oxalate/atrophy

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Acute oxalate nephropathy due to pancreatic atrophy in newly diagnosed pancreatic carcinoma.

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Acute oxalate nephropathy can occur due to primary hyperoxaluria and secondary hyperoxaluria. The primary hyperoxalurias are a group of autosomal recessive disorders of endogenous oxalate overproduction. Secondary hyperoxaluria may occur as a result of excess dietary intake, poisoning with oxalate

Diosmin reduces calcium oxalate deposition and tissue degeneration in nephrolithiasis in rats: a stereological study.

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OBJECTIVE Kidney stones (nephrolithiasis) are a widespread disease. Thus, blocking stone formation and finding new therapeutic methods is an important area of study. Diosmin (a major component of the bile) is known to have antioxidant as well as renoprotective effects. The present investigation

Ultrastructural localization of calcium in peripheral nerve fibres undergoing Wallerian degeneration: an oxalate-pyroantimonate and X-ray microanalysis study.

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Calcium was demonstrated ultrastructurally as an electron dense precipitate with the oxalate-pyroantimonate technique in rat sural nerves undergoing early Wallerian degeneration after surgical crush (30 h after lesion). In normal nerves (controls) the distribution of calcium precipitates in the

Vibrational dynamics of a non-degenerate ultrafast rotor: the (C12,C13)-oxalate ion.

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Molecular ions undergoing ultrafast conformational changes on the same time scale of water motions are of significant importance in condensed phase dynamics. However, the characterization of systems with fast molecular motions has proven to be both experimentally and theoretically challenging. Here,

Rapid renal deterioration secondary to oxalate in a patient with diabetic gastroenteropathy.

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We report the case of a white woman with insulin-dependent diabetes for 12 years who had rapid deterioration in renal function over a 7-month period. A renal biopsy showed widespread deposition of a polarizing crystalline material consistent with calcium oxalate. Fat malabsorption due to diabetic

Oral Reference Dose for ethylene glycol based on oxalate crystal-induced renal tubule degeneration as the critical effect.

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Several risk assessments have been conducted for ethylene glycol (EG). These assessments identified the kidney as the primary target organ for chronic effects. None of these assessments have incorporated the robust database of species-specific toxicokinetic and toxicodynamic studies with EG and its

Mode of action: oxalate crystal-induced renal tubule degeneration and glycolic acid-induced dysmorphogenesis--renal and developmental effects of ethylene glycol.

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Ethylene glycol can cause both renal and developmental toxicity, with metabolism playing a key role in the mode of action (MOA) for each form of toxicity. Renal toxicity is ascribed to the terminal metabolite oxalic acid, which precipitates in the kidney in the form of calcium oxalate crystals and

Renal calcium phosphate and oxalate deposition in prolonged vitamin B6 deficiency: studies on a rat model of urolithiasis.

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OBJECTIVE To determine the effect on kidney oxalate-salt deposition of a prolonged diet which induced vitamin B6 deficiency in adult rats, as there are reports of the pathogenic involvement of vitamin B6 deficiency in the formation of renal calcium oxalate calculi. METHODS The study comprised 24

Lithium Bis(oxalate)borate Reinforces the Interphase on Li-Metal Anodes.

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Li metal provides an ideal anode for the highest energy density batteries, but its reactivity with electrolytes brings poor cycling stability. Electrolyte additives have been employed to effectively improve the cycling stability, often with the underlying mechanism poorly understood. In this work,

Effects of naftidrofuryl oxalate on microsphere-induced changes in acetylcholine and amino acid content of rat brain regions.

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Effects of naftidrofuryl oxalate (naftidrofuryl) on neurotransmitter, acetylcholine, and amino acid content of brain regions following microsphere-induced cerebral embolism were examined to elucidate its possible therapeutic effects on ischemic brain. Rats received 900 microspheres (48 microns in

[The concentration of plasma anion oxalate in children treatment antibiotics beta lactame].

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The aim of work was the assessment of plasma anion oxalate (Ox) concentration in children during antibacterial treatment depending on way and time of antibiotic administration. METHODS The examinations were carried out in 80 children, without nephrolithiasis, aged 10.1 +/- 4.3 years with

[Urinary calcium oxalate supersaturation beyond nephrolithiasis. Relationship with tubulointerstitial damage].

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A number of studies have demonstrated that the urinary ion activity product (IAP) of calcium oxalate (CaOx), as an index of urinary CaOx supersaturation (SS), is higher in renal stone formers than in normal subjects. Besides, the relation between CaOx SS and lithogenesis, crystal CaOx exposition can

Pathological features of oxalate nephrosis in a population of koalas (Phascolarctos cinereus) in South Australia.

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The wild and captive koala population of the Mt Lofty Ranges in South Australia has a high level of renal dysfunction in which crystals consistent with calcium oxalate have been observed in the kidneys. This study aimed to describe the pathological features of the renal disease in this population,

Nuclear pore complex oxalate binding protein p62: its expression on oxalate exposure to VERO cells.

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Oxalate rich stones are the most common among the various stones. Oxalate binding protein plays a vital role in the transport of oxalate. Nuclear pore complex (NPC) contains a protein of molecular weight 62 kDa and it has maximum oxalate binding activity. The physiological significance of the

[Oxalate nephropathy due to malabsorption syndrome].

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Enteric oxalate nephropathy is caused by hyperoxaluria. Factors which contribute to excessive oxalate absorption are an abundance of free fatty acids in the intestine due to malabsorption, changes in the microbiome, and bowel inflammation. We present two cases that illustrate different

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