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Pagina 1 a partire dal 175 risultati
[Varioliform gastritis revealed by edema with hypoproteinemia (author's transl)].
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The authors report the case of a woman in whom edemas with hypoproteinemia, but without digestive symptoms, were present for several months. Roentgenography, fiberoptic endoscopy, and histopathology showed typical varioliform gastritis. Hypoproteinemia was due to plasma protein leakage into the
[Lymphocytic gastritis with anasarca and venous thrombosis].
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BACKGROUND
Venous thrombosis has not previously been reported as a complication of lymphocytic gastritis.
METHODS
A 47-year-old patient was admitted for anasarca related to varioliform gastritis. The densitometric examination showed two partial thromboses, at the confluence of the splenic and
Pathologic quiz case. Edema and diarrhea in a 2-year-old boy. Menetrier disease with cytomegalovirus gastritis.
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[Apropos of a case of giant hypertrophic gastritis with hypoproteinemia and edema].
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[PROTEIN LOSS IN THE GASTROINTESTINAL TRACT. GASTRITIS WITH HYPOPROTEINEMIA AND EDEMA].
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[Postoperative gastritis, edema of the anastomosed opening].
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Case of Puerperal Fever with Dropsy, Succeeded by Phlegmasia Dolens, Gastritis, and Spontaneous Hydrophobia.
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Medical Treatment of Postendoscopic Submucosal Dissection Phlegmonous Gastritis in an Elderly Diabetic Woman with Myelodysplastic Syndrome.
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Phlegmonous gastritis is a rare, suppurative disease characterized by full-thickness exudative changes, infiltration of inflammatory cells, and edema primarily in the submucosal layer. A 76-year-old woman with type 2 diabetes and myelodysplastic syndrome underwent endoscopic submucosal dissection
Russell body gastritis.
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Russell body gastritis is caused by an accumulation of plasma cells within the gastric mucosa. These plasma cells are characterized by eosinophilic cytoplasmic inclusions of immunoglobulin which are called "Russell bodies". We report a case of Russell body gastritis in a 28-year-old male
[Status of microcirculation in patients with chronic gastritis and its relation to morphofunctional changes in the stomach].
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Studies on 74 patients with chronic gastritis showed that the disease was accompanied by disorders of the microcirculatory bed of the bulbar conjunctiva characterized by the appearance of perivascular edema, inhomogeneity of the vascular caliber and coiling, change of arteriolovenular ratios, the
[Clinical study on Helicobacter pylori infected chronic gastritis treated with supplementing qi and promoting blood circulation drugs].
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The syndrome of Qi Deficiency and Blood Stasis of chronic gastritis infected by Helicobacter Pylori (HP) was treated with the principle of supplementing Qi and promoting blood circulation. The clinical symptoms, inhibiting and killing of HP and pathohistological study have been observed. It was
A hyaluronic acid- and chondroitin sulfate-based medical device improves gastritis pain, discomfort, and endoscopic features.
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Gastritis is an inflammation of the gastric mucosa. In this study, we investigated the efficacy of a medical device, Esoxx®, based on hyaluronic acid and chondroitin sulfate on gastritis-related upper abdominal pain/discomfort and endoscopic features. Fifty patients, affected by gastritis, were
[Gastritis and gastropathies].
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A review of the different types of gastritis, including features in which the edema and erythema can resemble the picture, but without the typical inflammatory reaction that is called gastropathy. A description of various epidemiological, aetiological, pathogenetic, are proposed. The review
[Effects of 3 Chinese medical prescriptions on alkaline reflux gastritis in rats].
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The experimental alkaline reflux gastritis in rats was established after 9 weeks of anastomosis between stomach and jejunum. The experiment shows that xiangsha liujunzi decoction, xiaochaihu decoction and dahuang gancao decoction all help to inhibit edema, hyperamia and extravasted blood, as well as
Spontaneous remission of hypertrophic lymphocytic gastritis associated with hypoproteinemia.
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A 54-year-old woman came to our office because of pretibial edema. She had no gastrointestinal symptoms. Laboratory tests revealed severe hypoproteinemia. Upper gastrointestinal endoscopy demonstrated enlarged gastric folds and multiple aphthoid nodules on the body and the fornix of the stomach. The
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