prolapse/tyrosine

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[ON THE DISTRIBUTION OF TYROSINE C-14 IN THE RAT BRAIN IN THE PRESENCE OF TRANSPLANTABLE BRAIN TUMORS AND ITS ACUTE PROLAPSE].

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Neurovascular alterations of muscularis propria in the human anterior vaginal wall in pelvic organ prolapse.

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In the pathophysiology and progression of pelvic organ prolapse (POP), it has been demonstrated that there is a reorganisation of the muscularis propria of the anterior vaginal wall due to a phenotypic smooth muscle cell to myofibroblast switch. An abnormal deposition of collagen type III seems to

A clinicoanatomical study of the novel nerve fibers linked to stress urinary incontinence: the first morphological description of a nerve descending properly along the anterior vaginal wall.

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When performing anterior colporrhaphy for cystocele, most pelvic surgeons have not considered the neuroanatomy that contributes to urethral function. The aim of the study was to anatomically identify nerve fibers located in the anterior vagina associated with the pathogenesis of incontinence and

c-Abl regulates gastrointestinal muscularis propria homeostasis via ERKs.

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The gastrointestinal tract is responsible for food digestion and absorption. The muscularis propria propels the foodstuff through the GI tract and defects in intestine motility may cause obstruction disorders. Our present genetic studies identified non-receptor tyrosine kinase c-Abl as an important

Systemic and topical hormone therapies reduce vaginal innervation density in postmenopausal women.

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OBJECTIVE Menopause is often accompanied by vaginal discomfort including burning, itching, dryness, and spontaneous or provoked pain. Although the direct effects of estrogen withdrawal on vaginal cells are implicated, surgical menopause in rodents causes autonomic and sensory nerves to proliferate,

Perioperative management of patient with alkaptonuria and associated multiple comorbidities.

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Alkaptonuria is a rare inherited genetic disorder of tyrosine metabolism characterized by a triad of homogentisic aciduria, ochronosis, and arthritis. The most common clinical manifestations of ochronosis involve the musculoskeletal, respiratory, airway, cardiovascular, genitourinary, cutaneous, and

MVP-Associated Filamin A Mutations Affect FlnA-PTPN12 (PTP-PEST) Interactions.

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Although the genetic basis of mitral valve prolapse (MVP) has now been clearly established, the molecular and cellular mechanisms involved in the pathological processes associated to a specific mutation often remain to be determined. The FLNA gene (encoding Filamin A; FlnA) was the first gene

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