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fawn/krabbamein

Krækjan er vistuð á klemmuspjaldið
GreinarKlínískar rannsóknirEinkaleyfi
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Natural killer cell activity in fawn-hooded rats.

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Fawn-hooded (FH) rats were shown to lack the genetically conditioned defect of natural killer (NK) activity hypothesized to be present by analogy with the Chediak-Higashi syndrome (CHS) in mice and human beings. In 4-h 51Cr release assays, splenic NK cells from FH rats killed YAC-1, RL male l and

FURTHER INVESTIGATIONS ON THE ORIGIN OF TUMORS IN MICE : I. TUMOR INCIDENCE AND TUMOR AGE IN VARIOUS STRAINS OF MICE.

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1. It is possible to split a strain of mice into certain substrains in which the tumor incidence is in some way linked to the color of the mice. Thus we could split off from the English strain, which as a whole and in various substrains with mixed colors (English A and Sable) has a high tumor rate,

Rearing performance of farmed fallow deer (Dama dama).

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The ability of fallow does to rear fawns to weaning age was examined in a group of 243 adult does of known age (Group 1) and 509 adult does of unknown age (Group 2). All does had between 1 and 8 opportunities to rear fawns. There was an increasing proportion of does that failed to rear a fawn to

Genetic susceptibility and loss of Nr4a1 enhances macrophage-mediated renal injury in CKD.

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Nuclear hormone receptors of the NR4A subgroup have been implicated in cancer, atherosclerosis, and metabolic disease. However, little is known about the role of these receptors in kidney health or disease. Nr4a1-deficient rats (Nr4a1(-/-)) developed on a genetic background susceptible to kidney
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Heillasta gagnagrunnur lækningajurtanna sem studdur er af vísindum

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