oxalate/vomiting

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[On the use of cerium oxalate, 8-chlorotheophyllinate diphenhydramine and D-N-methylamphetamine in some types of vomiting of children].

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In this issue - May 2019: West Nile virus in horses · Treating horses with dexamethasone via nebulisation · Animal hoarding in NSW · Dangers of powdered washing soda for emesis in dogs · Pneumocystis in a dog following toceranib phosphate · Oxalate nephrosis in koalas.

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The question of cerium oxalate as a prophylactic against postoperative vomiting.

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The Treatment of Gastric Vomiting by Oxalate of Cerium.

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Calcium oxalate urolithiasis in a cat with a functional parathyroid adenocarcinoma.

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A 9-year-old castrated male domestic shorthair cat with dysuria, anorexia, vomiting, and lethargy was admitted to the veterinary teaching hospital. A large, firm mass was palpable in the ventral cervical region. Hypercalcemia, azotemia, and nonregenerative anemia were evident on serum biochemical

Renal transplantation in cats with calcium oxalate urolithiasis: 19 cases (1997-2004).

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OBJECTIVE To determine outcome of renal transplantation in cats with renal failure associated with calcium oxalate urolithiasis. METHODS Retrospective case series. METHODS 19 cats. METHODS Medical records were reviewed for evaluation of signalment, preoperative clinical signs, physical examination

[Effectiveness of several antiemetics in vomiting induced by orally administrated copper sulfate in cats and dogs].

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Long marketed antiemetics, which are still used in medicine or veterinary medicine and whose sites of actions were either assumed to be peripheral or unknown, were subjected to evaluation as antiemetics, against oral copper sulfate emesis. In cats, ethyl aminobenzoate 0.5 g/head, cerium oxalate 0.1

Diet-induced oxalate nephropathy.

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Oxalate nephropathy is a rare condition and may be overlooked due to lack of recognition and understanding of triggers. An 81-year-old man was sent to nephrologist because of significantly increased creatinine (1.5-1.9 mg/dL) noted for 3 months. He had well-controlled diabetes but no history of

Orlistat-induced oxalate nephropathy may be dose-independent and present as a late manifestation.

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We present the case of a 61-year-old Caucasian male veteran who had been on orlistat (120mg dosing) for four years, and had changed to the over-the-counter (OTC) form, Alli (orlistat 60mg), about three months before presentation. He had been experiencing nausea and vomiting for three weeks prior to

Acute oxalate nephropathy after ingestion of star fruit.

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Acute oxalate nephropathy associated with ingestion of star fruit (carambola) has not been reported before. We report the first two cases. These patients developed nausea, vomiting, abdominal pain, and backache within hours of ingesting large quantities of sour carambola juice; then acute renal

Diagnosis and surgical management of ureteral calculi in dogs: 16 cases (1990-2003).

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OBJECTIVE To examine the clinical signs, laboratory and radiographic findings, surgical technique, ureterolith composition, and post-operative outcomes in dogs managed surgically for ureterolithiasis to determine whether surgical removal of ureteroliths in dogs resulted in a positive clinical

[Reversible hyperuricemia in case of ureteral colics (author's transl)].

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On about one fourth of the patients ureteral colics caused by oxalate and phosphate calculi lead to a reversible hyperuricemia. This result seems to be important, because we might wrongly diagnose a uric acid calculus. The reason for hyperuricemia is a temporary diuretic disturbance of the uric

Crystals seen on CSF microscopy in a case of suspected subarachnoid haemorrhage.

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Although crystals are rarely identified on cerebrospinal fluid (CSF) microscopy, their presence can be of significant diagnostic value. We report a case of oxalate crystals seen on CSF microscopy of a 43-year-old woman. The patient presented with headaches, nausea and vomiting. CT of the head showed

Clinicopathologic findings in dogs and cats with ethylene glycol intoxication.

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Clinicopathologic findings were retrospectively evaluated in 26 cats and 24 dogs with ethylene glycol intoxication. Common clinical signs were ataxia, depression, vomiting, and hypothermia. Characteristic alterations in the hemogram and serum chemical profile included neutrophilia, lymphopenia,

4-Methylpyrazole as treatment for naturally acquired ethylene glycol intoxication in dogs.

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Eight dogs with ethylene glycol intoxication were treated with 4-methylpyrazole, an alcohol dehydrogenase inhibitor. Dogs had clinical signs referable to ethylene glycol ingestion including ataxia, depression, vomiting, polyuria, and dehydration. Metabolic abnormalities included high anion gap

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