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hyponatremia/hypoxia
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Symptomatic hyponatremia with hypoxia is a medical emergency.
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Patients presenting with the combined finding of severe symptomatic hyponatremia and hypoxia have such high mortality rates that they should be admitted to an intensive care unit and intubated sooner rather than later. Without delay, these patients need rapid correction of their serum sodium by 8-10
Hypoxia is the cause of brain damage in hyponatremia.
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Effect of hypoxia on the cerebral adaptation to acute hyponatremia in experimental animals.
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Operative Hysteroscopy Intravascular Absorption Syndrome Causing Hyponatremia with Associated Cerebral and Pulmonary Edema.
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Operative hysteroscopy intravascular absorption syndrome is an iatrogenic syndrome caused by absorption of hypo-osmolar distension medium during hysteroscopy, which can lead to rapid hyponatremia with resulting cerebral and pulmonary edema. We present a case of a 47-year-old female who underwent
Cortical laminar necrosis caused by rapidly corrected hyponatremia.
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Cortical laminar necrosis (CLN) is radiologically characterized by hyperintense cortical lesions on unenhanced T1-weighted images. Hypoxia is the representative cause of CLN; however, the rapid correction of hyponatremia has also been suggested as another possible cause. We present a patient who
Metastasis to the colon from lung cancer presenting with severe hyponatremia and dyspnea in a young male: A case report and review of the literature.
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This study aimed to present the atypical clinical presentation and management of a metastatic lung cancer that had spread to an atypical location. Lung cancer is the most common cause of cancer-related mortality worldwide. The brain, liver, adrenal glands and bone are the most common sites of
Central and extrapontine myelinolysis in a patient in spite of a careful correction of hyponatremia.
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We report the case of a 54-year-old alcoholic female patient who was hospitalized for neurologic alterations along with a severe hyponatremia (plasma Na+: 97 mEq/l). She suffered from potomania and was given, a few days before admission, a thiazide diuretic for hypertension. A careful correction of
Miliary tuberculosis presenting with hyponatremia and thrombocytopenia.
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A 74-year-old woman with miliary tuberculosis had moderately severe hyponatremia due to inappropriate secretion of antidiuretic hormone (SIADH) and very severe thrombocytopenia without other hematologic abnormalities. She was treated with isoniazid, rifampin, ethambutol, prednisone, vincristine and
Plasma-electrolytes in natives to hypoxia after marathon races at different altitudes.
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OBJECTIVE
It is well known that altitude natives differ from sea level natives in aspects of fluid and electrolyte homeostasis.
METHODS
To evaluate exercise and environmental influences on the electrolyte and water status in hypoxia adapted subjects, we investigated 11 well-trained marathon runners
Hyponatremia: an update on current pharmacotherapy.
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BACKGROUND
Hyponatremia is the most common electrolyte disorder in clinical practice, and it is associated with adverse outcomes. Severe hyponatremia can result in cerebral edema and hypoxia. Moreover, even mild hyponatremia can lead to gait instability and cognitive dysfunction, especially in the
Hypoxic and ischemic hypoxia exacerbate brain injury associated with metabolic encephalopathy in laboratory animals.
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Hypoxemia is a major comorbid factor for permanent brain damage in several metabolic encephalopathies. To determine whether hypoxia impairs brain adaptation to hyponatremia, worsening brain edema, we performed in vitro and in vivo studies in cats and rats with hyponatremia plus either ischemic or
Acute symptomatic hyponatremia in a flight attendant.
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Acute symptomatic hyponatremia after thiazide diuretic initiation is a medical emergency. Here we describe the case of a flight attendant who developed acute hyponatremia during a flight and the potential risk factors for developing this condition. A 57-year-old flight attendant with history of
[Central pontine and extrapontine myelinolysis following rapid correction of hyponatremia--report of an autopsy case].
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A case of central pontine myelinolysis (CPM) following rapid correction of hyponatremia was reported and literatures were reviewed. The case was 61-year-old nonalcoholic female who had taken an operation of craniopharyngioma 23 years ago. Fifteen years later, she received re-operation for the
Influence of hypoxia and sex on hyponatremic encephalopathy.
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Over the past 20 years it has become increasingly apparent that hyponatremic encephalopathy is a major cause of inhospital morbidity and mortality, particularly in postoperative patients. The factors that may lead to death or permanent brain damage and the susceptible patient groups have been
Hospital-acquired hyponatremia--why are hypotonic parenteral fluids still being used?
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Hospital-acquired hyponatremia can be lethal. There have been multiple reports of death or permanent neurological impairment in both children and adults. The main factor contributing to the development of hospital-acquired hyponatremia is routine use of hypotonic fluids in patients in whom the
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