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Effects of ouabain on early ventricular relaxation in patients with acute myocardial infarction.
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The effects of the administration of ouabain on the peak negative left ventriculat (LV) dP/dt were studied in 14 patients with acute myocardial infarction. Prior to administration of ouabain, negative LV dP/at was lower than previously reported for patients without coronary arterial disease. One
Blockade of brain 'ouabain' prevents the impairment of baroreflexes in rats after myocardial infarction.
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BACKGROUND
The purpose of this study was to test whether increased brain "ouabain" contributes to impairment of both arterial and cardiopulmonary baroreceptor reflexes in congestive heart failure (CHF).
RESULTS
Two to 5 days after coronary artery ligation (MI) or sham surgery in male Wistar rats,
Enhancement of triggered activity in ischemic Purkinje fibers by ouabain: a mechanism of increased susceptibility to digitalis toxicity in myocardial infarction.
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Enhanced susceptibility to toxic arrhythmias by digitalis administration has been reported in clinical and experimental myocardial infarction. To investigate the mechanism responsible for this phenomenon, the effects of superfusion with normal Tyrode's solution and superfusion with Tyrode's solution
Brain "ouabain" and angiotensin II contribute to cardiac dysfunction after myocardial infarction.
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In chronic heart failure (CHF), sympathetic activity increases in parallel with the impairment of left ventricle (LV) function, and sympathetic hyperactivity has been postulated to contribute to the progression of heart failure. In the brain, compounds with ouabain-like activity ("ouabain," for
[Attempts at infarct prevention using orally administered ouabain (g-Strophantin)].
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[Attempts at prevention of infarct using orally administered ouabain (strophantine G)].
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Hemodynamic effects of ouabain in experimental acute myocardial infarction with shock.
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Effects of ouabain on the electrophysiological properties of subendocardial Purkinje fibers surviving in regions of acute myocardial infarction.
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[Left ventricular function in convalescent subjects with myocardial infarct: effects of ouabain].
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Effects of ouabain on impaired left ventricular function in acute myocardial infarction.
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Effects of ouabain on impaired left ventricular function during convalescence after acute myocardial infarction.
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Hemodynamic effects of ouabain in patients with acute myocardial infarction studies with an electromagnetic velometer.
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Augmentation of Ouabain-Induced Increase in Heart Muscle Contractility by Akt Inhibitor MK-2206.
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Cardiac steroids (CSs), such as ouabain and digoxin, increase the force of contraction of heart muscle and are used for the treatment of congestive heart failure (CHF). However, their small therapeutic window limits their use. It is well established that Na+, K+-ATPase inhibition mediates CS-induced
Ouabain induces nitric oxide release by a PI3K/Akt-dependent pathway in isolated aortic rings from rats with heart failure.
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BACKGROUND
Ouabain occurs in nanomolar concentrations in myocardial infarction and heart failure (HF). However, the effects of ouabain in vascular function in HF conditions were not investigated yet. Therefore, we analyzed the effects of acute administration of 3 nM ouabain in isolated aortic rings
Ouabain--a link in the genesis of high blood pressure?
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Hypertension or high blood pressure is a risk factor that increases risk of myocardial infarction, renal failure or cerebral stroke. The pathogenesis of hypertension is due to a variety of causes, including inherited predisposition, dietary habits, especially salt intake, smoking, and also 'general
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