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pseudoephedrine/infark
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Coronary artery spasm and myocardial infarction in a patient with normal coronary arteries: temporal relationship to pseudoephedrine ingestion.
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A 28 yr-old male presented with chest pain and acute ST elevation following ingestion of pseudoephedrine. The pain and electrocardiographic changes disappeared after the administration of sublingual Nitroglycerin. Myocardial enzymes did show some evidence for myocardial necrosis. A subsequent
Acute Myocardial Infarction from Coronary Vasospasm Precipitated by Pseudoephedrine and Metoprolol Use.
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Pseudoephedrine is a sympathomimetic α- and β-adrenergic receptor agonist that causes vasoconstriction and reduction in edema throughout the nasal passages. Coronary vasospasm associated with pseudoephedrine has been reported in the literature. We discuss the case of a patient with new-onset atrial
Acute myocardial infarction after over-the-counter use of pseudoephedrine.
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Pseudoephedrine is a commonly used over-the-counter decongestant with sympathomimetic activity. We present the case of a previously healthy young man who had an acute myocardial infarction 45 minutes after ingesting the recommended dose of an over-the-counter cold remedy containing pseudoephedrine.
Myocardial infarction associated with pseudoephedrine use and acute streptococcal infection in a boy with normal coronary arteries.
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A 16-year-old boy presented to the emergency department with chest pain. He had no history of other health problems and did not smoke. Acute myocardial infarction (AMI) in an adolescent boy with normal coronary arteries, associated with pseudoephedrine use and acute streptococcal infection, is
Rapidly progressive bilateral optic nerve and retinal infarctions due to rhinocerebral mucormycosis and pseudoephedrine use.
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Acute myocardial infarction after use of pseudoephedrine for sinus congestion.
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ST elevation myocardial infarction presenting after use of pseudoephedrine.
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ST elevation myocardial infarction after use of pseudoephedrine : which is more dangerous, the common cold itself or the medication used for it?
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Follow-up study of pseudoephedrine users.
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A follow-up study of over 100,000 persons below age 65 years who filled a total of 243,286 prescriptions for pseudoephedrine indicated that there were no hospitalizations among users that could be attributed to the drug. There were no admissions within 15 days of filling a prescription for
Acute myocardial infarction in sickle cell anemia.
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Cardiovascular dysfunction consistent with ischemia has been observed during episodes of painful crisis and following periods of heavy physical exertion in individuals with sickle cell disease. Similar findings have been observed in other individuals while taking the alpha-adrenergic agonist
Myocardial infarction with normal coronary arteriogram: the role of ephedrine-like alkaloids.
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BACKGROUND
The overwhelming majority of myocardial infarctions result from atherosclerosis, generally with superimposed coronary thrombosis. The prevalence of patients who develop myocardial infarction and in whom subsequent angiography shows normal coronary arteries is approximately 5%. Many of
Acute myocardial infarction related to methylphenidate for adult attention deficit disorder.
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Adult Attention Deficit Disorder is increasingly diagnosed and treated. Psychostimulant medications, such as methylphenidate, are commonly prescribed for this condition, but the long-term safety of such medications in an adult population is unknown at present. Because these medications are closely
Myocardial infarction temporally related to ephedra--a possible role for the coronary microcirculation.
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Myocardial infarction has been reported with ephedrine and pseudoephedrine use. It has been suggested that these agents may induce coronary artery spasm, hypercoagulable states, or oxygen demand imbalance. We report a 25-year-old male with myocardial infarction after receiving a diet pill containing
Acute myocardial ischemia associated with ingestion of bupropion and pseudoephedrine in a 21-year-old man.
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A 21-year-old man presented to the emergency department with atypical chest pain, diaphoresis and shortness of breath. His electrocardiogram revealed ST segment elevation in leads II, III, aVF, V5 and V6, elevated creatine kinase-MB subunit levels and positive troponin I. He denied the use of
Acute coronary syndrome presenting after pseudoephedrine use and regression with beta-blocker therapy.
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Pseudoephedrine, a common ingredient in cold relief drugs, dietary supplements and Chinese herbal tea, has potent sympathomimetic effects, impacting the cardiovascular system. The chemical properties and clinical effects of pseudoephedrine are similar to those of ephedrine, and its main effect is
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