actinomycin d/ניוון ביולוגיה

הקישור נשמר בלוח

מאמריםניסויים קלינייםפטנטים

בחר סוג מיון

עמוד 1 מ 90 תוצאות

[Wallerian degeneration of the rat sciatic nerve. Lack of effect of cycloheximide and of actinomycin D on the increase of radiophosphate incorporation in acid soluble phosphates observed after section of the nerve].

רק משתמשים רשומים יכולים לתרגם מאמרים

התחבר הרשם

Plasma inorganic phosphate incorporation into inorganic and organic acid-soluble phosphate fractions of the rat sciatic nerve increases markedly during Wallerian degeneration. Administration of cycloheximide and actinomycin D remains without effect on this specific increase which appears thus

Inhibitory effect of actinomycin D on tail atrophy in Xenopus larvae at metamorphosis.

רק משתמשים רשומים יכולים לתרגם מאמרים

התחבר הרשם

Effects of actinomycin D on developing hamster molar tooth germs in vitro.

רק משתמשים רשומים יכולים לתרגם מאמרים

התחבר הרשם

The aim of this study was to evaluate the toxic effects of actinomycin D on the developing hamster tooth germ in organ culture. Hamster tooth germs during early secretory amelogenesis were exposed in vitro for 24 h to 10(-9) M-5 x 10(-5) M actinomycin D. Actinomycin D dose-dependently (> or = 10(-7)

Caspase Inhibitor z-VAD-FMK Increases the Survival of Hair Cells After Actinomycin-D-induced Damage in Vitro

רק משתמשים רשומים יכולים לתרגם מאמרים

התחבר הרשם

Actinomycin-D (Act-D) is a highly effective chemotherapeutic agent that induces apoptosis in systemic tissues. Act-D combined with other chemotherapeutic agents exhibits ototoxic effects and causes hearing impairment. To investigate the potential toxic effects of Act-D in the inner ear, we treated

The differential sensitivity of cultured chick mesodermal cells to actinomycin D.

רק משתמשים רשומים יכולים לתרגם מאמרים

התחבר הרשם

Cells were isolated from the somite mesoderm and from the unsegmented (presomite) mesoderm of early chick embryos and exposed to actinomycin D in single cell culture. Actinomycin D inhibited proliferation in cell cultures derived from the unsegmented mesoderm, although the same concentrations of

[Postnatal growth inhibition in the rat retina after actinomycin D adminstration].

רק משתמשים רשומים יכולים לתרגם מאמרים

התחבר הרשם

The effects of actinomycin D on the development of the rats retina were observed. At the day of birth the inner neurons and the inner cells of the bipolar layer are vulnerable. The pale degeneration of these neurons accompanied by a dilatation of the endoplasmatic reticulum and the dark degeneration

Is actinomycin D suitable for the investigation of memory processes?

רק משתמשים רשומים יכולים לתרגם מאמרים

התחבר הרשם

The influence of Actinomycin D (AMD) applied intrahippocampally at doses of 1-6 mug/animal, on the acquisition and retention of a shock-motivated brightness discrimination was studied on rats in a semiautomatic Y-maze. The injection of AMD 4 hr prior to training did not influence the acquisition,

Nerve regeneration in Wld(s) mice is normalized by actinomycin D.

רק משתמשים רשומים יכולים לתרגם מאמרים

התחבר הרשם

Injured nerves of Wld(s) mice neither degenerate nor regenerate for several weeks. We have conjectured that Wld(s) axons have the ability to regenerate but its expression is impaired by the Schwann cells of the undegenerated distal stump. To test this conjecture, transcription was locally arrested

Neuronal chromatin changes in layer V pyramidal cells of somatomotor cortex after pyramidal tract lesions as demonstrated by [3H]actinomycin D binding.

רק משתמשים רשומים יכולים לתרגם מאמרים

התחבר הרשם

Changes in chromatin structure of pyramidal tract neurons after medullary pyramidal tract lesions were examined autoradiographically utilizing [3H]actinomycin D (Act D) binding to nuclei in frozen sections of brain. After a right pyramidal tract lesion, the binding of Act D to nuclei of axotomized

Downregulation of inhibitor of apoptosis proteins in apoptotic human chondrocytes treated with tumor necrosis factor-alpha and actinomycin D.

רק משתמשים רשומים יכולים לתרגם מאמרים

התחבר הרשם

OBJECTIVE Apoptosis of chondrocytes plays a pivotal role in cartilage degeneration. Tumor necrosis factor-alpha (TNF-alpha) is a proinflammatory cytokine and has been assumed to cause the degradation of human cartilage. To investigate the mechanisms of TNF-alpha-mediated apoptosis of human

Fibroblast growth factor and glutamate: opposing roles in the generation and degeneration of hippocampal neuroarchitecture.

רק משתמשים רשומים יכולים לתרגם מאמרים

התחבר הרשם

Neuritic regression and cell death (neurodegeneration) are common features of both normal nervous system development and neurodegenerative disorders. Growth factors and excitatory amino acid neurotransmitters have been suggested independently to play roles in neurodegenerative processes. The present

On the differential cytotoxicity of actinomycin D.

רק משתמשים רשומים יכולים לתרגם מאמרים

התחבר הרשם

Actinomycin D (AMD) at concentrations that inhibit cellular RNA synthesis by 85% or more causes an acute phase of lethal cell degeneration in HeLa cultures beginning as early as 3 hr after drug exposure, resulting in the nearly complete loss of viable cells by 12 hr. The loss of cells during this

Inhibitory effects of actinomycin D and cycloheximide on neuronal death in adult Manduca sexta.

רק משתמשים רשומים יכולים לתרגם מאמרים

התחבר הרשם

A decline in circulating 20-hydroxyecdysone permits the emergence of the adult Manduca sexta moth; this endocrine signal also triggers the death of approximately half of the neurons in the unfused abdominal ganglia of the moth central nervous system. This programmed death of neurons was markedly

Staurosporine-induced cell death in Tetrahymena thermophila has mixed characteristics of both apoptotic and autophagic degeneration.

רק משתמשים רשומים יכולים לתרגם מאמרים

התחבר הרשם

Staurosporine blocks signal transduction associated with cell survival, proliferation and chemosensory behaviour in the ciliated protozoan, Tetrahymena thermophila. Staurosporine inhibits cell proliferation and in vivo protein phosphorylation induced by phorbol ester. It also reduces the in vitro

The role of glucagon-like peptide-2 on apoptosis, cell proliferation, and oxidant-antioxidant system at a mouse model of intestinal injury induced by tumor necrosis factor-alpha/actinomycin D.

רק משתמשים רשומים יכולים לתרגם מאמרים

התחבר הרשם

Tumor necrosis factor-alpha (TNF-α) is a multifunctional cytokine, which has the ability to produce cytotoxicity via induction of cell death and cell cycle arrest. Blocking the synthesis of protective proteins through a transcriptional inhibitor such as actinomycin D (Act D) sensitizes many cell

קודם
1
2
3
4
5
6
הַבָּא

הצטרפו לדף הפייסבוק שלנו

Herbal & Natural Medicine

המאגר השלם ביותר של צמחי מרפא המגובה על ידי המדע

עובד ב 55 שפות
מרפא צמחי מרפא מגובה על ידי מדע
זיהוי עשבי תיבול על ידי דימוי
מפת GPS אינטראקטיבית - תייגו עשבי תיבול במיקום (בקרוב)
קרא פרסומים מדעיים הקשורים לחיפוש שלך
חפש עשבי מרפא על פי השפעותיהם
ארגן את תחומי העניין שלך והתעדכן במחקר החדשות, הניסויים הקליניים והפטנטים

הקלד סימפטום או מחלה וקרא על צמחי מרפא שעשויים לעזור, הקלד עשב וראה מחלות ותסמינים שהוא משמש נגד.
* כל המידע מבוסס על מחקר מדעי שפורסם