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Electron-microscopic examination of effects of yokukansan, a traditional Japanese medicine, on degeneration of cerebral cells in thiamine-deficient rats.
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We previously demonstrated that yokukansan ameliorated not only learning disturbance but also behavioral and psychological symptoms of dementia-like behaviors (anxiety, aggressiveness) and neurological symptoms (opisthotonus) induced in rats by dietary thiamine deficiency (TD). In the present study,
Low thiamine diphosphate levels in brains of patients with frontal lobe degeneration of the non-Alzheimer's type.
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We compared the thiamine and thiamine phosphate contents in the frontal, temporal, parietal, and occipital cortex of six patients with frontal lobe degeneration of the non-Alzheimer's type (FNAD) or frontotemporal dementia with five age-, postmortem delay-, and agonal status-matched control
Thiamine deficiency in vivo produces fiber cell degeneration in mouse lenses.
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Thiamine (Vitamin B1) is a co-factor for enzymes key in bridging aerobic and anaerobic metabolism. One such enzyme, transketolase, catalyzes two of three reactions for entry into the pentose-phosphate pathway, a major source of chemical reducing power. Thus, thiamine deprivation (TD) is considered a
Vermal atrophy of alcoholics correlate with serum thiamine levels but not with dentate iron concentrations as estimated by MRI.
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Chronic alcohol consumption is frequently accompanied by cerebellar degeneration. The exact aetiology of alcoholic cerebellar degeneration is still a matter of debate. The aim of the present study was to investigate whether patients with chronic alcohol consumption exhibit a decrease in dentate
Focal thalamic degeneration from ethanol and thiamine deficiency is associated with neuroimmune gene induction, microglial activation, and lack of monocarboxylic acid transporters.
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BACKGROUND
Wernicke's encephalopathy-Korsakoff syndrome (WE-KS) is common in alcoholics, caused by thiamine deficiency (TD; vitamin B1) and associated with lesions to the thalamus (THAL). Although TD alone can cause WE, the high incidence in alcoholism suggests that TD and ethanol (EtOH)
[Thiamine deficiency and brain atrophy in alcoholic patients].
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CT brain scans of 65 alcohol-dependent inpatients were compared before and after 6 weeks of confirmed abstinence. Linear measurements revealed a significant reduction of the enlargement of the ventricular system in accordance with the re-expansion of the brain after alcohol abstinence (ANOVA,
Shoshin Beriberi With Low Cardiac Output and Hemodynamic Deterioration Treated Dramatically by Thiamine Administration.
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"Shoshin beriberi", which is a fulminant form of cardiovascular beriberi accompanied by hemodynamic deterioration with high cardiac output and decreased systemic blood pressure, caused by thiamine deficiency due to alcoholic abuse or malnutrition, is often difficult to address because of its rarity
Thiamine deficiency encephalopathy with concurrent myocardial degeneration and polyradiculoneuropathy in a cat.
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Hyaline degeneration, myofiber fragmentation, and mineralization were present in the left ventricle of an aged, female Siamese cat with both clinical and histopathological evidence suggestive of a thiamine deficiency encephalopathy. Both a thiamine related biochemical lesion and neurogenic
Development of Myocardial Necrosis and Absence of Nerve Degeneration in Thiamine Deficiency in Pigs.
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Cerebellar degeneration in thiamine-deficient rats.
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Thiamine responsive congenital lactic acidemia and type 1 muscle fiber atrophy.
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Nutritional deficiency and wallerian degeneration in the rat. II. Effect of thiamine deficiency on the concentration of nucleic acid and phospholipid in intact and sectioned nerves.
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Massive thiamine dosage in an alcoholic with cerebellar cortical degeneration.
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[Studies on the effects of thiamine deficiency on rat testes (author's transl)].
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Using male rats fed a thiamine deficient diet from the age of 35-days, an investigation was made of the effects on testicular tissue after 30 days of thiamine deficiency. It was found that: 1) In the thiamine deficient group, the seminiferous tubuli of rats having erections had atrophied and there
Peripheral nerve changes in thiamine deficiency and starvation. An electron microscopic study.
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Electron microscopic investigations on sciatic and plantar nerves of thiamine deficient and starved rats show a distally pronounced axonal degeneration. The changes are present in starved and thiamine deficient animals, but the deficient animals are more severely affected. The earliest alterations
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