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Oldal 1 tól től 116 eredmények
Occurrence of acute pulmonary edema in experimental alkalosis.
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[Alkalosis during treatment of edema in patients with pulmonary insufficiency].
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HYPOKALEMIC ALKALOSIS EDEMA WITH INCREASED DESOXYCORTICOSTERONE EXCRETION.
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[Analysis of bronchoalveolar lavage fluid in a case of high altitude pulmonary edema].
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A 29-year-old male climber developed high altitude pulmonary edema (HAPE), progressing from headache and dyspnea to disturbance of consciousness in the Japan Alps. He was admitted to Shinshu University Hospital. Physical examination on admission revealed a cyanotic patient with coarse crackles and
Hypocapnic but not metabolic alkalosis impairs alveolar fluid reabsorption.
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Acid-base disturbances, such as metabolic or respiratory alkalosis, are relatively common in critically ill patients. We examined the effects of alkalosis (hypocapnic or metabolic alkalosis) on alveolar fluid reabsorption in the isolated and continuously perfused rat lung model. We found that
Buffered hydrochloric acid: a modern method of treating metabolic alkalosis.
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Twenty-one patients with metabolic alkalosis were treated successfully with intravenous hydrochloric acid (HCl) buffered in an amino acid solution (TPN). No complications of HCl were seen. TPN was used to meet energy needs and provide a buffering effect through the interaction of HCl and amino
Comparative efficacy and safety of bumetanide and furosemide in long-term treatment of edema due to congestive heart failure.
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Bumetanide and furosemide were compared for efficacy in reducing edema due to congestive heart failure in 28 patients (21 receiving bumetanide and seven receiving furosemide) in a long-term study for periods from one week to 18 months. In both groups the patients showed decreases in body weight,
Injurious effects of hypocapnic alkalosis in the isolated lung.
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Mechanical ventilation can worsen morbidity and mortality by causing ventilator-associated lung injury, especially where adverse ventilatory strategies are employed. Adverse strategies commonly involve hyperventilation, which frequently results in hypocapnia. Although hypocapnia is associated with
Salicylate-induced pulmonary edema--a near-miss diagnosis.
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A 43-year-old white woman presented to the emergency department with confusion, agitation, and progressive dyspnea. Chest x-ray revealed pulmonary edema. Initial diagnostic considerations were pneumonia, pulmonary embolism, sepsis, central nervous system infection, substance toxicity, and heart
High-altitude pulmonary edema: a collective review.
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In summary, HAPE is a potentially fatal form of noncardiogenic PE seen in a small number of individuals visiting above 9,000 ft in elevation. The pathophysiology is uncertain but is probably due, at least in part, to hydrostatic and capillary permeability abnormalities of the pulmonary vascular bed
Time course of postischemic intracellular alkalosis reflects the duration of ischemia.
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We investigated the long-term (up to 1 week) relationships between the duration of cerebral ischemia and postischemic energy metabolic profile, pH, and tissue edema in the rat. Ten rats each were subjected to 8 or 12 min of forebrain ischemia induced by bicarotid occlusion concurrent with systemic
Hypokalemia and metabolic alkalosis resulting from overuse of magnesium oxide.
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A 23 year old woman had taken large doses of magnesium oxide (up to 20 to 30g per day) initially for habitual constipation and later for idiopathic edema instead of diuretics, until the concealed abuse was discovered. During the abuse she showed hypokalemia and metabolic alkalosis. When the
[Effect of hypocapnia/alkalosis on the fluid filtration rate in isolated and perfused rabbit lungs].
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Hypocapnia/alkalosis is a consequence of several lung and metabolic pathologies. The aim of this study was to determine whether the increase of fluid filtration rate (FFR) that occurs during Hypocapnia/alkalosis circumstances is determined by hypocapnia, alkalosis or both. 7 groups were formed
Mediators of alkalosis-induced relaxation of piglet pulmonary veins.
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Pulmonary venous constriction leads to significant pulmonary hypertension and increased edema formation in several models using newborns. Although alkalosis is widely used in treating neonatal and pediatric pulmonary hypertension, its effects on pulmonary venous tone have not previously been
Acid-base disturbances in cardiogenic pulmonary edema.
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Eighty-one consecutive cases of uncomplicated cardiogenic pulmonary edema (CPE) were retrospectively graded for severity of chest roentgenogram (CXR) changes and grouped according to primary acid-base abnormalities, either single or mixed. Mean age was 72, 50 male, 31 female. Twenty-three percent
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