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myo inositol/seizures
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Pagina 1 a partire dal 69 risultati
Restoration of brain myo-inositol levels in rats increases latency to lithium-pilocarpine seizures.
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Lithium pretreatment in rats potentiates the epileptogenic effects of pilocarpine and other cholinergic agonists. In order to determine if this effect of lithium could be reversed by myo-inositol, rats were pretreated with intracerebroventricular (ICV) injections of myoinositol, artificial CSF or
Crystal structure of human myo-inositol monophosphatase 2, the product of the putative susceptibility gene for bipolar disorder, schizophrenia, and febrile seizures.
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The human IMPA2 gene, which encodes myo-inositol monophosphatase 2 (IMPA2), is mapped onto 18p11.2, a susceptibility region for bipolar disorder. This chromosomal region has also been proposed to include a susceptibility locus for schizophrenia and febrile seizures. Here we report the crystal
Kainic acid-induced seizure upregulates Na(+)/myo-inositol cotransporter mRNA in rat brain.
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A major organic osmolyte, myo-inositol protects cells from perturbing effects of high intracellular concentrations of electrolytes. Myo-inositol is accumulated into cells through Na(+)/myo-inositol cotransporter (SMIT). In order to investigate the regulation of SMIT in generalized seizure, we
Anticonvulsant activities of myo-inositol and scyllo-inositol on pentylenetetrazol induced seizures.
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Myo-inositol (MI) and its isomers are used for the treatment of various neuropathological conditions. The purpose of the present research was to study anticonvulsant properties of MI and scyllo-inositol (SCI) on pentylenetetrazol (PTZ) induced seizures in rats. Half an hour after treatment with MI
Effect of myo-inositol on convulsions induced by pentylenetetrazole and kainic acid in rats.
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We studied the effect of myo-inositol on pentylenetetrazole and kainic acid-induced seizures in rats. Myo-inositol significantly reduced seizure activity.
A comparison of the ability of myo-inositol and epi-inositol to attenuate lithium-pilocarpine seizures in rats.
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[An experimental study of anticonvulsant effects of myo-inositol and folic acid].
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OBJECTIVE
To study the micronutrients that potentiate the action of anticonvulsant drugs, and in particular, of myo-inositol (vitamin B8).
METHODS
An experimental study of neurotrophic and anticonvulsant effects of a preparation based on myo-inositol (inofert) on thiosemicarbazide models of seizures
Linkage and association of febrile seizures to the IMPA2 gene on human chromosome 18.
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BACKGROUND
Febrile seizures (FSs) are the most common form of childhood seizures, and genetic factors play a role in susceptibility to FS.
OBJECTIVE
To identify novel loci and genes associated with susceptibility to FS.
METHODS
Study participants were the FS probands and family members of 59
Magnetisation transfer ratio of choline is reduced following epileptic seizures.
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The purpose of this study was to characterise the concentration and magnetisation transfer ratio (MTR) of brain metabolites following epileptic seizures. Magnetic resonance spectroscopy was performed in 10 patients with temporal or extra-temporal lobe epilepsy as soon as possible after a seizure,
[(1)H-magnetic resonance spectroscopy on bilateral thalamus of patients with secondarily generalized tonic-clonic seizures].
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OBJECTIVE
To examine the changes of metabolites in the bilateral thalamus of patients with secondarily generalized tonic-clonic seizure (SGTCS) and to explore the mechanism of SGTCS.
METHODS
Thirty patients with SGTCS (epilepsy group) and 30 matched healthy controls (control group) were examined by
Genetic variants in the IMPA2 gene do not confer increased risk of febrile seizures in Caucasian patients.
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Pathogenesis of febrile seizures (FS), causing the most common of types of seizures in children, remains unknown. Genetic factors appear to play a pivotal role and FS can be inherited as a monogenic or genetically complex disorder. Several risks factors have been proposed but many of the previously
Myo-inositol attenuates two specific behavioral effects of acute lithium in rats.
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Acute and chronic lithium treatment reduces levels of brain myo-inositol in rats. Several biological effects of lithium can be reversed in vitro by addition of myo-inositol. The ability of myo-inositol to reverse behavioral effects of lithium was tested using chronic inositol administration or acute
Spatial expression patterns and biochemical properties distinguish a second myo-inositol monophosphatase IMPA2 from IMPA1.
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Lithium is used in the clinical treatment of bipolar disorder, a disease where patients suffer mood swings between mania and depression. Although the mode of action of lithium remains elusive, a putative primary target is thought to be inositol monophosphatase (IMPase) activity. Two IMPase genes
Lithium-pilocarpine seizures as a model for lithium action in mania.
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Lithium (Li) pre-treatment of rats or mice given low dose pilocarpine induces a unique limbic seizure syndrome. This syndrome is stereospecifically reversed by myo-inositol, which suggests that it is a behavioral model for Li depletion of brain inositol. However, this syndrome has little face
Behavioural phenotyping of sodium-myo-inositol cotransporter heterozygous knockout mice with reduced brain inositol.
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Inositol plays a key role in dopamine, serotonin, noradrenaline and acetylcholine neurotransmission, and inositol treatment is reported to have beneficial effects in depression and anxiety. Therefore, a reduction in brain intracellular inositol levels could be a cause of some psychiatric disorders,
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