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glomerulonephritis/edema

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Glomerulonephritis and anasarca in a colony of frogs.

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A colony of frogs experienced excessive mortality due to glomerulonephritis. The presenting symptom in all cases was bloating due to hydrocoelom and anasarca. Mycobacteria sp was suspected to be a source of chronic antigenic stimulation that resulted in the glomerulonephritis. The prognosis was

[The participation of the kallikrein-kinin system in edema formation in glomerulonephritis].

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Involvement of the kallikrein-kinin system in the pathogenesis of renal edemas may be mediated by increase of vascular permeability, proteinuria, diuresis and natriuresis. Proceeding from these points, in 27 patients with morphologically proved chronic glomerulonephritis and the nephrotic syndrome,

[The x-ray diagnosis of a predisposition to nephrogenic pulmonary edema in glomerulonephritis patients].

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To develop criteria for determining predisposition to pulmonary edema in patients with glomerulonephritis, clinical, laboratory and X-ray examinations were made in 697 patients with glomerulonephritis at different stages of its development. X-ray examination included chest tele X-ray and its

Poststreptococcal glomerulonephritis with pulmonary edema presenting as respiratory distress.

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Acute poststreptococcal glomerulonephritis(PSGN) is characterized by an abrupt onset of edema,hypertension, and hematuria. Although the association of pulmonary edema with acute glomerulonephritis has been established, it is uncommon for children with PSGN to present with respiratory distress due to
Ion- and osmoregulatory capacity of the kidneys under induced water diuresis have been investigated in 100 patients with chronic glomerulonephritis (CGN) with reference to CGN clinical and morphological variants. An independent morphologist conducted a blind assessment of the results obtained at

Late-onset cystoid macular edema as a presenting symptom of anca-negative pauciimmune crescentic glomerulonephritis.

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OBJECTIVE The purpose of this study was to report the appearance of late-onset cystoid macular edema (CME) as an initial manifestation of antineutrophil cytoplasmic antibody-negative pauciimmune glomerulonephritis. METHODS Optical coherence tomography was obtained on a female patient after
A 28-year-old woman had hypothalamic disorders (amenorrhea, obesity, psychiatric abnormalities, polydipsia and fever) and chronic glomerulonephritis. She also suffered from general edema associated with cyclical oliguria and polyuria. Her body weight and plasma osmolality increased during the

[Study of various factors of the pathogenesis of nephrotic edema in children with glomerulonephritis].

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As many as 26 children (20 boys and 6 girls) aged 2.5 to 15.5 years suffering from active nephrotic glomerulonephritis were examined for the intravascular liquid volume and antidiuretic hormone in blood plasma. In accordance with the magnitude of the circulating blood volume, 3 groups of patients

[PULMONARY EDEMA IN ACUTE GLOMERULONEPHRITIS].

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Acute glomerulonephritis and cerebral edema.

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[Proceedings: Role of physical and natriuretic factors in the pathology of edema in glomerulonephritis].

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[Acute edema of the lung. Isolated primary interstitial myocarditis (Fiedler's). Acute diffuse ischemic glomerulonephritis].

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[Pulmonary edema during the course of acute glomerulonephritis treated with chlorpromazine].

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Some effects of cortisone on the metabolic disturbance associated with glomerulonephritis with edema.

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