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protein s deficiency/triglyceride

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ArticoliTest cliniciBrevetti
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OBJECTIVE A single kindred in North America with venous thrombosis was described as having defective fibrinolysis because of increased levels of plasminogen activator inhibitor-1 (PAI-1). Our study describes the discovery of protein S deficiency in this kindred and its association with venous

Associations of protein C and protein S with serum lipid concentrations.

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Difficulties in the laboratory measurement of protein C and protein S levels cause problems in the diagnosis of deficiency states in individual patients and may complicate estimation of the prevalence of these states in the general population. Some difficulties may be due to unappreciated influences

Markers of low activity of tissue plasminogen activator/plasmin are prevalent in schizophrenia patients.

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BACKGROUND Clot buster tissue plasminogen activator (tPA) and its end-product plasmin play a well-defined role in neurochemistry. They mediate a number of events that culminate in tolerance against excitotoxicity, hippocampal neurogenesis, synaptic remodeling, neuronal plasticity, cognitive and

Lack of association between hemorheological alterations and upper-extremity deep vein thrombosis.

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BACKGROUND There is an association between rheological alterations and lower extremity deep vein thrombosis, although no research has been carried out regarding the possible influence of rheological alterations in the development of venous thrombosis in the upper limbs. METHODS We have determined in
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