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15 niðurstöður
Histopathologic study of experimental Sarcocystis hemionilatrantis infection in fawns.
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Mule deer fawns (Odocoileus hemionus hemionus) inoculated with sporocysts of Sarcocystis hemionilatrantis became infected, developed clinical signs of disease, and died, due to the infection itself or from intercurrent pneumonia. Clinical signs were first noticed 18 days after infection and fawns
Epizootiology of Sarcocystis infections in mule deer fawns in Oregon.
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From 1974 to 1977, 62 wild mule deer (Odocoileus hemionus) fawns from Steens Mountain, Ore were euthanatized in autumn (23 deer), winter (21 deer), and spring (18 deer). The number of sarcocysts of Sarcocystis spp was counted in histologic sections of various muscular organs. Sarcocysts were seen in
Spontaneous hypertension in the fawn-hooded rat: a cardiovascular disease model.
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The fawn-hooded (FH) rat develops hypertension spontaneously. Systolic blood pressure is already elevated at 5 weeks of age, increases with age, and the final range is 180-240 mmHg at the age of 1 year. Concomitantly with the rise in blood pressure proteinuria occurs and increases with age.
Development of vascular pole-associated glomerulosclerosis in the Fawn-hooded rat.
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Fawn-hooded hypertensive (FHH) rats constitute a spontaneous model of chronic renal failure with early systemic and glomerular hypertension, proteinuria, and development of focal and segmental glomerulosclerosis. The goal of the present study was to elucidate a step-by-step sequence of
Effects of dietary protein on the progression of renal failure in the Fawn-Hooded rat.
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Fawn-Hooded (FH) rats on a normal protein intake develop focal glomerulosclerosis, proteinuria and hypertension and die prematurely because of renal failure. In the present study we examined the effect of a life long feeding of a low (12%)-protein (LP) diet and a high (36%)-protein (HP) diet on
Neuroepithelial bodies in the Fawn Hooded rat lung: morphological and neuroanatomical evidence for a sensory innervation.
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The lungs of young Fawn Hooded (FH) rats contain neuroepithelial bodies (NEB) with a very dense innervation. They are covered by a unicellular layer of flattened Clara cells and their apices protrude slightly into the airway lumen. The NEB corpuscle cells are loaded with dense core vesicles and are
Relationship between blood pressure level, renal histopathological lesions and plasma renin activity in fawn-hooded rats.
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The fawn-hooded rat (FH rat) develops hypertension accompanied with focal and segmental glomerulosclerosis and proteinuria, resulting in premature death. In a first experiment the relationship between renal lesions and blood pressure at various ages was investigated. In a second experiment blood
Characterization of mule deerpox virus in Florida white-tailed deer fawns expands the known host and geographic range of this emerging pathogen.
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Infections caused by mule deerpox virus (MDPV) have been sporadically reported in North American cervids. White-tailed deer (Odocoileus virginianus) fawns from a farm located in South Central Florida presented with ulcerative and crusting lesions on the coronary band as well as the mucocutaneous
From segmental glomerulosclerosis to total nephron degeneration and interstitial fibrosis: a histopathological study in rat models and human glomerulopathies.
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BACKGROUND
Focal segmental glomerulosclerosis (FSGS) is consistently associated with tubular degeneration and interstitial fibrosis, altogether, accounting for the progressive decline in renal function. The mechanisms which link glomerular injury to tubulo-interstitial fibrosis are controversial.
Tracer studies in the rat demonstrate misdirected filtration and peritubular filtrate spreading in nephrons with segmental glomerulosclerosis.
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In two genetic models of "classic" focal segmental glomerulosclerosis (FSGS), the Milan normotensive and the Fawn-hooded hypertensive rats, tracer studies were performed to test the hypothesis that misdirected glomerular filtration and peritubular filtrate spreading are relevant mechanisms that
Myopathy in the nyala.
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Between January 1973 and June 1981, a total of 21 nyalas (Tragelaphus angasi) died with clinical and/or pathologic evidence of myopathy. The main clinical signs were stiffness, inability to rise, and failure to suckle in newborn fawns. Death usually occurred without premonitory signs of disease.
Pathogenicity of immature Fascioloides magna in white-tailed deer.
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The pathogenesis of early prepatent Fascioloides magna infection was investigated in seven fawns (Odocoileus virginianus) given 500 metacercariae and examined at one, two, three, five, eight, 12 and 13 weeks postinoculation. Blood samples were taken from eight inoculated deer every two weeks up to
Genetic susceptibility and loss of Nr4a1 enhances macrophage-mediated renal injury in CKD.
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Nuclear hormone receptors of the NR4A subgroup have been implicated in cancer, atherosclerosis, and metabolic disease. However, little is known about the role of these receptors in kidney health or disease. Nr4a1-deficient rats (Nr4a1(-/-)) developed on a genetic background susceptible to kidney
[Cockayne syndrome].
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Cockayne syndrome is a rare autosomal recessive disease. This paper reports a case of Cockayne syndrome confirmed by gene analysis. The baby (male, 7 years old) was referred to Peking University Third Hospital with recurrent desquamation, pigmentation and growth and development failure for 6 years,
Clinical and pathological manifestations of cardiovascular disease in rat models: the influence of acute ozone exposure.
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Rodent models of cardiovascular diseases (CVD) and metabolic disorders are used for examining susceptibility variations to environmental exposures. However, cross-model organ pathologies and clinical manifestations are often not compared. We hypothesized that genetic CVD rat models will exhibit
Heillasta gagnagrunnur lækningajurtanna sem studdur er af vísindum
- Virkar á 55 tungumálum
- Jurtalækningar studdir af vísindum
- Jurtaviðurkenning eftir ímynd
- Gagnvirkt GPS kort - merktu jurtir á staðsetningu (kemur fljótlega)
- Lestu vísindarit sem tengjast leit þinni
- Leitaðu að lækningajurtum eftir áhrifum þeirra
- Skipuleggðu áhugamál þitt og vertu vakandi með fréttarannsóknum, klínískum rannsóknum og einkaleyfum
Sláðu inn einkenni eða sjúkdóm og lestu um jurtir sem gætu hjálpað, sláðu jurt og sjáðu sjúkdóma og einkenni sem hún er notuð við.
* Allar upplýsingar eru byggðar á birtum vísindarannsóknum
