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Pagina 1 a partire dal 53 risultati
Lead induced thiamine deficiency in the brain decreased the threshold of electroshock seizure in rat.
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Many neurological disorders that occur frequently in lead intoxicated animals, have also been observed in thiamine deficient animals. To test whether lead intoxication could decrease the thiamine status and thresholds of electroshock seizure in rats, 3-week-old Wistar rats were treated with lead or
[Lactic acidosis caused by convulsions and shoshin beriberi].
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[A case with severe neurological involvement due to vitamin B1 deficiency associated with megaduodenum].
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We reported a case of 4-year-old boy with multiple vitamin deficiencies, especially vitamin B1 deficiency. He had megaduodenum associated with membranous stenosis on upper jejunum. He showed recurrent vomiting at his infantile period, and recently intermittent neurological symptoms. When he was
[Effect of pyrithiamine on rat sciatic nerve. (II) Morphological changes during the last stages of thiamine deficiency (author's transl)].
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We observed under light and electron microscopes morphological changes in the rat sciatic nerve during the last stages of a thiamine deficient state as induced by pyrithiamine (PT: 50 microgram/100g X 11 days) and thiamine deficient diet (TDD), in which rats experienced severe tetanic convulsions.
Epileptic manifestations and vitamin B1 deficiency.
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Sixteen of 50 consecutive neurological patients with a diagnosis of thiamine deficiency showed epileptic (10) or epileptiform (6) manifestations. A survey of the literature revealed only few reports on a possible relationship between epilepsy and thiamine deficiency. It appears that thiamine
Anticonvulsant effects of thiamine on pentylenetetrazole-induced seizure in mice.
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OBJECTIVE
Thiamine serves as a cofactor for several enzymes involved in brain function and neurotransmitters biosynthesis. Thiamine-dependent enzymes are important for oxidant stress defenses. Several studies have reported that thiamine deficiency in the central nervous system reduces seizure
Thiamine deficiency in a dog associated with exclusive consumption of boiled sweet potato (Ipomoea batatas): Serial changes in clinical findings, magnetic resonance imaging findings and blood lactate and thiamine concentrations
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Thiamine (vitamin B1 ) is an essential nutrient that significantly influences ATP production in the body. It needs to be supplemented consistently through an exogenous source to prevent deficiency; however, it is easily affected by a variety of mitigating factors. Additionally, thiamine
Thiamine deficiency in a cat: resolution of MRI abnormalities following thiamine supplementation.
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Thiamine (vitamin B(1)) is an essential component of a number of metabolic pathways and thiamine deficiency results in a progressive encephalopathy in both humans and animals. Confirming thiamine deficiency is problematic and relies on demonstrating reduced red blood cells transketolase activity, or
Depletion of brain histamine produces regionally selective protection against thiamine deficiency-induced lesions in the rat.
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Breakdown of the blood brain barrier and the subsequent accumulation of free radicals, lactate, and glutamate appear to be the immediate causes of thiamine deficiency (TD)-induced damage to thalamus. The mechanisms triggering these events are unknown but recent evidence suggests an important role of
Polioencephalomalacia of dogs with thiamine deficiency.
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A naturally occurring neurological disease occurred in six dogs fed cooked meat. Clinical signs were anorexia, progressive spastic paraparesis, recumbency, convulsions and death. The disease was characterized by bilaterally symmetrical spongy change and necrosis of brainstem nuclei with a lesion
Epilepsy in children with infantile thiamine deficiency.
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OBJECTIVE
To report the follow-up findings of 7 children with severe epilepsy as a result of thiamine deficiency in infancy caused by a defective soy-based formula.
METHODS
The medical records of 7 children aged 5-6 years with thiamine deficiency in infancy who developed epilepsy were reviewed and
Thiamine deficiency in dogs due to the feeding of sulphite preserved meat.
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A 6-year-old dog, a 4-year-old dog and three 7-week-old puppies were diagnosed with thiamine deficiency caused by feeding sulphite treated meat. The 6-year-old dog presented with a history of inappetence, weight loss and vomiting that rapidly progressed to signs of multifocal intracranial disease
Outbreak of thiamine deficiency in cats associated with the feeding of defective dry food.
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Objectives The objective of this study was to determine disease progression, association between neurological signs and magnetic resonance imaging (MRI) findings, and long-term outcome in feline thiamine deficiency associated with defective dry food. Methods The clinical records of 17 cats diagnosed
Activities of thiamine-dependent enzymes in two experimental models of thiamine-deficiency encephalopathy: 1. The pyruvate dehydrogenase complex.
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Chronic thiamine deprivation in the rat leads to selective neuropathological damage in brainstem structures whereas treatment with the central thiamine antagonist, pyrithiamine, results in more widespread damage. In order to further elucidate the neurochemical mechanisms responsible for this
Extracellular glutamate is increased in thalamus during thiamine deficiency-induced lesions and is blocked by MK-801.
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The current study measured extracellular fluid (ECF) levels of excitatory amino acids before and during the onset of thiamine deficiency-induced pathologic lesions. Male Sprague-Dawley rats were treated with daily pyrithiamine (0.25 mg/kg i.p.) and a thiamine-deficient diet (PTD). Microdialysates
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