mastocytosis/hypoxia

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Respiratory distress and hypoxemia in systemic mastocytosis.

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A 25-year-old woman with documented mastocytosis developed hypoxemia with pruritus, diarrhea, headache, and hypotension on two separate occasions. The hypoxemia appeared to be related to a massive release of histamine. Resolution of the patient's symptoms was accompanied by the return of her

Parkinsonism due to bilateral basal ganglia lesions following mastocytosis-induced hypoxia.

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Hypoxia and lung mast cells: influence of disodium cromoglycate.

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Rats kept in 10% O2 for three or more weeks developed mast cell hyperplasia in the lungs, especially round the alveoli and the small peripheral blood vessels, which became thickened during chronic hypoxia. There was a significant correlation between the degree of right ventricular hypertrophy (RVH)

Lung mast cells in rats exposed to acute hypoxia, and chronic hypoxia with recovery.

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Exposure to acute hypoxia (barometric pressure 263 mmHg) for 8 hours did not lead to increased numbers of mast cells in the lungs of rats. In contrast, in adult rats kept for 35 days at a barometric pressure of 380 mmHg there was a proliferation of mast cells around the pulmonary blood vessels and

Mast cell stabilizing compound FPL 55618 reduces right ventricular hypertrophy and lung mast cell hyperplasia in chronically hypoxic rats.

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Rats treated with chronic hypobaric hypoxia (21 days, 380 Torr) and mast cell stabilizing compound FPL 55618 had significantly less right ventricular hypertrophy and lung mast cell hyperplasia than rats subjected to chronic hypoxia alone. Right ventricular blood pressure was not reduced.

Restless legs syndrome is associated with mast cell activation syndrome.

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Mast cell activation syndrome (MCAS) is an inflammatory and allergic disorder. We determined the prevalence of Restless Legs Syndrome (RLS) in MCAS since each common syndrome may be inflammatory in nature and associated with dysautonomia.MCAS subjects were

Possible contributions of mastocytosis, apoptosis, and hydrolysis in pathophysiology of randomized skin flaps in humans and guinea pigs.

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To understand better the pathophysiology of random skin flaps, randomized skin flaps of human (3 cases) and guinea pig (53 cases) were investigated. Proximal (normal), proximomedial (viable), mediodistal (between viable and necrotic parts), and distal (necrosis) locations of the skin flaps were

Severe life-threatening or disabling anaphylaxis in patients with systemic mastocytosis: a single-center experience.

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BACKGROUND Mediator-related symptoms in patients with systemic mastocytosis (SM) range from mild episodic to severe life-threatening events. METHODS We examined a series of 137 consecutive patients with mastocytosis (63 females and 74 males) referred to our center between 1988 and 2010. Almost all

Idiopathic Anaphylaxis: A Form of Mast Cell Activation Syndrome.

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Idiopathic anaphylaxis is a condition caused by paroxysmal episodes of sudden-onset multiorgan involvement variably including laryngeal edema, urticaria, bronchoconstriction, dyspnea, hypoxia, abdominal pain, nausea, vomiting, diarrhea, and hypotension. Rarely, the episodes can lead to

Cardiovascular disease and anaphylaxis.

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OBJECTIVE In severe anaphylaxis, the cardiovascular system is often heavily involved. Preexisting cardiovascular disease may therefore influence the course of anaphylaxis in a negative way. RESULTS Systemic mastocytosis and elevated baseline serum tryptase are associated with severe and fatal

Lung mast cell density and distribution in chronically hypoxic animals.

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Changes in the density and distribution of pulmonary mast cells were determined in six mammalian species exposed to hypobaric hypoxia (PB = 435 Torr) for 19-48 days. Control animals were studied at 1,600 m (PB = 635 Torr). Total lung mast cell hyperplasia was observed only in calves exposed to high

Uremic gastropathy in the dog.

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Stomachs of four dogs with uremia and four normal dogs were examined. Uremic stomachs represented four types of disease: atrophic, amyloidotic, ulcerative and necrotic gastropathy. Pathologic changes common to all uremic stomachs were expansion of the lamina propria, atrophy of gastric glands, and

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