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Pagina 1 a partire dal 482 risultati
Effect of dopamine on hemodynamics and myocardial metabolism in shock following acute myocardial infarction in man.
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Eight patients in shock associated with acute myocardial infarctions were treated with dopamine. We titrated the dopamine dose to increase mean arterial pressure to 65-70 mm Hg and urine output to greater than 40 ml/hr. Increase of heart rate to 120-125 beats/min and occurrence of potentially
Bilateral retinal infarction associated with high dose dopamine.
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A 30-year-old unconscious woman in a hypoglycemic coma responded rapidly to intravenous glucose administration with full neurologic recovery. She was diagnosed with fulminant non-A-non-B hepatitis. On the fourth hospital day, she became suddenly lethargic and required dopamine and dobutamine for
Contrasting effects of dopamine and dobutamine on myocardial release of norepinephrine during acute myocardial infarction.
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The effects of dopamine and dobutamine on release of norepinephrine from normal and ischemic myocardium were compared in 2 groups of open-chest anesthetized dogs. Both agents were infused intravenously at a rate of 10 micrograms/Kg/min for 2 hours, beginning 40 min after snare occlusion of the left
Effect of dopexamine hydrochloride in the early stages of experimental myocardial infarction and comparison with dopamine and dobutamine.
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The effect of dopexamine hydrochloride on myocardial performance, and on the susceptibility of the myocardium to generation of arrhythmias during the development of myocardial infarction has been compared with dopamine and dobutamine in 2 experimental models of myocardial ischemia. All 3 agents
Effects of intravenous amrinone on heart failure complicated by acute myocardial infarction: comparative study with dopamine and dobutamine.
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The hemodynamic effects of a newly developed inotropic agent, amrinone (AMN) were studied and compared with those of dopamine (DA) and dobutamine (DB) in forty patients with pump failure due to acute myocardial infarction. Hemodynamic measurements were taken using a Swan-Ganz thermodilution catheter
Acupuncture stimulation at Baihui acupoint reduced cerebral infarct and increased dopamine levels in chronic cerebral hypoperfusion and ischemia-reperfusion injured sprague-dawley rats.
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The Baihui acupoint has three Yang and five convergences; it is needled in order to activate spirit and resuscitate the brain in traditional Chinese medicine. Therefore, the purpose of the present study is to investigate the effect of acupuncture stimulation at the Baihui acupoint on cerebral
Dopamine-beta-hydroxylase activity in serum following acute myocardial infarction: an evaluation of this parameter for routine use as an index of sympathetic activity.
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Dopamine-beta-hydroxylase activity was measured in sera from 114 normal males and from 11 patients on the 1st, 2nd, 3rd, 5th and 10th day following acute myocardial infarction. A significant elevation of dopamine-beta-hydroxylase levels (P less than 0.001) was found during the first two days after
Dopamine-beta-hydroxylase activity after acute myocardial infarction.
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After acute myocardial infarction, serial serum dopamine-beta-hydroxylase (DBH) activity was elevated in both high and low DBH subgroups. The observed increase in DBH activity on the first, second, and third days after acute myocardial infarction suggests an augmentation in sympathetic nervous
Propofol reduces infarct size and striatal dopamine accumulation following transient middle cerebral artery occlusion: a microdialysis study.
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Acute cerebral ischemia is associated with an increased extracellular dopamine accumulation. Attenuation or prevention of excessive dopamine accumulation alleviates the cerebral ischemic damage. Propofol, an intravenous anesthetic, has been suggested to have neuroprotective properties. The effect of
Effects of combined dopamine and nitroprusside therapy in patients with severe pump failure and hypotension complicating acute myocardial infarction.
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In severe pump failure with hypotension complicating acute myocardial infarction, dopamine has been useful in raising arterial pressure by increasing myocardial performance and augmenting peripheral resistance. Once adequate blood pressure are obtained, vasodilators may be used to reduce peripheral
Observation on the effect of sodium nitroprusside and dopamine on acute myocardial infarction during hospitalization.
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With improvements in cardiac monitoring systems and the effective treatment of arrhythmias, the case-fatality rate of acute myocardial infarction (AMI) has been declining gradually since 1972. But since 1977, despite the great efforts of medical workers in many countries, the lowest case-fatality
Dopamine D1 receptor gene polymorphism is associated with myocardial infarction.
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Dopamine D1 receptor (DRD1) gene is associated with the pathogenesis of myocardial infarction (MI) in aspects of plaque rupture, platelet aggregation, and neutrophil-mediated injury of cardiac myocytes. Thus, the study was designed to explore whether the A-48G polymorphism of the DRD1 gene was
Inotropic stimulation of reperfused myocardium with dopamine: effects on infarct size and myocardial function.
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Prolonged postischemic ventricular dysfunction ("stunned myocardium") may be responsible for heart failure after myocardial reperfusion. Although inotropic stimulation can enhance the contractility of stunned myocardium, it could potentially increase infarct size and thereby impair ultimate recovery
Dopamine-beta-hydroxylase activity after acute myocardial infarction.
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Serum dopamine-beta-hydroxylase (DBH) activities of 74 apparently healthy adults were not distributed normally, but skewed to the right. There were at least 2 subgroups in the distribution of DBH activity. Accordingly, 27 serial serum DBH activities following acute myocardial infarction were divided
[Comparative effects of dopamine and dobutaine in subendocardial perfusion in the acute phase of myocardial infarct].
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Dobutamine, a selective agonist of beta I adrenergic receptors was proposed as an inotropic support of the failing left ventricle in the acute phase of myocardial infarction. We studied the action of Dobutamine (10 micrograms/kg/min) on subendocardial perfusion, compared to the effects of an
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