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coronary thrombosis/nikotin

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Effects of cigarette smoke and nicotine on platelets and experimental coronary artery thrombosis.

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The causal link between smoking, atherosclerosis and an increased risk for acute platelet mediated coronary events such as acute platelet thrombus formation, myocardial infarction, and sudden coronary death is not clear. Our studies suggest that there may be a transient increase in in vivo platelet

Smoking and cardiovascular health: a review of the epidemiology, pathogenesis, prevention and control of tobacco.

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The causal associations between cigarette smoking and human diseases are irrefutable. In this review, we focus on the epidemiological pattern of cigarette smoking on cardiovascular risk, the underlying mechanistic process of such a causal link, how to prevent premature cardiovascular morbidity and

Smoking Cessation Strategies.

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Cigarette smoking is a substantial social and public health problem contributing to about half of the 400,000 cardiovascular deaths in the United States. Nicotine and carbon monoxide are the components in cigarette smoke that contribute to cardiovascular morbidity and mortality. Nicotine is a

Myocardial infarction with normal coronary arteriogram: the role of ephedrine-like alkaloids.

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BACKGROUND The overwhelming majority of myocardial infarctions result from atherosclerosis, generally with superimposed coronary thrombosis. The prevalence of patients who develop myocardial infarction and in whom subsequent angiography shows normal coronary arteries is approximately 5%. Many of

[Adverse cardiac effects of smoking].

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Cigarette smoking is a main risk-factor for enhanced cardiovascular morbidity and mortality. Some studies have even suggested that unvoluntary smoking increases the atherosclerotic risk. Smoking related cardiovascular diseases include coronary heart disease, acute myocardial infarction, sudden

[Myocardial infarction in the adult under 35 years of age].

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Infarction in the adult aged less than 35 is almost always accompanied by risk factors, the most common of which are tobacco abuse, lipid disturbances, obesity, a strong family history of cardiovascular disease and, in the woman, the use of contraceptives. The coronary arteries are sometimes normal

Third-hand Smoke: Impact on Hemostasis and Thrombogenesis.

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Cigarette smoking is a major risk factor for acute coronary thrombosis. In fact, both active/first-hand smoke and passive/second-hand smoke exposure are known to increase the risk of coronary thrombosis. Although recently a new risk has been identified and termed third-hand smoke (THS), which is the

A Left Main Coronary Artery Thrombus Presenting as a Non St Elevation MI.

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BACKGROUND Left main coronary artery (LMCA); thrombus with an acute myocardial infarction identified with coronary angiography is a clinically rare condition with an extremely high mortality rate. We present a case of LMCA thrombus that presented as a non-ST elevation myocardial infarction

[Myocardial infarction related to the practice of sports. clinical and coronary angiography apropos of 35 cases].

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The clinical and coronary angiographic features of 35 patients who survived the acute phase of a myocardial infarction that occurred while they were practising sports were studied. 97 p. 100 of the patients were male. Mean age was 42.8 years (range 26-71 years), 60 p. 100 being over 40. All but 2

Predictors of long-term adverse outcomes in patients with congenital coronary artery fistulae.

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BACKGROUND Significant morbidities, including angina, symptomatic heart failure, and myocardial infarction, have been reported after coronary artery fistula (CAF) closure; however, predictors that may be associated with adverse outcomes have not been established. The goal of this investigation is to
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