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liver cirrhosis/phosphatase
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[Gamma-glutamyltransferase and alkaline phosphatase in the serum and saliva of patients with hepatic tumors and hepatic cirrhosis].
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The authors have previously described the presence of gammaglutamyl transferase and alkaline phosphatase in the saliva of healthy individuals and of patients with hepatobiliary and pancreatic diseases. The authors show in this work the values of said enzymes in the blood serum and saliva of 23 cases
Src-homology protein tyrosine phosphatase-1 agonist, SC-43, reduces liver fibrosis.
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This study aimed to investigate the role of src-homology protein tyrosine phosphatase-1 (SHP-1)-signal transducer and activator of transcription 3 (STAT3) pathway in liver fibrogenesis and the anti-fibrotic effect of SHP-1 agonist. The antifibrotic activity of SC-43, a sorafenib derivative with an
Activation of protein serine/threonine phosphatase PP2Cα efficiently prevents liver fibrosis.
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BACKGROUND
Over-activation of TGFβ signaling pathway and uncontrolled cell proliferation of hepatic stellate cells (HSCs) play pivotal roles in liver fibrogenesis, while the protein serine/threonine phosphatase PP2Cα was reported to negatively regulate TGFβ signaling pathway and cell cycle. Our
Intestinal type alkaline phosphatase hyperphosphatasemia associated with liver cirrhosis.
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Hyperphosphatasemia due to increased intestinal type serum alkaline phosphatase was noted in a 48-year-old male who had asymptomatic liver cirrhosis. The alkaline phosphatase activity in the serum was 828 U/l (our reference range in adults: 57-194 U/l), 94% of which was of the intestinal type as
Curcumin up-regulates phosphatase and tensin homologue deleted on chromosome 10 through microRNA-mediated control of DNA methylation--a novel mechanism suppressing liver fibrosis.
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Phosphatase and tensin homologue deleted on chromosome 10 (PTEN) has been reported to play a role in the suppression of activated hepatic stellate cells (HSCs). Moreover, it has been demonstrated that hypermethylation of the PTEN promoter is responsible for the loss of PTEN expression during HSC
Alkaline phosphatase isoenzymes in liver cirrhosis.
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The study of a group of 114 hepatic patients allowed us to demonstrate the presence of an isophosphatase band, called ALP4, which can be of value as an index of evolutive cirrhosis. A certain correlation between the presence of this alkaline phosphatase isoenzyme in serum and histological evidence
Effects and mechanism of adenovirus-mediated phosphatase and tension homologue deleted on chromosome ten gene on collagen deposition in rat liver fibrosis.
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OBJECTIVE
To evaluate the effects of phosphatase and tension homologue deleted on chromosome ten (PTEN) gene on collagen metabolism in hepatic fibrosis and the underlying mechanisms.
METHODS
Rat primary hepatic stellate cells (HSCs) and human LX-2 cells were transfected with adenovirus containing
GAUCHER'S DISEASE (WITH ELEVATED SERUM ACID PHOSPHATASE LEVEL) MASQUERADING AS CIRRHOSIS OF THE LIVER.
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[Correlation between small intestine phosphatase and serum pseudocholinesterase in liver cirrhosis].
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[Nutrition-related shifts and clinical diagnostic significance of alkaline phosphatase of the small intestine in liver cirrhosis].
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[Value of determination of alkaline phosphatase isoenzymes for differential diagnosis of obstructive jaundice and biliary liver cirrhosis].
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[Changes in the blood alkaline phosphatase level under the influence of vitamin B12 and choline in liver cirrhosis].
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[The activity of alkaline and acid phosphatase in chronic hepatitis and cirrhosis of the liver].
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[Blood phosphatase activity in liver cirrhosis & obstructive jaundice].
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Alkaline phosphatase in liver cirrhosis and rectal cancer.
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