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salicylate/obesitas
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Influence of age, gender, and obesity on salicylate kinetics following single doses of aspirin.
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Salicylate kinetics following single, 650-mg intravenous and oral doses of aspirin were evaluated in humans in 2 studies. Complete conversion of aspirin to salicylate was assumed. The first study involved 25 young (25-40 years) and 21 elderly (66-89 years) healthy male and female volunteers. Mean
Salicylates increase insulin secretion in healthy obese subjects.
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BACKGROUND
Conflicting results on the effects of salicylates on glucose tolerance in subjects with normal glucose tolerance or type 2 diabetes have been reported.
OBJECTIVE
The objective of the study was to investigate the effects of a salicylate derivative (triflusal) on insulin sensitivity and
Salicylate downregulates 11β-HSD1 expression in adipose tissue in obese mice and in humans, mediating insulin sensitization.
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Recent trials show salicylates improve glycemic control in type 2 diabetes, but the mechanism is poorly understood. Expression of the glucocorticoid-generating enzyme 11β-hydroxysteroid dehydrogenase type 1 (11β-HSD1) in adipose tissue is increased in vitro by proinflammatory cytokines and
The effect of high-dose sodium salicylate on chronically elevated plasma nonesterified fatty acid-induced insulin resistance and β-cell dysfunction in overweight and obese nondiabetic men.
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Prolonged elevation of plasma nonesterified fatty acids (NEFA) induces insulin resistance and impairs pancreatic β-cell adaptation to insulin resistance. Studies in rodents suggest that inflammation may play a role in this "lipotoxicity." We studied the effects of sodium salicylate, an
Reversal of obesity- and diet-induced insulin resistance with salicylates or targeted disruption of Ikkbeta.
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We show that high doses of salicylates reverse hyperglycemia, hyperinsulinemia, and dyslipidemia in obese rodents by sensitizing insulin signaling. Activation or overexpression of the IkappaB kinase beta (IKKbeta) attenuated insulin signaling in cultured cells, whereas IKKbeta inhibition reversed
Medicine-Thymol Treatment of Anchylostomiasis-Salicylic Acid in Rheumatism-Salicylate of Methyl-Cimicifuga Racemosa-Thyroidin in Obesity-Guaiacol as an Anæsthetic-Diagnosis of Diabetes.
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Sodium salicylate reduced insulin resistance in the retina of a type 2 diabetic rat model.
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Sodium salicylate has been reported to reduce markers of diabetic retinopathy in a type 1 rat model. Because rates of type 2 diabetes are on the rise, we wanted to determine whether salicylate could improve insulin resistance in a type 2 rat model, as well as improve retinal function. We treated
Conditional disruption of IkappaB kinase 2 fails to prevent obesity-induced insulin resistance.
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The inhibitor of NF-kappaB (IkappaB) kinases (IKK1[alpha] and IKK2[beta]), the catalytic subunits of the IKK complex, phosphorylate IkappaB proteins on serine residues, targeting them for degradation and thus activating the transcription factor NF-kappaB. More recently, IKK2 has been implicated in
Low level of trans-10, cis-12 conjugated linoleic acid decreases adiposity and increases browning independent of inflammatory signaling in overweight Sv129 mice.
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The objective of this study was to determine the extent to which a low level of trans-10, cis-12 (10,12) conjugated linoleic acid (CLA) decreases adiposity and increases browning in overweight mice, its dependence on inflammatory signaling and potential synergistic effects of daily exercise. Young,
[Low salicylate diet and the possibility of nutrient deficiencies].
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Unfavourable reactions of the body to consumed food and/or medicines are an increasing epidemiological problem. Intolerance to acetylsalicylic acid is connected to the intake of some drugs and food containing salicylic acid. People suffering from this intolerance have to follow an elimination diet
Salicylate blocks lipolytic actions of tumor necrosis factor-alpha in primary rat adipocytes.
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Increased systemic free fatty acids (FFA) impair insulin sensitivity. In obese and diabetic subjects, production of tumor necrosis factor-alpha (TNF-alpha), a proinflammatory cytokine, is elevated. TNF-alpha has a variety of effects by inducing inflammation, decreasing glucose utilization, and
Regulation of diet-induced adipose tissue and systemic inflammation by salicylates and pioglitazone.
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It is increasingly accepted that chronic inflammation participates in obesity-induced insulin resistance and type 2 diabetes (T2D). Salicylates and thiazolidinediones (TZDs) both have anti-inflammatory and anti-hyperglycemic properties. The present study compared the effects of these drugs on
[Prevalence of intolerance to salicylates in patients with nasal polyposis].
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BACKGROUND
Salicylates intolerance is related to alteration in the metabolism of arachidonic acid leading to increased leukotrienes. The condition may be manifested with respiratory, skin or systemic symptoms or associated with sinonasal polyposis. Salicylates are present in anti-inflammatory drugs,
Adipose tissue biglycan as a potential anti-inflammatory target of sodium salicylate in mice fed a high fat diet.
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BACKGROUND
Inflammation in adipose tissue (AT) during obesity causes impaired AT function. Although multiple extracellular matrix (ECM) proteins are expressed in AT their potential role in adipose tissue inflammation is unclear. Biglycan, a pro-inflammatory ECM gene, is highly enriched in adipose
Potential role of salicylates in type 2 diabetes.
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OBJECTIVE
To review the evidence base supporting the use of salicylates for glucose level control in patients with type 2 diabetes and provide a comprehensive review of available information describing the potential role of salicylates and, in particular, salsalate, for glucose control in type 2
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