lysozyme/hitasótt

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Hyperthermia Induced by Near-Infrared Laser-Irradiated CsWO3 Nanoparticles Disintegrates Preformed Lysozyme Amyloid Fibrils.

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This research study attempts to prove the concept of the applicability of hyperthermia to treating the lysozyme amyloid fibrils (LAF)'s self-assembled fibrillary aggregates by a feedback-modulated temperature controller ranging from 26 °C to 80 °C, and separately, by near-infrared (NIR)

Mapping quantitative trait loci for the lysozyme level and immunoglobulin G blocking percentage of classical swine fever virus.

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Increased disease resistance through improved general immune capacity would be beneficial for the welfare and productivity of farm animals. Classical swine fever (CSF) is a contagious disease in farm animals. The immunoglobulin G (IgG) blocking percentage of CSF virus (CSFV) in serum is an essential

[The content of rosette-forming neutrophils and lysozyme in patients with Q-fever].

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The article deals with the results of study of 35 patients with Q-fever and 30 donors to determine the content of rosette-forming neutrophils, lysozyme activity of blood and saliva. On the second week of disease it was revealed the activation of compensative immune mechanisms and on third and fourth

A neutrophil lysozyme leak in patients with familial Mediterranean fever.

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Polymorphonuclear cells derived from the peripheral blood of patients with Familial Mediterranean Fever release more lysozyme in response to high temperature (42 degrees, 46 degrees C) than do control cells. No differences between the FMF and control cells were observed in the release of acid

Influence of fever temperatures and of some cytoactive drugs on in vitro lysozyme release from monocytes and granulocytes.

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The selective in vitro release of lysozyme from human monocytes and granulocytes was not greatly influenced by temperatures above 37 degrees C and up to 40 degrees C. The release was markedly inhibited by preincubation with phenylbutazone, oxyphenylbutazone, colchicine and vincristine. A

[Behavior of the blood agglutinin rate and blood protein picture after administration of parenteral lysozyme in patients with typhoid fever and brucellosis].

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[Dynamic changes of lysozyme concentration in the blood serum in patients with typhoid fever treated levomycetin in conjunction with prednisolone and butadione].

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Renal damage in hemorrhagic fever with renal syndrome. Measurements of urine and serum lysozyme and urine IgG.

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Lysozymes in yellow fever infection.

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[The importance of lysozyme in rheumatic fever].

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Plasma lysozyme activity of Polish primitive horses under physiological conditions and in experimental fever.

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Novel protagonists in autoinflammatory arthritis of familial Mediterranean fever.

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To clarify mechanisms responsible for the self-limiting and nonerosive features of autoinflammatory joint disease in familial Mediterranean fever (FMF), we performed a study on synovial tissue obtained surgically from an acutely inflamed hip joint from a boy feared to have septic arthritis but later

Functional and morphologic changes during experimental Rocky Mountain spotted fever in guinea pigs.

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Experimental Rocky Mountain spotted fever was studied in guinea pigs following intraperitoneal inoculation of 10(7) Rickettsia rickettsii. After a 2-day incubation period, animals developed fever, progressive emaciation, and scrotal swelling with necrosis. Vasculitis, with increased small vessel

Increased procoagulant response of monocytes from patients with familial Mediterranean fever.

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Familial Mediterranean Fever (FMF) is an inherited disease of unknown etiology characterized by recurrent inflammatory episodes. Circulating fibrin was found in patients with FMF in absence of clinical manifestation of thrombosis and was statistically less frequently observed in patients treated

Familial Mediterranean fever. Recent advances in pathogenesis and management.

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The success of colchicine therapy in the management of familial Mediterranean fever has provided new direction to investigations into the pathogenesis of this disease. Examination of HLA antigen frequencies in 53 patients with familial Mediterranean fever and appropriate controls, as well as various

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