goiter/phosphatase

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[Acid phosphatase of leukocytes in patients with diffuse toxic goiter in evaluating the stage of the disease].

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Acid phosphatase (AP) of lymphocytes and neutrophils was examined cytochemically in 23 patients with associated diffuse toxic goiter (DTG) and thyrotoxicosis during thiamazol treatment, in 20 persons with a DTG remission, and in 5 patients with postradiation and postoperative hypothyroses. AP

[The activity of nonspecific phosphatases of the thyroid gland in diffuse toxic goiter and thyrotoxic adenoma].

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COMPARISON OF ESTIMATES FOR THYROXINE SECRETION RATE DETERMINED BY THE GOITER PREVENTION ASSAY AND BY MEASURING THE ACTIVITY OF SERUM ALKALINE PHOSPHATASE IN THE CHICKEN.

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[Changes in the lactate dehydrogenase and alkaline and acid phosphatase activity in the arterial and venous blood serum and thyroid tissue in different forms of goiter].

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[Studies on the thyroid gland. II. Phosphatase and aldolase activity in surgical material from goiter changed human thyroid glands].

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[Alkaline phosphatase of granulocytes in patients before and after the surgical treatment of neutral goiter].

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The ultrastructural localization of acid phosphatase, alkaline phosphatase and adenosine triphosphatase in induced goitres of Xenopus laevis Daudin tadpoles.

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[Neutrophil alkaline phosphatase in hyperthyroidism and neuter goiter].

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[Alkaline phosphatase and acid phosphatase in toxic and non-toxic goitre].

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Effects on bone mineral density by treatment of benign nodular goiter with mildly suppressive doses of L-thyroxine in a cohort women study.

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OBJECTIVE Thyroid diseases and their treatment may influence the osseous system. The influence that prolonged suppressive L-thyroxine (LT4) therapy may have on inducing subclinical hyperthyroidism on bone metabolism is still a matter of debate. The aim of the present study was to assess the effects

[Prospective study on incidence of primary hyperparathyroidism in patients with goiter].

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Primary hyperparathyroidismus (PHPT) may coexist with goitre. Measurement of calcium concentration in blood serum is recommended before goiter operation. However, because of subclinical PHPT with normocalcemia this screening is insufficient for diagnosis of all PHPT cases. OBJECTIVE of the study is

Prospective study of bone loss in pre- and post-menopausal women on L-thyroxine therapy for non-toxic goitre.

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OBJECTIVE Hyperthyroidism is associated with increased bone turnover and bone resorption, but the effects of suppressive doses of thyroxine in treating non-toxic goitre remain unclear. We carried out a longitudinal study to evaluate the effect on bone of L-thyroxine (L-T4) therapy in women with

Treatment of benign nodular goitre with mildly suppressive doses of L-thyroxine: effects on bone mineral density and on nodule size.

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OBJECTIVE To evaluate (i) the demineralizing effect of L-thyroxine (LT4) therapy at doses mildly inhibiting serum thyroid stimulating hormone (TSH) in patients with benign nodular goitre; (ii) the efficacy of treatment on nodule size. METHODS Cross-sectional study comparing euthyroid women with

Reduced forearm bone mineral content and biochemical evidence of increased bone turnover in women with euthyroid goitre treated with thyroid hormone.

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We used single-photon absorptiometry to assess forearm bone mineral content (BMC/BW) (arbitrary units normalized for bone width) at a proximal site (PBMC/BW) and at a more distal site (DBMC/BW) in 60 women treated with 25-50 micrograms T3 or 50-100 micrograms T4 for euthyroid goitre, in 13 untreated

A slightly suppressive dose of L-thyroxine does not affect bone turnover and bone mineral density in pre- and postmenopausal women with nontoxic goitre.

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There are controversial reports on the potential role of L-thyroxine administration as a risk factor for osteoporosis. We studied bone mass and metabolism in a homogeneous series of 50 Caucasian women, 25 premenopausal and 25 postmenopausal, having nontoxic goitre treated with slightly suppressive

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