allopurinol/pendarahan

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The protective action of allopurinol in an experimental model of haemorrhagic shock and reperfusion.

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Haemorrhagic shock was induced in anaesthetized, open-chest dogs by controlled arterial bleeding, sufficient to reduce and maintain mean arterial blood pressure at 40 mmHg for 30 min. The blood volume was then restored to the pre-shock level by rapid, intravenous reinfusion of the blood shed during

The effect of allopurinol and catalase on cardiovascular hemodynamics during hemorrhagic shock.

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The primary objective of this study was to determine the effect that the xanthine oxidase inhibitor allopurinol (ALLO) and the hydrogen peroxide scavenger catalase (CAT) have on the cardiovascular compensatory ability of the dog to respond to severe hemorrhagic hypotension. Twenty-four mongrel dogs

Allopurinol plus standard resuscitation preserves hepatic blood flow and function following hemorrhagic shock.

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To determine the contribution of ischemia-reperfusion injury (IRI) to the blood flow deficit and hepatocellular dysfunction seen after resuscitation from hemorrhagic shock, the xanthine oxidase inhibitor allopurinol was given to rats as a 50 mg/kg bolus after shock but before resuscitation and

Effect of allopurinol on venous endothelial dysfunction after resuscitated hemorrhagic shock.

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BACKGROUND Blood flow deficits contribute to organ dysfunction in patients resuscitated from hemorrhage. OBJECTIVE To determine the contribution of xanthine oxidase mediated reperfusion injury to venous endothelial function after resuscitated hemorrhagic shock. METHODS Rats were prepared for

Histamine release and SOD, allopurinol and ranitidine pretreatment in haemorrhagic shock in the rat.

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Histamine release have been demonstrated in haemorrhagic shock. There are some observations that oxygen free radicals can cause histamine release. Oxygen free radicals play a role in the pathogenesis of gastric mucosal lesions. The goal of this study was to determine whether ranitidine or SOD and

Superoxide dismutase and allopurinol improve survival in an animal model of hemorrhagic shock.

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We studied the efficacy of resuscitation with antioxidants in an animal model of hemorrhagic shock. Male Sprague-Dawley rats were anesthetized, and 27 mL/kg of blood was withdrawn from the carotid artery over 2 minutes. The animals remained in hemorrhagic shock for 45 minutes, followed by 1 hour of

[The possibility of superoxide-production during hemorrhagic shock observing the effect of allopurinol on the level of serum lipidperoxide and kynurenine (author's transl)].

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Effect of allopurinol on hemorrhagic shock.

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Protection against irreversible hemorrhagic shock by allopurinol.

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Allopurinol protection of hepatic nuclear function during hemorrhagic shock.

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Blood glucose levels in dogs pretreated with allopurinol during hemorrhagic shock.

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Effect of allopurinol and/or catalase on hemorrhagic shock and their potential application to multiple organ harvesting.

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Effects of allopurinol on hepatic adenosine nucleotides in hemorrhagic shock.

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Risk Factors for Androgen Deficiency with Daily Opioid Use; Co-trimoxazole and Sudden Cardiac Death in Patients Receiving ACE Inhibitors; Clindamycin-Induced Myelosuppression; Apixaban-Induced Diffuse Alveolar Hemorrhage; DRESS Syndrome Induced by Allopurinol.

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The purpose of this feature is to heighten awareness of specific adverse drug reactions (ADRs), discuss methods of prevention, and promote reporting of ADRs to the US Food and Drug Administration's (FDA's) MedWatch program (800-FDA-1088). If you have reported an interesting, preventable ADR to

Ascorbate preserves gastric mucosal metabolism and microcirculation after hemorrhagic shock and retransfusion in rats.

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The gastric mucosal microcirculation and purine nucleotide metabolism were studied in rats after hemorrhagic shock and retransfusion. The mucosal surface density of perfused vessels (SDPV) and the mucosal levels of ATP, ADP, AMP, IMP, hypoxanthine and uric acid were measured following 15 min of

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