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amnesia/atrofi

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Evaluation of a neural network model of amnesia in diffuse cerebral atrophy.

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A digital computer program generating a simulated neural network was used to construct a model which can show behaviour resembling human associative memory. The experimental network uses distributed storage, and, in this respect, its functional organisation resembles that suggested by reported

[Pure progressive amnesia, isolated for 16 years with focal hippocampus atrophy].

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BACKGROUND Pure progressive amnesia is a rare and unusual syndrome involving long preservation of autonomy and absence of progression in other cognitive domains. METHODS We report a case which remained quiescent for 16 years characterized by severe isolated episodic amnesia and preservation of

Magnetic resonance volumetry reveals focal brain atrophy in transient epileptic amnesia.

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Transient epileptic amnesia (TEA) is a recently described epilepsy syndrome characterized by recurrent episodes of isolated memory loss. It is associated with two unusual forms of interictal memory impairment: accelerated long-term forgetting (ALF) and autobiographical amnesia. We investigated the

Severe anterograde amnesia with extensive hippocampal degeneration in a case of rapidly progressive frontotemporal dementia.

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Frontotemporal dementia (FTD) is usually characterized as a spectrum of relatively slowly progressive disorders with largely focal frontal or temporal presentations. The development of clinical and research criteria for discriminating FTD from Alzheimer's disease has relied, in part, on the relative

Transient epileptic amnesia: regional brain atrophy and its relationship to memory deficits.

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Transient epileptic amnesia (TEA) is a recently recognised form of epilepsy of which the principle manifestation is recurrent, transient episodes of isolated memory loss. In addition to the amnesic episodes, many patients describe significant interictal memory difficulties. Performance on standard

Hippocampal atrophy is the critical brain change in patients with hypoxic amnesia.

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Anoxia is considered a good model for studying amnesia. However, not all individuals who experience anoxic events develop memory problems. Moreover, the question still remains about whether, after anoxia, damage is limited to the hippocampus in patients with amnesia and without other significant

Post-traumatic amnesia predicts long-term cerebral atrophy in traumatic brain injury.

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OBJECTIVE To examine post-traumatic amnesia (PTA) and its relation to long-term cerebral atrophy in persons with traumatic brain injury (TBI) using objective indicators of PTA duration and Quantitative Magnetic Resonance Imaging (QMRI). It was hypothesized that longer PTA would predict later

Transient Global Amnesia Deteriorates the Network Efficiency of the Theta Band.

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Acute perturbation of the hippocampus, one of the connector hubs in the brain, is a key step in the pathophysiological cascade of transient global amnesia (TGA). We tested the hypothesis that network efficiency, meaning the efficiency of information exchange over a network, is impaired during the

Developmental amnesia and its relationship to degree of hippocampal atrophy.

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Two groups of adolescents, one born preterm and one with a diagnosis of developmental amnesia, were compared with age-matched normal controls on measures of hippocampal volume and memory function. Relative to control values, the preterm group values showed a mean bilateral reduction in hippocampal

Amnesia, cerebral atrophy, and autoimmunity.

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Does hippocampal atrophy explain anterograde and retrograde amnesia following autoimmune limbic encephalitis?

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[Clinical study of Pyribedil in syndromes of intellectual deterioration with amnesia].

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Polygalae Radix Extract Prevents Axonal Degeneration and Memory Deficits in a Transgenic Mouse Model of Alzheimer's Disease.

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Memory impairments in Alzheimer's disease (AD) occur due to degenerated axons and disrupted neural networks. Since only limited recovery is possible after the destruction of neural networks, preventing axonal degeneration during the early stages of disease progression is necessary to prevent AD.

Network-wide abnormalities explain memory variability in hippocampal amnesia.

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Patients with hippocampal amnesia play a central role in memory neuroscience but the neural underpinnings of amnesia are hotly debated. We hypothesized that focal hippocampal damage is associated with changes across the extended hippocampal system and that these, rather than hippocampal atrophy per

Growing up with bilateral hippocampal atrophy: from childhood to teenage.

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The respective roles of the medial temporal lobe (MTL) structures in memory are controversial. Some authors put forward a modular account according to which episodic memory and recollection-based processes are crucially dependent on the hippocampal formation whereas semantic acquisition and
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