pulmonary edema/albumin

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Colloid solutions in the critically ill. A randomised comparison of albumin and polygeline 2. Serum albumin concentration and incidences of pulmonary oedema and acute renal failure.

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All patients admitted to an Intensive Care Unit were assigned randomly to one of two groups, A and B. Group A received colloid volume replacement as 4.5% albumin whilst group B received a synthetic colloid, polygeline. This study describes the changes in serum albumin concentration in survivors and

Effects of albumin and/or furosemide therapy on pulmonary edema induced by hydrochloric acid aspiration in rabbits.

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Aspiration of hydrochloric acid in rabbits resulted in an increased P(A-a)O2 together with increases in both lung water volume and lung extravascular albumin. This finding suggests lung damage following acid aspiration is related to changes in capillary permeability, with pulmonary edema resulting

[Role of albumin in pulmonary edema and septic shock, plasma volume expansion excluded].

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Patients with septic shock deserve a global approach. Intravascular volume loading is part of the treatment. However several questions remain open: what are the respective contributions of volume expansion and vaso-active drugs in the restoration of blood pressure and increase of cardiac output,

Bolus intravenous 0.9% saline, but not 4% albumin or 5% glucose, causes interstitial pulmonary edema in healthy subjects.

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Rapid intravenous (iv) infusion of 0.9% saline alters respiratory mechanics in healthy subjects. However, the relative cardiovascular and respiratory effects of bolus iv crystalloid vs. colloid are unknown. Six healthy male volunteers were given 30 ml/kg iv 0.9% saline, 4% albumin, and 5% glucose at

[Albumin exchange and polymorphonuclear vascular transit in hyperoxic pulmonary oedema in awake rats].

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Vascular inflammatory response to hyperoxia (FIO2 greater than 0.98) was studied in awake jugular and carotid catheterized rats. Simultaneous i.v. injection of 125I albumin (125I A), 99mTc polymorphonuclear cells (99mTc PMN) and 51Cr red blood cells (51Cr RBC) allowed to study both the macromolecule

Interstitial exclusion of albumin in rabbit lung during development of pulmonary oedema.

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The modifications of the macromolecular sieving properties of the pulmonary extracellular tissue matrix were studied in adult anaesthetized rabbits (n = 10) exposed to increased tissue hydration. Exclusion of albumin from the perivascular pulmonary interstitial space was determined by using the

Confocal- and electron-microscopic localization of FITC-albumin in H2O2-induced pulmonary edema.

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Fluorescein isothiocyanate-conjugated bovine serum albumin (FITC-BSA) was used to detect sites of protein leakage in rat lungs perfused for 15-60 min with H2O2 (75, 150, and 300 microM). Leaky vessels were localized by confocal laser microscopy. Endothelial routes of protein leakage were identified

Albumin therapy in permeability pulmonary edema.

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The effect of albumin on extravascular lung water in animals and patients with low-pressure pulmonary edema.

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[Studies of the albumin-I 131 content of certain organs during pulmonary edema induced with adrenaline in rabbits].

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Possible increased risk of pulmonary edema in patients with hepatorenal syndrome on adding octreotide to albumin / noradrenaline therapies.

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Albumin and pulmonary oedema.

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Transvascular albumin flux and the interstitial water volume in experimental pulmonary oedema in dogs.

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[Treatment of acute pulmonary edema by human serum albumin in cardiovascular surgery].

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Association Between Angiopoietin-2 and Enterovirus 71 Induced Pulmonary Edema.

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OBJECTIVE To characterize pulmonary edema (PE) fluid induced by enterovirus 71 (EV71) infection, elucidate the relationship between angiopoietin-2 (Ang-2) and PE, and explore the pathogenesis of PE. METHODS Clinical data were collected from critical infants with EV71 infection. The infants were

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