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Pagina 1 a partire dal 129 risultati
Changes of the plasma ketone body level and arterial ketone body ratio at the onset of mild aneurysmal subarachnoid hemorrhage.
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OBJECTIVE
The purpose of this study was to investigate the physiologic changes of ketone bodies in patients with aneurysmal subarachnoid hemorrhage. We tested the hypothesis that the plasma ketone bodies are associated with the vasoconstrictor and lipolysis effect of circulating
[Changes in arterial ketone body ratio (AKBR) in subarachnoid hemorrhage patients].
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The arterial ketone body ratio (AKBR) is considered an accurate index of the functional reserve of the liver, and its validity has been confirmed in the field of abdominal surgery. We found low AKBR values subarachnoid hemorrhage patients and discuss the clinical significance of this finding in this
[Changes of arterial ketone body ratio (AKBR) in patients with intracerebral hemorrhage].
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The arterial ketone body ratio (AKBR) is considered to be an accurate index of the functional reserve of the liver, and the validity of this idea has been confirmed in the field of abdominal surgery. We found low AKBR value intracerebral hemorrhage patients and discussed the clinical significance of
Arterial ketone body ratio as a parameter of hepatic mitochondrial redox state during and after hemorrhagic shock.
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This study was aimed at clarifying the usefulness of the arterial ketone body ratio (AKBR), which reflects hepatic mitochondrial redox state and closely correlates with hepatic energy production, for understanding the degree of hepatic mitochondrial damage and the extent of the deterioration in
Pathophysiology of hemorrhagic shock. A role of arterial ketone body ratio as an index of anoxic metabolism of the liver in acute blood loss.
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The changes in the oxido-reduction state and the adenylate energy charge of the liver were investigated in rats having 16%, 50%, and 84% mortality rates after acute hemorrhage. In the arterial blood, concentrations of ketone bodies, pyruvate, lactate, and the ratios (acetoacetate to
Ketone and pyruvate Ringer's solutions decrease pulmonary apoptosis in a rat model of severe hemorrhagic shock and resuscitation.
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BACKGROUND
Resuscitation fluids containing beta-hydroxybutyrate (BHB) have been shown to decrease cellular injury after hemorrhagic shock and resuscitation through an unknown mechanism. We tested whether this effect was related to BHB-induced metabolic modulations.
METHODS
Male Sprague Dawley rats
Effect of ketone bodies on hyperglycemia and lactic acidemia in hemorrhagic stress.
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BACKGROUND
To investigate the effect of hyperketonemia on altered glucose metabolism under stress conditions, we infused sodium D-3-hydroxybutyrate (3-OHB) into rats in hemorrhagic hypotension and evaluated the plasma concentration of substrates related to glucose metabolism.
METHODS
Three groups of
Fatty acid and ketone metabolism during hemorrhage and shock in the rat.
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[Striatal petechial hemorrhage and non-ketone hyperglycemia as the cause of hemichorea in a patient with unknown diabetes mellitus].
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Inhibition of human leukocyte elastase (HLE) by N-substituted peptidyl trifluoromethyl ketones.
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A series of tripeptides possessing trifluoromethyl or aryl ketone residues at P1 were prepared and evaluated both in vitro and in vivo as potential inhibitors of human leukocyte elastase (HLE). Tripeptides containing non naturally occurring N-substituted glycine residues at the P2-position have been
Synthesis, characterization, molecular docking, analgesic, antiplatelet and anticoagulant effects of dibenzylidene ketone derivatives.
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In this study dibenzylidene ketone derivatives (2E,5E)-2-(4-methoxybenzylidene)-5-(4-nitrobenzylidene) cyclopentanone (AK-1a) and (1E,4E)-4-(4-nitrobenzylidene)-1-(4-nitrophenyl) oct-1-en-3-one (AK-2a) were newly synthesized, inspired from curcuminoids natural origin. Novel scheme was used for
Optimization of ketone-based P2Y(12) receptor antagonists as antithrombotic agents: pharmacodynamics and receptor kinetics considerations.
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Modification of a series of P2Y12 receptor antagonists by replacement of the ester functionality was aimed at minimizing the risk of in vivo metabolic instability and pharmacokinetic variability. The resulting ketones were then optimized for their P2Y12 antagonistic and anticoagulation effects in
The inhibitory effect of 2-thienyl 2'-hydroxyphenyl ketone (C85) on platelet thromboxane formation.
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A new synthetic compound, 2-thienyl 2'-hydroxyphenyl ketone (C85), was demonstrated as an antiplatelet agent. In rabbit washed platelets, C85 dose-dependently inhibited arachidonic acid(AA), collagen and platelet activating factor(PAF)-induced platelet aggregation and ATP release with IC50 values of
Interruption of acute platelet-dependent thrombosis by the synthetic antithrombin D-phenylalanyl-L-prolyl-L-arginyl chloromethyl ketone.
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Since the antithrombin action of heparin fails to interrupt arterial thrombosis, a mediating role for thrombin (EC 3.4.21.5) in the formation of high-shear platelet-dependent thrombus has been unproven. To determine whether thrombin is important in acute arterial thrombus formation and to assess the
Hepatic apoptosis after hemorrhagic shock in rats can be reduced through modifications of conventional Ringer's solution.
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BACKGROUND
Resuscitation with racemic lactated Ringer's solution induces cellular apoptosis. This study was conducted to determine if the elimination of D-lactate isomer would attenuate apoptosis in the liver, and to investigate the possible mechanisms.
METHODS
Sprague Dawley rats (n=30, 5 per
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