polyuria/hemorrhage

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[Massive natriuresis and polyuria after triple craniocervical subarachnoid hemorrhage: cerebral salt wasting syndrome?].

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A thirty-year-old male patient suffered subarachnoidal haemorrhage from an angioma positioned in the cranio-cervical transition. After rebleeding twice the patient developed a hydrocephalus internus malresorptivus and excessive natriuresis and polyuria, accompanied by depressed renin activity and

Polyuria and cerebral vasospasm after aneurysmal subarachnoid hemorrhage.

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BACKGROUND Natriuresis with polyuria is common after aneurysmal subarachnoid hemorrhage (aSAH). Previous studies have shown an increased risk of symptomatic cerebral vasospasm or delayed cerebral ischemia (DCI) in patients with hyponatremia and/or the cerebral salt wasting syndrome (CSW). However,

Use of the vasopressin analogue desmopressin for polyuria and bleeding disorders.

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[Cerebral salt wasting syndrome as a complication of subarachnoid hemorrhage in a girl with nephrotic syndrome - a case report].

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Cerebral salt wasting syndrome (CSWS), characterized by natriuresis, polyuria, and hypovolemia, is a rare complication of central nervous system injury or disease.12-year-old girl was admitted with second attack of nephrotic syndrome (NS). On admission she

[Hyponatremia and cerebral vasospasm in patients with aneurysmal subarachnoid hemorrhage].

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We studied retrospectively the relationship between hyponatremia and cerebral vasospasm in 121 consecutive patients with aneurysmal subarachnoid hemorrhage. In 19 patients sodium levels fell below 130 mEq/l on at least two consecutive days. Hyponatremia developed at average 8.9 hospital day and

[Vascular permeability and microcirculation in patients with hemorrhagic fever associated with renal syndrome].

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Vascular permeability and microcirculation (MC) were studied in 25 patients with hemorrhagic fever associated with the renal syndrome (HFRS). A considerable increase in vascular permeability for liquid and protein was recorded during oligoanuria and polyuria. These parameters slowly returned to

A case report of dengue hemorrhagic fever complicated with diabetic ketoacidosis in a child: challenges in clinical management

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Background: Diabetic ketoacidosis (DKA) is a common presentation of type 1 diabetes mellitus (T1DM) precipitated by various bacterial and viral infections. Dengue infection is no exception for this and can be a precipitating factor for

Relationship between the clinical features and the viral antigen in the extremity blood of the patients with hemorrhagic fever with renal syndrome.

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The direct immunogold silver staining (D-IGSS) method was used to detect the viral antigen in the extremity blood of 67 cases of hemorrhagic fever with renal syndrome. The positive rate of viral antigen was the highest during the fever, hypotension and oligouria phrase; and the rate dropped

Hanta hemorrhagic fever with renal syndrome: a case report and review.

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Hantavirus infection is seldom reported in Taiwan. The spectrum of clinical severity ranges from mild to severe and may cover Hantavirus pulmonary syndrome and hemorrhagic fever with renal syndrome (HFRS). We report a case of HFRS in a 24-year-old soldier presenting with fever, chills, abdominal

Epidemic hemorrhagic fever revisited.

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Epidemic hemorrhagic fever is a disease characterized by phases of sequential fever, hypotension, oliguria, and polyuria. This disease represented a major hazard for American soldiers during the Korean War (1950-1953) and is a persistent problem in Asia, the USSR, and northern Europe. Advances in

[Von Willebrand factor, antithrombin III and 5'-nucleotidase in the blood and the athrombogenic properties of the vascular wall in patients with hemorrhagic fever with renal syndrome].

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Willebrand's factor (WF), antithrombin III (AT-III), blood 5'-nucleotidase and athrombogenic properties of the vascular wall were studied in the course of the disease in 69 patients suffering from hemorrhagic fever with renal syndrome (HFRS). The patients were chiefly under 27 years, had severe or

Role of the kidney and lung in the handling of prostaglandin E in hemorrhagic shock.

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The polyuria and hyposthenuria noted particularly following blood transfusion after prolonged periods of hypotension (dog, monkey) seem best explained by a prostaglandin-antidiuretic hormone (PG-ADH) antagonism, operating primarily in the renal medulla. The kidney releases greatly enhanced amounts

Neuroendocrine dysfunction in patients recovering from subarachnoid hemorrhage.

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OBJECTIVE Subarachnoid hemorrhage (SAH) is a recently identified risk factor for hypopituitarism, particularly growth hormone (GH) and corticotrophins deficiencies. The aim of our study was to identify possible predictor(s) for neuroendocrine dysfunction in SAH survivors. METHODS Pituitary function

Acute renal failure in hemorrhagic hypotension: cellular energetics and renal function.

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In male Wistar rats, renal adenosine triphosphate (ATP), inorganic phosphate (Pi) and intracellular pH were measured by 31phosphorus nuclear magnetic resonance (31P NMR) and correlated with renal function before, during, and for one hour after a period of 30 to 40 minutes hemorrhagic hypotension. In

[The biggest epidemic of hemorrhagic fever with renal syndrome in Croatia].

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BACKGROUND The largest outbreak of hemorrhagic fever with renal syndrome (HFRS) to date occurred in Croatia in the year 2002. The epidemic started in winter, lasted throughout spring to summer months, ending not earlier than November. OBJECTIVE The aim of this study was to investigate and analyze

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