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Effects of caffeine and ryanodine on delayed afterdepolarizations and sustained rhythmic activity in 1-day-old myocardial infarction in the dog.
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Caffeine and ryanodine are known to modulate oscillatory release of Ca2+ from the sarcoplasmic reticulum. The effects of caffeine and ryanodine on delayed afterdepolarizations (DADs) and sustained rhythmic activity in subendocardial Purkinje fibers surviving 1-day-old myocardial infarction in the
Action potentials of cardiac muscle in healing infarcts: response to norepinephrine and caffeine.
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Previous studies have shown that the muscle fibers that survive on the epicardial surface of a 5- or 14-day-old infarct in canine hearts have reduced or absent plateau phase during repolarization. Since the absence of a plateau phase could be related to a decrease in the slow inward current, we
Caffeine as a possible cause of ventricular arrhythmias during the healing phase of acute myocardial infarction.
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Caffeine (300 mg) was administered to each of 70 patients a mean (+/- standard error of the mean) of 7 +/- 1 days after the onset of acute myocardial infarction to determine its effects on ventricular arrhythmias. The study was designed as a randomized, double-blind, within-patient comparison
Coronary precautions: should caffeine be restricted in patients after myocardial infarction?
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The effect of caffeine on heart rate, blood pressure, and cardiac rhythm has long been a controversial issue. A review of the literature reveals numerous articles evaluating the effects of caffeine on normal subjects, fewer evaluating the effects of caffeine on patients with cardiac disease and
Myocardial infarction resulting from caffeine overdose in an anorectic woman.
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A 20-year-old bulimic woman ingested 20 g of caffeine in a suicide attempt. After being evaluated and discharged from the emergency department, she was readmitted with ECG changes and ultimately found to have sustained a subendocardial infarction. This case highlights the wide-ranging health
Caffeine Restores Background EEG Activity Independent of Infarct Reduction after Neonatal Hypoxic Ischemic Brain Injury
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In human preterm newborns, caffeine increases brain activity and improves neurodevelopmental outcomes. In animal models of hypoxic ischemic brain injury, caffeine pretreatment reduces infarct volume. We studied the relationship between tissue neuroprotection and brain activity after injury to
[Sleep disturbances in multi-infarction dementia and trials of treatment with caffeine].
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Atherosclerotic cerebrovascular lesions leading to repeated ischaemic strokes produce a wealth of clinical symptoms and signs with dementia. Sleep disturbances are frequent and take the form of difficult falling asleep and shifting of the sleep/waking rhythm to late night and morning hours. Hypnotic
Effect of caffeine on the ventricular fibrillation threshold in normal dogs and dogs with acute myocardial infarction.
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Effects of caffeine ingestion on heart rate, blood pressure, myocardial oxygen consumption, and cardiac rhythm in acute myocardial infarction patients.
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Combination of low dose ethanol and caffeine protects brain from damage produced by focal ischemia in rats.
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Caffeine and ethanol are two commonly overused psychoactive dietary components. The purpose of this study was to assess the effects of acute, chronic, oral (p.o.) and intravenous (i.v.) caffeine, ethanol and their combination on infarct volume following focal ischemia in rats. Rats received
Ethanol plus caffeine (caffeinol) for treatment of ischemic stroke: preclinical experience.
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OBJECTIVE
Ethanol and caffeine are 2 common psychoactive dietary components. We have recently shown that low-dose ethanol plus caffeine results in a 70% to 80% reduction of infarct volume after reversible common carotid/middle cerebral artery (CCA/MCA) occlusion in rats. The combination (caffeinol)
Acute effects of caffeine on heart rate variability in habitual caffeine consumers.
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During the last years, heart rate variability (HRV) has become a promising risk factor for cardiovascular events. However, the effect of caffeine on HRV in habitual caffeine consumers has barely been investigated. Therefore, we treated 30 male habitual caffeine users in a randomized double-blinded
High dose caffeine and ventricular arrhythmias.
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The effect of high dose caffeine on ventricular arrhythmias was examined in 35 patients with recent myocardial infarctions. All patients received caffeine 450 mg or placebo on separate days using a randomized double-blind study design. Continuous Holter electrocardiographic recordings were performed
Incidence of caffeine in serum of patients undergoing dipyridamole myocardial perfusion stress test by an intensive versus routine caffeine history screening.
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The coronary vasodilatory effect of dipyridamole is competitively blocked by caffeine. The purposes of this study were to (1) assess the incidence of having detectable serum caffeine and (2) evaluate whether an intensive caffeine history screening strategy was superior to routine history screening
Cardiovascular lesions in Sprague-Dawley rats induced by long-term treatment with caffeine.
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Male Sprague-Dawley rats were treated with phenacetin, phenazone or caffeine in the diet or with combinations of these chemicals for up to 117 weeks. Twenty of twenty-eight rats receiving caffeine only developed moderate to severe myocardial fibrosis compared with five control rats, in spite of the
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