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adriamycin/stroke
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Effects of doxorubicin (adriamycin) on systemic hemodynamics and myocardial blood flow.
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The effect of adriamycin (doxorubicin) on systemic hemodynamics was assessed in the anesthetized rabbit. Blood flow was measured by the radioactive microsphere method before and 20 min after adriamycin. Cardiac output and stroke volume fell significantly (p less than 0.05), while myocardial blood
Adriamycin-induced cardiomyopathy in the dog--an appropriate model for research on partial left ventriculectomy?
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OBJECTIVE
To evaluate the adriamycin-induced cardiomyopathy in the dog for research on partial left ventriculectomy (PLV).
METHODS
An intracoronary catheter was introduced into the left main stem via the first diagonal branch in a retrograde fashion in 6 adult FBI (Foxhound Boehringer Ingelheim)
[Evaluation of adriamycin cardiotoxicity using equilibrium radionuclide ventriculography. II. Results during exercise].
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The aim of the study was to evaluate the influence of a cardiotoxic therapy with Adriamycin (ADM) on left ventricular pump function. In 348 patients with malignant tumors, 606 equilibrium radionuclide ventriculographies (ERNV) were performed. In 90 patients, resting studies (R-ERNV) were followed by
[Evaluation of adriamycin cardiotoxicity using equilibrium radionuclide ventriculography. I. Results at rest].
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Results of 606 equilibrium radionuclide ventriculographies performed in 348 non-selected patients receiving Adriamycin (ADM) therapy were stored in a data base system. The aim of the study was to assess the influence of a potential cardiotoxic therapy on left ventricular pump function. Increasing
Selective left ventricular adriamycin-induced cardiomyopathy in the pig.
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OBJECTIVE
The aim of this study was the development of an experimental cardiomyopathy induced with Adriamycin (Pharmacia & Upjohn, Erlangen, Germany) with selective toxic damage of the left ventricular myocardium that avoided an ischemic component.
METHODS
An intracoronary catheter was implanted
[Contractile function of the isolated heart in chronic adriamycin-induced myocardial lesions].
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Adriamycin, administered to rats for 4 weeks, caused insufficiency of isolated heart contractility with a twofold reduction of cardiac output in surviving animals. The same cumulative dose of adriamycin, administered to rats over 10 weeks, was not associated with any significant reduction of the
Effect of carvedilol on pulse pressure and left ventricular hypertrophy in spontaneously hypertensive rats with adriamycin nephropathy.
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Recent studies indicated pulse pressure as a risk factor for left ventricular hypertrophy, myocardial infarction, congestive heart failure and stroke as well as chronic renal failure progression. The present study examined the effects of carvedilol and its combination with captopril on blood
Adriamycin cardiomyopathy: implications of cellular changes in a canine model with mild impairment of left ventricular function.
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The present study has examined early cellular effects of chronic adriamycin administration to dogs using a protocol (1 mg/kg/week to a total cumulative dose of 240 mg/m2) producing significant but small reductions in ejection fraction and stroke volume as determined echocardiographically prior to
"New" causes of heart disease and stroke.
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I have discussed 10 "new" diseases. Some produce heart disease, some produce strokes, and some may produce both heart disease and stroke. It is of great interest to me that some of them are due not to nature's ravages but to the ingestion of agents such as toxic oil, L-tryptophan, ergot, adriamycin,
Protective effects of O-(beta-hydroxyethyl)-rutosides (HR) against adriamycin-induced toxicity in rats.
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Adriamycin (Adriablastine), administered weekly at the dose of 5 mg/kg i.p. for 3 weeks in rats, produced a general decrease of vitality associated with a decrease of body weight, hypothermia, decreases of stroke volume and cardiac output. Hematocrit was decreased. Renal blood flow decreased whereas
Noninvasive monitoring of adriamycin cardiotoxicity by "Sphygmo-Recording" of the pulse wave delay (QKd interval).
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Eighty patients with Ewing's sarcoma, bronchogenic carcinoma, and other neoplasms receiving adriamycin were monitored by a Sphygmo-Recording of the pulse wave delay (QKd time interval). The QKd interval, which is the sum of the cardiac pre-ejection period and the pulse transmission time, is
Beneficial effects of fenoldopam on systemic and regional hemodynamics in rabbits with congestive heart failure.
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Fenoldopam (SKF 82526 J) is a selective DA-1 receptor agonist and thus of a potential benefit for promoting afterload reduction, renal vasodilatation, and diuresis in congestive heart failure. To examine the acute effects of fenoldopam in heart failure, studies were performed in control rabbits (n =
Clinical significance of bifid T waves.
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An attempt was made to analyze bifid T waves which appeared in different clinical conditions. Bifid T waves occurred in 16% of 600 normal children, 92% of 37 cases of childhood ventricular septal defect (VSD), 6 of 10 cases of tetralogy of Fallot (children) and 33% of 193 patients with
Multiple admissions to the coronary care unit due to falsely elevated cardiac troponin.
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The measurement of cardiac troponin, released from injured cardiomyocytes, is of paramount importance in the diagnosis of acute myocardial infarction. Elevated troponin can be encountered, however, in patients with cardiomyopathy, significant cardiac arrhythmias, vasculitis, right-sided heart
Regional distribution of the cardiac output and renal responses to atrial natriuretic peptide infusion in rabbits with congestive heart failure.
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1. A biventricular, low-output congestive cardiomyopathy was induced in 19 rabbits by administering adriamycin (16 mg/kg). The effects of alpha-rat atrial natriuretic peptide (ANP) infused at 0.1, 0.2 and 0.4 micrograms/kg per min, were then examined in terms of (i) central haemodynamics (ii)
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