic necrosis caused by halothane].
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CENTRILOBULAR HEPATIC NECROSIS IN CARDIAC FAILURE. ONE CASE WITH SEVERE ACUTE JAUNDICE.
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