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carotene/infark

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Beta-carotene intake and risk of nonfatal acute myocardial infarction in women.

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There are indications that beta-carotene, but not pre-formed vitamin A, is protective on the risk of acute myocardial infarction (AMI). The relationship between nonfatal AMI and the intake of beta-carotene and retinol was investigated in a case-control study conducted between 1983 and 1992 in

Role of lipoperoxidation in the remodeling intensification induced by beta-carotene after infarction.

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BACKGROUND The mechanisms involved in the biggest remodeling caused by the post-infarct beta-carotene are unknown. OBJECTIVE To analyze the role of lipoperoxidation in the ventricular remodeling after infarct of the myocardium in rats supplemented with beta-carotene. METHODS Rats were infarcted and

Effects of beta-carotene and smoking on heart remodeling after myocardial infarction.

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OBJECTIVE To analyze the effects of beta-carotene on the ventricular remodeling process following myocardial infarction (MI) in rats exposed to cigarette smoke. METHODS After acute myocardial infarction (AMI), the animals were divided into four groups: 1) Group C, 24 animals that were given standard

Low serum lycopene and β-carotene increase risk of acute myocardial infarction in men.

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OBJECTIVE Previous studies have shown that high intake or concentrations of serum carotenoids may protect against acute myocardial infarction (AMI). The role of carotenoids on the risk of AMI remains inconsistent. The aim of the present study was to examine if serum concentrations of major
Because antioxidants may play a role in the prevention of coronary heart disease by inhibiting the peroxidation of polyunsaturated fatty acids (PUFAs), the combined association of diet-derived antioxidants and PUFAs with acute myocardial infarction (MI) was investigated. This multicenter
The objective of this study was to investigate the effects of exposure to tobacco smoke (ETS) in rats that were or were not supplemented with dietary beta-carotene (BC), on ventricular remodeling and survival after myocardial infarction (MI). Rats (n = 189) were allocated to 4 groups: the control
BACKGROUND Epidemiological data suggest that the intake of antioxidants such as alpha-tocopherol (vitamin E) and beta-carotene has an inverse correlation with the incidence of coronary heart disease. The results from clinical trials of antioxidant supplementation in people with known coronary heart

Beta-carotene supplementation results in adverse ventricular remodeling after acute myocardial infarction.

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OBJECTIVE We studied the effects of beta-carotene (BC) on ventricular remodeling after myocardial infarction. METHODS Myocardial infarction was induced in Wistar rats that were then treated with a BC diet (500 mg/kg of diet per day; MI-BC; n = 27) or a regular diet (MI; n = 27). Hearts were analyzed
BACKGROUND Oxidized low-density lipoprotein is involved in the pathogenesis of atherosclerosis. In epidemiological studies antioxidants have been inversely related with coronary heart disease. Findings from controlled trials are inconclusive. METHODS We studied the primary preventive effect of

Trans-fatty acids intake and risk of myocardial infarction.

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BACKGROUND Concern that trans-fatty acids formed in the partial hydrogenation of vegetable oils may increase the risk of coronary disease has existed for several decades, but direct evidence on this relation in humans is limited. RESULTS With a case-control design, we studied the association between
The Physicians' Health Study is a randomized, double-blind, placebo-controlled trial of primary prevention designed to assess the effects of low-dose aspirin on cardiovascular disease and of beta-carotene on risks of cancer. A total of 22,071 U.S. male physicians 40 to 84 years of age were

Plasma antioxidants and lipid peroxidation in acute myocardial infarction and thrombolysis.

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OBJECTIVE The aim of this study was to investigate the balance between prooxidative and protective mechanisms in patients with acute myocardial infarction (AMI) throughout streptokinase (STK) therapy. METHODS Patients who presented to coronary care unit within 3 hours of infarction were followed.

Randomized, double-blind placebo-controlled trial of coenzyme Q10 in patients with acute myocardial infarction.

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The effects of oral treatment with coenzyme Q10 (120 mg/d) were compared for 28 days in 73 (intervention group A) and 71 (placebo group B) patients with acute myocardial infarction (AMI). After treatment, angina pectoris (9.5 vs. 28.1), total arrhythmias (9.5% vs. 25.3%), and poor left ventricular

[Diet and risk of myocardial infarction. A case-control community-based study].

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OBJECTIVE To evaluate diet as a risk factor for myocardial infarction. METHODS Community based case-control study. METHODS University Hospital, Oporto. METHODS First time consecutive cases of acute myocardial infarction (n = 100) and 198 community controls, older than 39 years and living in Oporto,

Plasma carotenoids and risk of acute myocardial infarction in the Singapore Chinese Health Study.

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OBJECTIVE Modification of low-density lipoprotein due to oxidative stress is essential in the development of coronary atherosclerosis. Data of specific carotenoids except β-carotene on cardioprotective effects in humans are limited. RESULTS This study examined the associations between plasma
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