juglans/nekrózis

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First Report of Pantoea agglomerans Causing Brown Apical Necrosis of Walnut in China.

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Belépés Regisztrálás

Brown apical necrosis (BAN) of walnut (Juglans regia L.) causes premature fruit drop and yield losses and has been reported to be an important walnut production problem in Spain, Italy, France, and Turkey (1,2). A number of organisms have been associated with BAN on walnut: Xanthomonas arboricola

Environmental and Pathogenic Factors Inducing Brown Apical Necrosis on Fruit of English (Persian) Walnut.

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Brown apical necrosis (BAN) is a most recently described disease affecting English (Persian) walnut fruit. BAN was only recorded in intensively managed walnut orchards and was found to be a disease complex mainly caused by Fusarium species. All fungi associated with this disease are polyphagous and

Apical Necrosis and Premature Drop of Persian (English) Walnut Fruit Caused by Xanthomonas arboricola pv. juglandis.

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Etiological and epidemiological aspects of apical necrosis of walnut fruit were studied on cultivars Chandler, Franquette, and Hartley in a Spanish walnut orchard during 2007 and 2008. Affected fruit showed brown necrosis beginning at the blossom end of nuts; these symptoms differed from lesions of

Occurrence and Etiology of Brown Apical Necrosis on Persian (English) Walnut Fruit.

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In 1998, a severe fruit drop was observed in Italy, principally on cv. Lara Persian (English) walnut (Juglans regia). Dropped fruit showed a brown patch at the blossom end and blackening and rot of inner tissues. The disease, called brown apical necrosis (BAN), was investigated on fruit collected in

Evaluating the effects of Juglans regia L. extract on hyperglycaemia and insulin sensitivity in experimental type 2 diabetes in rat.

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Objective: The present study aimed to investigate the effects of Juglans regia (JR) extract on hyperglycaemia and insulin sensitivity. Methods: Forty rats were divided into 4 groups: Control (C), Diabetes Control (DC), Diabetes + Juglans regia (D + JR) and Diabetes +

Olive oil and walnut breakfasts reduce the postprandial inflammatory response in mononuclear cells compared with a butter breakfast in healthy men.

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BACKGROUND Inflammation is crucial in all stages of atherosclerosis, and few studies have investigated the effect of dietary fat on markers of inflammation related to this disease during the postprandial period. OBJECTIVE To evaluate the chronic effects of dietary fat on the postprandial expression

Inhibitory effects of Juglans mandshurica leaf on allergic dermatitis-like skin lesions-induced by 2,4-dinitrochlorobenzene in mice.

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Allergic dermatitis among common skin diseases is a chronic and recurrent inflammatory skin disorder caused by genetic, environmental, allergens as well as microbial factors. Allergic dermatitis patients clinically present skin erythematous plaques, eruption, elevated serum immunoglobulin E (IgE)

Juglone, a naphthoquinone from walnut, exerts cytotoxic and genotoxic effects against cultured melanoma tumor cells.

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This study demonstrates cytotoxic and genotoxic potential of juglone, a chief constituent of walnut, and its underlying mechanisms against melanoma cells. MTT assay and clonogenic assay were used to study cytotoxicity, micronucleus assay to assess genotoxicity, glutathione (GSH) assay and

Rice-induced enterocolitis in an infant: TH1/TH2 cellular hypersensitivity and absent IgE reactivity.

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BACKGROUND Although food allergy is common in children, rice allergy is unusual in Western cultures. OBJECTIVE To report a case of T-cell-mediated rice intolerance in an 11-month-old girl. METHODS To evaluate the intolerance to rice in this patient, a graded rice food challenge was performed. To

Black walnut (Juglans nigra) toxicosis: a model for equine laminitis.

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Twelve light horse geldings developed laminitis within 8 to 12 h of being dosed by nasogastric tube with an aqueous extract of black walnut (Juglans nigra). Four of the 12 horses developed the severe signs of grade 3 laminitis (lame at a walk, refused to lift feet). Laminitis was accompanied by mild

Hepatoprotective effects of Juglans regia extract against CCl4-induced oxidative damage in rats.

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BACKGROUND Different parts of the walnut [Juglans regia L. (Juglandaceae)] have been used in folk medicine for protection against liver injury, although its actual efficacy remains uncertain. OBJECTIVE The present study investigated the protective effect of walnut leaf extract against carbon

First Report of Fusarium semitectum as the Agent of Twig Cankers on Persian (English) Walnut in Italy.

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English (Persian) walnut (Juglans regia) is the most widely cultivated walnut species. During the last 10 years, the increment of walnut cultivation in Italy has been accompanied by changes in cultural management. Changes were addressed to develop highly specialized cultivations with intensive

First Report of Twig and Branch Dieback of English Walnut (Juglans regia) Caused by Neofusicoccum mediterraneum in California.

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California produces 99% of the U.S. English walnut crop with more than 30 cultivars on ~89,000 ha. Production for 2008 was ~436,000 tons with a value of $527 million. In early summer of 2009 and 2010, branch and twig dieback of English walnut (Juglans regia L.) was detected in orchards in Yolo

Walnut-Associated Fatty Acids Inhibit LPS-Induced Activation of BV-2 Microglia.

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Walnuts have high levels of the omega-3 fatty acid alpha-linolenic acid (C18:3n-3, ALA) and the omega-6 fatty acid linoleic acid (C18:2n-6, LA). Previous research has demonstrated that pre-treatment of BV-2 microglia with walnut extract inhibited lipopolysaccharide (LPS)-induced activation of

Walnut phenolic extract inhibits nuclear factor kappaB signaling in intestinal epithelial cells, and ameliorates experimental colitis and colitis-associated colon cancer in mice.

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OBJECTIVE Walnuts (Juglans regia) are known to have anti-cancer and immunomodulatory effects. However, little information is available on the effects of walnut phenolic extract (WPE) on intestinal inflammation and colitis-associated colon cancer. METHODS COLO205 cells were pretreated with WPE and

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