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The ultrastructure and reversibility of testicular atrophy induced by ethylene glycol monomethyl ether (EGME) in the rat.
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Inhalation exposure to 300 ppm ethylene glycol monomethyl ether (EGME) for 3 days produced degenerative changes in spermatocytes of pachytene and meiotic division at spermatogenic stage XIV in rats. However, a wide range of germ cell types including spermatogonia was affected and the stage-specific
Ethylene oxide induces central-peripheral distal axonal degeneration of the lumbar primary neurones in rats.
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Wistar rats subjected to a single exposure lasting six hours to ethylene oxide (EO) at a concentration of 500 parts per million three times a week for 13 weeks developed ataxia in the hindlegs. Myelinated fibres in hindleg nerves and in the fasciculus gracilis showed axonal degeneration sparing the
[Testicular atrophy of mice induced by ethylene glycol mono alkyl ethers (author's transl)].
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Toxicities of ethylene glycol (EG) and 6 ethylene glycol mono alkyl ethers administered orally were studied. Mice were given various doses (62.5, 125, 250, 500, 1,000, 2,000 and 4,000 mg/kg body weight) of the compounds daily for 5 days/week, for 5 weeks. High doses of ethylene glycol mono methyl
Decreased formation of ethoxyacetic acid from ethylene glycol monoethyl ether and reduced atrophy of testes in male rats upon combined administration with toluene and xylene.
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Male painters are commonly exposed to ethylene glycol monoethyl ether (EGE), a well known reproductive toxic agent causing testicular atrophy, in the form of solvent mixture containing toluene (TOL) and xylene (XYL). This study was carried out to determine the effect of exposing male rats to solvent
Anther-specific expression of mutated melon ethylene receptor gene Cm-ERS1/H70A affected tapetum degeneration and pollen grain production in transgenic tobacco plants.
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To develop a new system for inducible male sterility without any modification of the floral architecture in tobacco plants, a mutated ethylene receptor gene Cm-ERS1/H70A was fused either to the tobacco Nin88 promoter known to function mainly in the tapetum and microspore or to the CaMV 35S promoter
Exposure to glyphosate- and/or Mn/Zn-ethylene-bis-dithiocarbamate-containing pesticides leads to degeneration of γ-aminobutyric acid and dopamine neurons in Caenorhabditis elegans.
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Previous studies demonstrate a positive correlation between pesticide usage and Parkinson's disease (PD), which preferentially targets dopaminergic (DAergic) neurons. In order to examine the potential relationship between two common pesticides and specific neurodegeneration, we chronically (24 h) or
Vibrational quasi-degenerate perturbation theory with optimized coordinates: applications to ethylene and trans-1,3-butadiene.
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A perturbative extension to optimized coordinate vibrational self-consistent field (oc-VSCF) is proposed based on the quasi-degenerate perturbation theory (QDPT). A scheme to construct the degenerate space (P space) is developed, which incorporates degenerate configurations and alleviates the
Oral Reference Dose for ethylene glycol based on oxalate crystal-induced renal tubule degeneration as the critical effect.
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Several risk assessments have been conducted for ethylene glycol (EG). These assessments identified the kidney as the primary target organ for chronic effects. None of these assessments have incorporated the robust database of species-specific toxicokinetic and toxicodynamic studies with EG and its
Mode of action: oxalate crystal-induced renal tubule degeneration and glycolic acid-induced dysmorphogenesis--renal and developmental effects of ethylene glycol.
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Ethylene glycol can cause both renal and developmental toxicity, with metabolism playing a key role in the mode of action (MOA) for each form of toxicity. Renal toxicity is ascribed to the terminal metabolite oxalic acid, which precipitates in the kidney in the form of calcium oxalate crystals and
[Evaluation of testicular damage by flow cytometry: testicular atrophy caused by ethylene oxide].
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The new method using flow cytometry was applied to analyse the testicular toxicity of ethylene oxide, and the usefulness of this method is discussed. When Wistar male rats were exposed to ethylene oxide for six hours a day, three times a week for six weeks, the testicular weights of the exposed
Induction of water-soaking deterioration in watermelon fruit by ethylene.
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Watermelon (Citrullus lanatus Thunb. Mansfeld) fruit were held in 50 microL/L of ethylene at 20 degrees C following 18 h application with 5 microL/L of 1-methylcyclopropene (1-MCP) to investigate the involvement of ethylene in the regulation of water-soaking. The early responses of watermelon fruit
Mechanistic studies on 14-electron ruthenacyclobutanes: degenerate exchange with free ethylene.
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The phosphonium alkylidene [(NHC)Cl2Ru=CH(PCy3)]+[B(C6F5)4]-, 1, (NHC = N-heterocyclic carbene, Cy = cyclohexyl, C6H11) reacts with 2.2 equiv of ethylene at -50 degrees C to form the 14-electron ruthenacyclobutane (NHC)Cl2Ru(CH2CH2CH2), 2. NMR spectroscopic data indicates that 2 has a C2v symmetric
INHIBITORS OF HEATING AND DETERIORATION IN SEEDS. I. ETHYLENE CHLORHYDRIN AND RELATED COMPOUNDS.
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Computational insights into degenerate ethylene exchange with a Grubbs-type catalyst.
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Testicular atrophy from inhalation of ethylene oxide cyclic tetramer.
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