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laryngismus/seizures
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Pagina 1 a partire dal 64 risultati
Laryngospasm, central and obstructive apnea during seizures: Defining pathophysiology for sudden death in a rat model.
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Seizure spread into the autonomic nervous system can result in life-threatening cardiovascular and respiratory dysfunction. Here we report on a less-studied consequence of such autonomic derangements-the possibility of laryngospasm and upper-airway occlusion. We used parenteral kainic acid to induce
Laryngospasm and neonatal seizure due to hypocalcaemia and vitamin D deficiency: an emergency condition in NICU and challenge to the neonatologist.
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Transformed ECG Signals Highlight Similarities Between Obstructive Sleep Apnea and Obstructive Apnea due to Seizure-Induced Laryngospasm.
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Transformed ECG Signals Highlight Similarities Between Obstructive Sleep Apnea and Obstructive Apnea due to Seizure-Induced Laryngospasm.
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[Laryngospasm with convulsion in an infant. Caution: danger of confusion with the bottles unidoses of chlorhexidine].
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Temporal lobe seizure presenting as "laryngospasm".
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On Respiratory Convulsions, with Especial Reference to Laryngismus Stridulus and Allied Conditions in Infancy.
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Electrical Stimulations of the Human Insula: Their Contribution to the Ictal Semiology of Insular Seizures.
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BACKGROUND
Stereotactic stimulations of the insular cortex through intracranial electrodes aim at characterizing the semiology of insular seizures. These stimulations, carried out in the context of Stereo-Electro-Encephalography (SEEG) during presurgical monitoring of epilepsy, reproduce the ictal
Possible role of SCN4A skeletal muscle mutation in apnea during seizure.
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SCN4A gene mutations cause a number of neuromuscular phenotypes including myotonia. A subset of infants with myotonia-causing mutations experience severe life-threatening episodic laryngospasm with apnea. We have recently identified similar SCN4A mutations in association with sudden
An unusual cause of stridor in childhood due to focal epileptic seizures.
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Respiratory disorders with stridor are a frequent cause of admission for children in an emergency department. Laryngospasm, as an isolated symptom of epilepsy, is a rare phenomenon [1, 3, 5]. Other respiratory symptoms of epilepsy, rarely seen in childhood, might be apnoeic spells [2, 4]. We report
Severe postictal laryngospasm as a potential mechanism for sudden unexpected death in epilepsy: a near-miss in an EMU.
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A 42-year-old man with refractory epilepsy experienced a 1-min generalized tonic-clonic seizure followed by persistent inspiratory stridor and cyanosis while being monitored in our epilepsy monitoring unit (EMU). Although his cardiac parameters remained stable throughout the event, the patient's
Ictal laryngospasm monitored by video-EEG and polygraphy: a potential SUDEP mechanism.
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A 56-year-old man with refractory bitemporal epilepsy was monitored in the Epilepsy Monitoring Unit (EMU). In a video-EEG captured seizure, brief oroalimentary automatisms were followed by increased inspiratory effort, accompanied by prominent, visible tracheal movements and audible inspiratory
[Postoperative acute laryngeal spasm in 1 cases of thyroid cancer].
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Thirty-eight years old male patient. Accepted the radical thyroidectomy for thyroid cancer in our department. When surgery was ended, laryngeal spasm occurred during pulling out the tracheal intubation, the quick check of calcium was 1.87 mmol/L, after intravenous injection the calcium gluconate the
Obstructive apnea due to laryngospasm links ictal to postictal events in SUDEP cases and offers practical biomarkers for review of past cases and prevention of new ones.
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Seizure spread into autonomic and respiratory brainstem regions is thought to play an important role in sudden unexpected death in epilepsy (SUDEP). As the clinical dataset of cases of definite SUDEP available for study grows, evidence points to a sequence of events that includes postictal apnea,
Acid reflux induced laryngospasm as a potential mechanism of sudden death in epilepsy.
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OBJECTIVE
Recent research suggests that obstructive laryngospasm and consequent respiratory arrest may be a mechanism in sudden unexpected death in epilepsy. We sought to test a new hypothesis that this laryngospasm is caused by seizures driving reflux of stomach acid into the larynx, rather than
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