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3 rezilta yo
[Influence of rapeseed oil feeding on morphology and oxidative properties of mitochondria isolated from the rat heart].
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Mitochondria from heart of rats fed a diet rich in rapeseed oil for two months from weaning, oxidized oleic acid a little better than those from animals fed a control diet containing peanut oil; but their activity decreased much more in presence of potassium cyanide which is an oxidation inhibitor.
[Effects of hypobaric hypoxia and erucic acid on mitochondrial triglycerides in rat heart].
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At normal atmospheric pressure, an erucic acid-supplemented diet produces an increase of heart mitochondrial triglycerides. With the same quantity of erucic acid, hypoxia emphasizes the triglyceride overload in rat heart mitochondria. Thus, hypoxia increases the risk caused by rapeseed-oil. In
Toxic oil syndrome: a current clinical and epidemiologic summary, including comparisons with the eosinophilia-myalgia syndrome.
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In the spring and summer of 1981, an epidemic of a new illness now referred to as the toxic oil syndrome occurred in central and northwestern Spain, resulting in some 20,000 cases, 12,000 hospital admissions and greater than 300 deaths in the 1st year of the epidemic. The initial onset of illness
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