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myxedema/sembap

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Mechanisms of edema formation in myxedema--increased protein extravasation and relatively slow lymphatic drainage.

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We assessed extravascular accumulation of albumin and fluid in primary myxedema by measuring metabolic turnover and transcapillary escape of 131I-labeled human albumin in seven patients. In the hypothyroid state, we found a low plasma volume (P less than 0.05), a reduced rate of albumin synthesis

[Myxedematous dropsy (internal myxedema)].

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Iodide myxedema with facial swelling simulating angioneurotic edema.

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[History of a so-called globular heart or of a myxedema without edema].

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Results of the examination of patients with secondary lymphedema in the period 2006-2010. Revealed that the most frequent concomitant diseases in 150 (57,6%) patients with secondary lymphostasis lower extremities is hyperthyroidism. RESULTS an increase in thyroid size, infiltrative endocrine
OBJECTIVE Pretibial myxedema (PM) is a manifestation of Graves' disease (GD). Currently, its diagnosis depends on physicians' observation and biopsy. No satisfactory, objective, and non-invasive tool is available to record and investigate lesions. Digital infrared thermal imaging (DITI) detects

Localized myxedema of the toe: a rare presentation of graves' dermopathy.

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Infiltrative dermopathy is an uncommon manifestation of Graves' disease. The most frequent location of infiltration is the lower extremities, especially the pretibial areas and on the dorsum of the foot. Rarely the hand, elbow, arm, and forearm are affected. We report a 66-year-old man with Graves'

Severe angioedema in myxedema coma: a difficult airway in a rare endocrine emergency.

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Myxedema coma is the most lethal manifestation of hypothyroidism. It is a true medical emergency and can result in profound hemodynamic instability and airway compromise. Myxedema coma currently remains a diagnostic challenge due to the rarity of cases seen today, and failure to promptly initiate

[Thyrotoxic storm and myxedema coma].

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Thyrotoxic or hyperthyroid storm is a grave, life-threatening, but relatively infrequent medical emergency. Immediate causes of death in this emergency are severe hyperpyrexia and pulmonary edema associated with arrhythmias, shock, and coma. This emergency is found in Graves' patients most

[Myxedema coma in a patient with type 1 neurofibromatosis: rare association].

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Myxedema coma, a rare but fatal emergency, is an extreme expression of hypothyroidism. We describe a 51-year-old male patient who has discontinued hypothyroidism treatment 10 months earlier and developed lethargy, edema, and cold intolerance symptoms. He also had a previous diagnosis of

Pretibial Myxedema: Case Presentation and Review of Treatment Options.

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Pretibial myxedema (PM) is a rare autoimmune manifestation of Graves' disease, which commonly presents as diffuse, nonpitting edema of shins and less often as plaques, nodules, or elephantiasis lesions mimicking lymphedema. We present a 57-year-old woman with 12-month history of PM, which occurred a

Early onset pretibial myxedema in the absence of ophthalmopathy: a morphologic evolution.

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Pretibial myxedema (PTM) is an uncommon, late manifestation of Graves' disease. It is nearly always preceded by significant ophthalmopathy and classically presents as nonpitting (brawny) edema of the lower extremities. We report a case of PTM that is unusual because of its morphologic evolution,
BACKGROUND Pretibial myxedema (PTM) is a rare manifestation of Graves' disease. There is paucity of data regarding long-term follow-up and response to treatment in PTM. METHODS Retrospective study wherein 30 patients of PTM presenting during 2001-2011 attending dermatology and endocrinology
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