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Serum urea and total cholesterol independently predict re-hospitalisation with a cardiac-related event following an acute ST-elevation myocardial infarction.
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BACKGROUND
Although elevated serum urea and low serum sodium have been shown to be associated with increased short-term (30-day) mortality following an ST-elevation myocardial infarction (STEMI), little is known about the role of these biochemical markers as predictors of intermediate-term (1-year)
Urea cycle defect: a case with MR and CT findings resembling infarct.
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A 2 1/2 year old girl was admitted to the hospital because of recurrent vomitting, impaired consciousness, and hyperammonemia. MR and CT findings resembled an infarct, but she was found to have a defect in the urea cycle, partial ornithine transcarbamylase deficiency.
Plasma cortisol, corticosterone and urea in acute myocardial infarction: clinical and biochemical correlations.
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22 patients suffering from acute myocardial infarction were subjected to daily blood sampling for up to 6 days after admission to hospital. 5 patients with myocardial ischaemia and chest pain, but no evidence of infarction, were similarly investigated as controls. Daily measurements of plasma
Raised plasma urea levels after myocardial infarction.
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Plasma and urine urea and creatinine levels were measured in 50 consecutive patients admitted to a coronary care unit. Forty of the patients had had myocardial infarction; their average plasma urea level increased substantially by the third day after admission, when 50% of the patients had an
Predictive value of blood urea nitrogen/creatinine ratio in the long-term prognosis of patients with acute myocardial infarction complicated with acute heart failure.
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At present, the long-term prognosis of patients with acute myocardial infarction (AMI) after emergency percutaneous coronary intervention is the focus of attention, and relevant research is actively investigating the risk factors associated with prognosis. Poor prognosis often exists in Patients
Blood urea nitrogen has additive value beyond estimated glomerular filtration rate for prediction of long-term mortality in patients with acute myocardial infarction.
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OBJECTIVE
Blood urea nitrogen (BUN) has been shown to independently predict short- and intermediate-term outcomes in patients with acute myocardial infarction (AMI). We aimed to assess the additive predictive value of BUN beyond estimated glomerular filtration rate (eGFR) in AMI patients with an
A High Level of Blood Urea Nitrogen Is a Significant Predictor for In-hospital Mortality in Patients with Acute Myocardial Infarction.
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High levels of blood urea nitrogen (BUN) have been demonstrated to significantly predict poor prognosis in patients with acute decompensated heart failure. However, this relationship has not been fully investigated in patients with acute myocardial infarction (AMI). We investigated whether a high
Serum blood urea nitrogen and long-term mortality in acute ST-elevation myocardial infarction.
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BACKGROUND
Renal dysfunction is associated with increased mortality in acute coronary syndromes and other cardiovascular diseases. The prognostic value of kidney dysfunction has been investigated using creatinine-based measures of renal function. Few data are available on the prognostic significance
Reduced hydration status characterized by disproportionate elevation of blood urea nitrogen to serum creatinine among the patients with cerebral infarction.
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The significance of fluid metabolism among the patients with cerebral infarction has barely mentioned in the literature despite the several reports suggesting the potential risk of reduced hydration status for the development of cerebral infarction. The aim of the this study is to explore the
[Diagnostic value of urea and potassium oxalate in recent myocardial infarction for the inhibition of lactate dehydrogenase and alpha-hydroxybutyrate dehydrogenase].
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Blood urea in myocardial infarction.
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[Diagnosis of myocardial infarct by determination of urea stabile lactate dehydrogenase].
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[Application of urea for differentiation of lactate dehydrogenase in myocardial infarct].
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[Inhibition of serum lactate dehydrogenase activity with urea in diagnosed cases of myocardial infarct].
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[Urea-stable lactate dehydrogenase. Diagnostic value in myocardial infarction].
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