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Pagina 1 a partire dal 39 risultati
BRL 38227--a potassium channel opener, antagonizes digoxin-induced convulsions.
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Experimental evidence suggests that potassium channel openers play an important role in convulsions. In this study, the anticonvulsant activity of BRL 38227, a new potassium channel opener against digoxin-induced convulsions, is reported. Intraventricular administration of digoxin (7.5 micrograms),
Possible mechanism of digoxin-induced convulsions.
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Intraventricular (IVT) administration of digoxin (7.5 micrograms) induced 'popcorn-type' convulsions in rats. Though the convulsions looked similar to morphine-induced seizures, naloxone failed to antagonize these effects. Other anticonvulsants like phenobarbitone, ethosuximide, or GABAergic
Digoxin enhances the effect of antiepileptic drugs with different mechanism of action in the pentylenetetrazole-induced seizures in mice
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The worldwide prevalence of epilepsy with high percentage of multidrug-resistant patients make it urgent to find new approaches to treating, including the use of combinations of classic anticonvulsants with drugs that have an exclusively original mechanism of action, in particular digoxin. The aim
GABA/BZ-and NMDA-receptor interaction in digoxin-induced convulsions in rats.
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Digoxin (7.5 micrograms icv) induced 'pop-corn' type of convulsions and 100% mortality. The GABA-ergic agents produced varying degree of protection against digoxin-induced neurotoxicity. Diazepam (4 mg/kg) offered significant protection whereas pentobarbital (5 mg/kg) and baclofen (5 mg/kg) markedly
Epileptiform seizures and electroencephalographic abnormalities as manifestations of digoxin toxicity.
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A digoxin induced epileptiform seizure: an unusual toxic manifestation.
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The endogenous digoxin-like factor enhances bupivacaine toxicity in rats.
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OBJECTIVE
The influence of the immunoreactive endogenous digoxin-like factor (IEDLF) on the systemic toxic effects of bupivacaine was studied in a rodent model.
METHODS
During 5 weeks, IEDLF secretion was promoted in 10 Wistar male rats by allowing them to drink saline 0.5% in place of water. Ten
Chemical Meningitis and Status Epilepticus Caused by Accidental Epidural Administration of Digoxin.
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Intrathecal administration of digoxin occurs very rarely. Some case reports of inadvertently administering it when performing spinal/epidural anesthesia were described. We report for the first time a case of a chemical meningitis and status epilepticus caused by accidental epidural administration of
Changes in the isoprenoid pathway with transcendental meditation and Reiki healing practices in seizure disorder.
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A quantal perceptive model of brain function has been postulated by several groups. Reiki-like healing practices in seizure disorder (ILAE classification-II E-generalized seizures-tonic clonic), involving transfer of life force or low level of electromagnetic force (EMF) from the healer to the
A child with supraventricular tachycardia and convulsions.
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We present an 11-year-old girl, referred for management of episodes of supraventricular tachycardia followed by generalised tonic-clonic convulsions. The episodes did not respond to digoxin and propranolol, but subsided after initiation of carbamazepine therapy. Seizures following supraventricular
Autopsy report on central pontine myelinolysis triggered by vomiting associated with digoxin intoxication.
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An 87-year-old male, prescribed digoxin and furosemide for congestive heart failure and Alzheimer disease, had dehydration and anemia due to poor food intake and hemorrhagic cystitis. Repeated vomiting due to an upper respiratory infection caused disturbance of consciousness and hypotension. The
Hypothalamic digoxin-mediated model for epileptogenesis.
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OBJECTIVE
This study assessed the changes in the isoprenoid pathway and its metabolites in seizure disorder (ILAE classification - I generalized - idiopathic generalized epilepsy with age-related onset - epilepsy with generalized tonic clonic seizures on awakening) and the metabolic cascade produced
Digoxin enhances bupivacaine toxicity in rats.
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The influence of digoxin on the systemic toxic effects of bupivacaine was studied in a rodent animal model. The experiment was undertaken with consideration to clinical situations such as pregnancy that are associated with the secretion of an endogenous digoxin-like factor. Twenty Wistar male rats,
Levetiracetam. A review of its adjunctive use in the management of partial onset seizures.
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Levetiracetam, the S-enantiomer of alpha-ethyl-2-oxo-1-pyrollidine acetamide, is approved for use as adjunctive therapy in adult patients with partial onset seizures. Oral levetiracetam 1000, 2000 and 3000 mg/day administered as adjunctive therapy for up to 18 weeks significantly increased responder
[Digoxin--an active antiepileptic agent].
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A new pathogenetically grounded approach to the treatment of resistant forms of epilepsy is proposed. Digoxin suppresses the activity of experimental epileptogenic foci produced in the hippocampus of animals (frogs and rats) and also convulsions induced by corazol, bemegrid or electroshock. The
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