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ulcer/phosphatase
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[Lipid peroxidation, the enzyme antioxidative system and acid phosphatase content of the gastric mucosa in stomach ulcer].
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Lipid peroxidation, a state of the antioxidative system and activity of acid phosphatase were studied in mucosal membrane of 53 patients with ulcerous disease of stomach. Increase of the acid phosphatase activity in cytoplasm, activation of lipid peroxidation and inhibition of the antioxidative
[Changes in alkaline phosphatase activity and blood phosphorus levels in healthy persons and patients with peptic ulcer].
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Venous blood total phosphatase activity, its thermostable fraction, and inorganic phosphorus levels were measured thrice within 20-64 min in the course of registration of regular motor activity of the gastrointestinal tract in 38 men aged 18 to 57 (9 healthy and 29 with peptic ulcer). These
[Distribution and properties of the alkaline phosphatase in the human gastric mucosa in peptic ulcer].
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In duodenal ulcer alkaline phosphatase activity was the highest in mucosa of pyloric region of curvature ventriculi minor and it decreased distinctly in the tissues removed from pylorus. In cases of gastric ulcer, the enzymatic activity was high throughout the curvatura ventriculi minor reaching the
[Phagocytosis and NBT test and phosphatase activity of peripheral blood neutrophils in patients with untreated duodenal ulcer].
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In 21 patients with active, endoscopically confirmed untreated duodenal ulcer and in a control group of 20 persons, the studies were carried out of peripheral blood neutrophils which included: 1) test with the assessment of neutrophil phagocytic activity against live bacterial cells of the
Study of serum intestinal alkaline phosphatase isoenzyme in correlation with the ABO blood group system and secretory status in ulcer patients.
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Serum alkaline phosphatase (AP) electrophoretic phenotype and the level of its activity inhibited by l-phenylalanine 5 mM (the stereospecific inhibitor of the intestinal AP isoenzyme) were investigated in 312 subjects (132 healthy controls, 89 patients with duodenal ulcer, 31 with gastric ulcer and
[The alkaline phosphatase activity of the blood serum as a factor reflecting the chronic inflammatory process in the skin in generalized forms of psoriasis, neurodermatitis, eczema and trophic ulcers].
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Blood serum alkaline phosphatase (AP) level was found elevated in the patients suffering from eczema, neurodermatitis, eczema with large skin sites involved, and in those with trophic ulcers of various origins, as against normal subjects and reference patients with sarcoidosis and Kaposi's sarcoma.
Mice deficient in protein tyrosine phosphatase receptor type Z are resistant to gastric ulcer induction by VacA of Helicobacter pylori.
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The vacuolating cytotoxin VacA produced by Helicobacter pylori causes massive cellular vacuolation in vitro and gastric tissue damage in vivo, leading to gastric ulcers, when administered intragastrically. Here we report that mice deficient in protein tyrosine phosphatase receptor type Z (Ptprz,
[Acid and alkaline phosphatases in the vessels of posttraumatic wounds and ulcers that have not healed for a long period].
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[Serum phosphatase activity in parodontosis in patients with duodenal peptic ulcer].
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[Change in the blood picture, serum protein makeup and leukocyte phosphatase activity in peptic ulcer].
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[The behavior of alkaline phosphatase and nucleic acids in normal gastric mucosa, in peptic ulcer and in epithelial gastric tumors with special reference to the alkaline phosphatase and thymonucleic acid relationship].
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Isoenzymes of alkaline phosphatase in patients operated upon for peptic ulcer.
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[Distribution and activity of acid phosphatase in the liver of patients affected with stomach and duodenum ulcers].
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[Histochemical study of phosphatase activity in gastroduodenal ulcer].
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[Phosphatase distribution and activity in the liver cells of patients suffering from stomach and duodenum ulcers. II. Adenosinotriphosphatase, 5-nucleotidase and alkaline phosphatase].
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