liver cirrhosis/rapeseed

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Liver fibrosis of the turkey on rapeseed products.

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Sclerosis of the liver accompanied by hydropericardium of long standing was observed in turkeys fed rapeseed products for 16 weeks. Histologically, extensive increases of fibrous tissue composed predominantly of reticular fibres and centrilobular degeneration of parenchymal cells were the main

Ultrastructural study of liver fibrosis in turkeys fed diets containing rapeseed meal.

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Livers of turkeys fed low and high glucosinolate rapeseed meal diets for 16 weeks were examined ultrastructurally. Hepatocytes from birds fed on Tower rapeseed meal (0.15 mg of glucosinolate per g) showed a mild accumulation of lipid droplets and an apparent increase in lysosomes and microbodies.

[Rapeseed in experimental cirrhosis in the rat].

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It has been reported that rapeseed in hens has been able to induce liver reticulum lysis and in rats it was possible to prevent the development of liver cirrhosis induced by carbon tetrachloride (CCl 4). The objective of these experiments is 1st. to confirm by a more extensive study the prevention

Rapeseed Protein-Derived Antioxidant Peptide RAP Ameliorates Nonalcoholic Steatohepatitis and Related Metabolic Disorders in Mice.

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Rapeseed protein hydrolysates have recently shown in vitro antioxidant and anti-inflammatory activities. However, scant data exist about their in vivo activities. Here, we report that the peptide DHNNPQIR (hereinafter referred to as RAP-8), a bioactive peptide originated from rapeseed protein,

RAP-8 ameliorates liver fibrosis by modulating cell cycle and oxidative stress.

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The rapeseed protein derived peptide DHNNPQIR (named as RAP-8) has been previously reported to possess antioxidant activity and alleviate liver fibrosis. The purpose of the present study was to investigate the potential crucial pathways involved in ameliorating liver fibrosis of

Toxic oil stimulates collagen synthesis acting at a pretranslational level in cultured fat-storing cells.

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OBJECTIVE The toxic oil syndrome appeared in Spain in 1981 as a result of ingestion of rapeseed oil denatured with aniline. Some patients developed scleroderma-like skin lesions and liver cirrhosis. Mechanisms of these fibrotic lesions are not known. The present study was designed to investigate the

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