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Oldal 1 tól től 58 eredmények
Alteration of the serine protease PRSS56 causes angle-closure glaucoma in mice and posterior microphthalmia in humans and mice.
Csak regisztrált felhasználók fordíthatnak cikkeket
Belépés Regisztrálás
Angle-closure glaucoma (ACG) is a subset of glaucoma affecting 16 million people. Although 4 million people are bilaterally blind from ACG, the causative molecular mechanisms of ACG remain to be defined. High intraocular pressure induces glaucoma in ACG. High intraocular pressure traditionally was
Single nucleotide polymorphisms of metabolic syndrome-related genes in primary open angle glaucoma.
Csak regisztrált felhasználók fordíthatnak cikkeket
Belépés Regisztrálás
OBJECTIVE
To analyze single nucleotide polymorphisms (SNP) of primary open angle glaucoma- and metabolic syndrome-related genes in primary open angle glaucoma (POAG), in order to elucidate the roles of metabolic syndrome as a risk factor in POAG progress.
METHODS
SNP genotypes and alleles of
Extracellular proteases and neuronal cell death.
Csak regisztrált felhasználók fordíthatnak cikkeket
Belépés Regisztrálás
Neuronal cell death occurs during development of the central nervous system as well as in pathological situations such as acute injury and progressive degenerative diseases. For instance, granule cells in the developing cerebellum and neuronal precursor cells in the cortex undergo programmed cell
Secreted leukocyte protease inhibitor is present in aqueous humours from cataracts and other eye pathologies.
Csak regisztrált felhasználók fordíthatnak cikkeket
Belépés Regisztrálás
Previous studies identified serine, cysteine and metalloproteases in normal aqueous humours (AH) and suggested that a balance between proteases and their inhibitors may play a role in the modulation of the AH outflow. We aimed to determine whether secretory leukocyte protease inhibitor (SLPI), a
Heat shock proteins, immunity and glaucoma.
Csak regisztrált felhasználók fordíthatnak cikkeket
Belépés Regisztrálás
Glaucoma is no longer viewed simply as elevated intraocular pressure (IOP) that damages the optic nerve. In addition to high IOP, evidence is rapidly accumulating that suggests damage to the optic nerve may be initiated or sustained by any number of factors including ischemia, excitotoxicity,
Synthetic Polyclonal-Derived CDR Peptides as an Innovative Strategy in Glaucoma Therapy.
Csak regisztrált felhasználók fordíthatnak cikkeket
Belépés Regisztrálás
The pathogenesis of glaucoma is strongly associated with the occurrence of autoimmune-mediated loss of retinal ganglion cells (RGCs) and additionally, recent evidence shows that specific antibody-derived signature peptides are significantly differentially expressed in sera of primary-open angle
Potential drug delivery approaches for XFS-associated and XFS-associated glaucoma.
Csak regisztrált felhasználók fordíthatnak cikkeket
Belépés Regisztrálás
Key tissue targets in treating exfoliation syndrome (XFS) and the associated glaucoma include lens, iris, and ciliary body, which produce the exfoliative material, and the trabecular meshwork, which may be impaired by the exfoliative material. In addition to antiglaucoma drug therapy, strategies for
Proteases in eye development and disease.
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Belépés Regisztrálás
The eye is one of the classical systems in developmental biology. Furthermore, diseases of the eye, many of which have a developmental basis, have devastating effects that often result in blindness. Proteases have diverse roles in ocular physiology and pathophysiology. Here, a broad overview is
Neuroprotection in glaucoma using calpain-1 inhibitors: regional differences in calpain-1 activity in the trabecular meshwork, optic nerve and implications for therapeutics.
Csak regisztrált felhasználók fordíthatnak cikkeket
Belépés Regisztrálás
Glaucoma is a group of irreversible blinding eye diseases affecting over 70 million people worldwide. Systemic delivery of calpain-1 inhibitors was proposed as a neuroprotection strategy for the prevention of progressive optic nerve damage in glaucoma. We present a general review of calpain-1 and an
Tissue differential microarray analysis of dexamethasone induction reveals potential mechanisms of steroid glaucoma.
Csak regisztrált felhasználók fordíthatnak cikkeket
Belépés Regisztrálás
OBJECTIVE
To identify myocilin (TIGR/MYOC) properties that are specific to the human trabecular meshwork (HTM). To search for genes highly expressed in dexamethasone (DEX)-induced HTM cells that are barely expressed or absent in DEX-induced cells from other tissues.
METHODS
TIGR/MYOC induction by
Aqueous humor protein dysregulation in primary angle-closure glaucoma.
Csak regisztrált felhasználók fordíthatnak cikkeket
Belépés Regisztrálás
OBJECTIVE
Primary angle-closure glaucoma (PACG) is associated with increased intraocular pressure, optic nerve damage, and progressive vision loss, but the molecular mechanism that underpins retinal ganglion neuropathy in PACG remains poorly understood. To better understand the pathogenesis of human
Apoptosis of retinal ganglion cells in glaucoma: an update of the molecular pathways involved in cell death.
Csak regisztrált felhasználók fordíthatnak cikkeket
Belépés Regisztrálás
Apoptosis is a genetically controlled form of cell death that ganglion cells undergo during normal development of the retina and in diseases affecting the optic nerve, such as glaucoma. This mechanism of cell death is controlled by specific genes and their products that are activated in the dying
Cystatin a, a potential common link for mutant myocilin causative glaucoma.
Csak regisztrált felhasználók fordíthatnak cikkeket
Belépés Regisztrálás
Myocilin (MYOC) is a 504 aa secreted glycoprotein induced by stress factors in the trabecular meshwork tissue of the eye, where it was discovered. Mutations in MYOC are linked to glaucoma. The glaucoma phenotype of each of the different MYOC mutation varies, but all of them cause elevated
Glaucoma: ocular Alzheimer's disease?
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Belépés Regisztrálás
Glaucoma is a chronic neurodegeneration of the optic nerve and one of the leading causes of vision loss in the world among the aging. Retinal ganglion cells (RGCs) have been shown to die by apoptosis, or programmed cell death. Central to apoptosis is the activation of specific proteases, termed
Glaucoma is associated with plasmin proteolytic activation mediated through oxidative inactivation of neuroserpin.
Csak regisztrált felhasználók fordíthatnak cikkeket
Belépés Regisztrálás
Neuroserpin is a serine protease inhibitor that regulates the activity of plasmin and its activators in the neuronal tissues. This study provides novel evidence of regulatory effect of the neuroserpin on plasmin proteolytic activity in the retina in glaucoma. Human retinal and vitreous tissues from
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Írjon be egy tünetet vagy betegséget, és olvassa el azokat a gyógynövényeket, amelyek segíthetnek, beírhat egy gyógynövényt, és megtekintheti azokat a betegségeket és tüneteket, amelyek ellen használják.
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